Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis

Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been c...
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Autor*in:	A. V. Ershov [verfasserIn] V. T. Dolgikh [verfasserIn] O. V. Korpacheva [verfasserIn] V. V. Rusakov [verfasserIn] N. N. Nikolaev [verfasserIn] S. V. Palianov [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch ; Russisch

Erschienen:	2017

Schlagwörter:	острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет

Übergeordnetes Werk:	In: Общая реаниматология - Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russia, 2017, 13(2017), 1, Seite 34-44
Übergeordnetes Werk:	volume:13 ; year:2017 ; number:1 ; pages:34-44

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc Journal toc

DOI / URN:	10.15360/1813-9779-2017-1-34-44

Katalog-ID:	DOAJ000341991

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520			\|a Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization.
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allfields	10.15360/1813-9779-2017-1-34-44 doi (DE-627)DOAJ000341991 (DE-599)DOAJd84724515107499d9d82d2c7a51be9f8 DE-627 ger DE-627 rakwb eng rus RC86-88.9 A. V. Ershov verfasserin aut Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization. острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет Medical emergencies. Critical care. Intensive care. First aid V. T. Dolgikh verfasserin aut O. V. Korpacheva verfasserin aut V. V. Rusakov verfasserin aut N. N. Nikolaev verfasserin aut S. V. Palianov verfasserin aut In Общая реаниматология Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russia, 2017 13(2017), 1, Seite 34-44 (DE-627)822100088 (DE-600)2817379-X 24117110 nnns volume:13 year:2017 number:1 pages:34-44 https://doi.org/10.15360/1813-9779-2017-1-34-44 kostenfrei https://doaj.org/article/d84724515107499d9d82d2c7a51be9f8 kostenfrei https://www.reanimatology.com/rmt/article/view/1572 kostenfrei https://doaj.org/toc/1813-9779 Journal toc kostenfrei https://doaj.org/toc/2411-7110 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2017 1 34-44
spelling	10.15360/1813-9779-2017-1-34-44 doi (DE-627)DOAJ000341991 (DE-599)DOAJd84724515107499d9d82d2c7a51be9f8 DE-627 ger DE-627 rakwb eng rus RC86-88.9 A. V. Ershov verfasserin aut Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization. острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет Medical emergencies. Critical care. Intensive care. First aid V. T. Dolgikh verfasserin aut O. V. Korpacheva verfasserin aut V. V. Rusakov verfasserin aut N. N. Nikolaev verfasserin aut S. V. Palianov verfasserin aut In Общая реаниматология Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russia, 2017 13(2017), 1, Seite 34-44 (DE-627)822100088 (DE-600)2817379-X 24117110 nnns volume:13 year:2017 number:1 pages:34-44 https://doi.org/10.15360/1813-9779-2017-1-34-44 kostenfrei https://doaj.org/article/d84724515107499d9d82d2c7a51be9f8 kostenfrei https://www.reanimatology.com/rmt/article/view/1572 kostenfrei https://doaj.org/toc/1813-9779 Journal toc kostenfrei https://doaj.org/toc/2411-7110 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2017 1 34-44
allfields_unstemmed	10.15360/1813-9779-2017-1-34-44 doi (DE-627)DOAJ000341991 (DE-599)DOAJd84724515107499d9d82d2c7a51be9f8 DE-627 ger DE-627 rakwb eng rus RC86-88.9 A. V. Ershov verfasserin aut Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization. острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет Medical emergencies. Critical care. Intensive care. First aid V. T. Dolgikh verfasserin aut O. V. Korpacheva verfasserin aut V. V. Rusakov verfasserin aut N. N. Nikolaev verfasserin aut S. V. Palianov verfasserin aut In Общая реаниматология Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russia, 2017 13(2017), 1, Seite 34-44 (DE-627)822100088 (DE-600)2817379-X 24117110 nnns volume:13 year:2017 number:1 pages:34-44 https://doi.org/10.15360/1813-9779-2017-1-34-44 kostenfrei https://doaj.org/article/d84724515107499d9d82d2c7a51be9f8 kostenfrei https://www.reanimatology.com/rmt/article/view/1572 kostenfrei https://doaj.org/toc/1813-9779 Journal toc kostenfrei https://doaj.org/toc/2411-7110 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2017 1 34-44
allfieldsGer	10.15360/1813-9779-2017-1-34-44 doi (DE-627)DOAJ000341991 (DE-599)DOAJd84724515107499d9d82d2c7a51be9f8 DE-627 ger DE-627 rakwb eng rus RC86-88.9 A. V. Ershov verfasserin aut Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization. острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет Medical emergencies. Critical care. Intensive care. First aid V. T. Dolgikh verfasserin aut O. V. Korpacheva verfasserin aut V. V. Rusakov verfasserin aut N. N. Nikolaev verfasserin aut S. V. Palianov verfasserin aut In Общая реаниматология Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russia, 2017 13(2017), 1, Seite 34-44 (DE-627)822100088 (DE-600)2817379-X 24117110 nnns volume:13 year:2017 number:1 pages:34-44 https://doi.org/10.15360/1813-9779-2017-1-34-44 kostenfrei https://doaj.org/article/d84724515107499d9d82d2c7a51be9f8 kostenfrei https://www.reanimatology.com/rmt/article/view/1572 kostenfrei https://doaj.org/toc/1813-9779 Journal toc kostenfrei https://doaj.org/toc/2411-7110 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2017 1 34-44
