Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease

Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to...
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Gespeichert in:

Autor*in:	Sebastiano Gangemi [verfasserIn] Giorgio Basile [verfasserIn] Rosaria Alba Merendino [verfasserIn] Antonio Epifanio [verfasserIn] Giuseppe Di Pasquale [verfasserIn] Benito Ferlazzo [verfasserIn] Vittorio Nicita-mauro [verfasserIn] Letterio Morgante [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2003

Schlagwörter:	interleukin-15 RANTES Parkinson's disease levodopa.

Übergeordnetes Werk:	In: Mediators of Inflammation - Hindawi Limited, 2002, 12(2003), 4, Seite 251-253
Übergeordnetes Werk:	volume:12 ; year:2003 ; number:4 ; pages:251-253

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc Journal toc

DOI / URN:	10.1080/09629350310001599701

Katalog-ID:	DOAJ005387655

Internformat


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520