allfieldsSound	10.15360/1813-9779-2017-1-34-44 doi (DE-627)DOAJ000341991 (DE-599)DOAJd84724515107499d9d82d2c7a51be9f8 DE-627 ger DE-627 rakwb eng rus RC86-88.9 A. V. Ershov verfasserin aut Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization. острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет Medical emergencies. Critical care. Intensive care. First aid V. T. Dolgikh verfasserin aut O. V. Korpacheva verfasserin aut V. V. Rusakov verfasserin aut N. N. Nikolaev verfasserin aut S. V. Palianov verfasserin aut In Общая реаниматология Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russia, 2017 13(2017), 1, Seite 34-44 (DE-627)822100088 (DE-600)2817379-X 24117110 nnns volume:13 year:2017 number:1 pages:34-44 https://doi.org/10.15360/1813-9779-2017-1-34-44 kostenfrei https://doaj.org/article/d84724515107499d9d82d2c7a51be9f8 kostenfrei https://www.reanimatology.com/rmt/article/view/1572 kostenfrei https://doaj.org/toc/1813-9779 Journal toc kostenfrei https://doaj.org/toc/2411-7110 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2017 1 34-44
language	English Russian
source	In Общая реаниматология 13(2017), 1, Seite 34-44 volume:13 year:2017 number:1 pages:34-44
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topic_facet	острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет Medical emergencies. Critical care. Intensive care. First aid
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container_title	Общая реаниматология
authorswithroles_txt_mv	A. V. Ershov @@aut@@ V. T. Dolgikh @@aut@@ O. V. Korpacheva @@aut@@ V. V. Rusakov @@aut@@ N. N. Nikolaev @@aut@@ S. V. Palianov @@aut@@
publishDateDaySort_date	2017-01-01T00:00:00Z
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topic_title	RC86-88.9 Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis острый деструктивный панкреатит хронический панкреатит постнекротические кисты поджелудочной железы панкреатогенный сахарный диабет
topic	misc RC86-88.9 misc острый деструктивный панкреатит misc хронический панкреатит misc постнекротические кисты поджелудочной железы misc панкреатогенный сахарный диабет misc Medical emergencies. Critical care. Intensive care. First aid
topic_unstemmed	misc RC86-88.9 misc острый деструктивный панкреатит misc хронический панкреатит misc постнекротические кисты поджелудочной железы misc панкреатогенный сахарный диабет misc Medical emergencies. Critical care. Intensive care. First aid
topic_browse	misc RC86-88.9 misc острый деструктивный панкреатит misc хронический панкреатит misc постнекротические кисты поджелудочной железы misc панкреатогенный сахарный диабет misc Medical emergencies. Critical care. Intensive care. First aid
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title	Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis
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title_full	Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis
author_sort	A. V. Ershov
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author_browse	A. V. Ershov V. T. Dolgikh O. V. Korpacheva V. V. Rusakov N. N. Nikolaev S. V. Palianov
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doi_str_mv	10.15360/1813-9779-2017-1-34-44
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title_sort	pathogenetic factors contributing to the development of delayed complications of acute destructive pancreatitis
callnumber	RC86-88.9
title_auth	Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis
abstract	Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization.
abstractGer	Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization.
abstract_unstemmed	Purpose. To evaluate pathogenic factors contributing to the development of delayed complications of newly diagnosed acute destructive pancreatitis (ADP).Materials and methods. A comprehensive examination, treatment and 5-year follow%up of 83 patients who underwent the first attack of ADP have been conducted. The day of admission to the hospital, 24 hours from the beginning of the intensive care, and Days 3 and 5 after the attack were selected as the timepoints of the study. Complete diagnostic procedures were performed once every six months after hospitalization for five years. The patients were followed%up every month. Depending on the further progression of ADP, the patients were divided into four groups. Group I included 27 patients with acute pancreatitis which evolved into pancreatic pseudocysts.Group II (n=43) was comprized of the patients with chronic pancreatitis; group III (n=8) included the patients who presented no pathological processes associated with structural and functional pancreatic changes over the next 5 years; and group IV (n=5) included the patients with pancreatogenic diabetes mellitus.Results. The study confirmed the role of autoimmune mechanisms and abnormal foci localization in pancreas in the development of pancreatogenic diabetes mellitus, contribution of inflammation to formation of post-necrotic pancreatic pseudocysts, as well as the significance of the patient compliance for development of chronic pancreatitis with extensive pancreatic lesions.Conclusion. Statistical analysis of the long-term monitoring of patients allowed to set the odds ratio evolution acute pancreatitis depending on its initial characteristics. At the same time we take into account the parameters and interpretation of the study which is possible in the first few days hospitalization.
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title_short	Pathogenetic Factors Contributing to the Development of Delayed Complications of Acute Destructive Pancreatitis
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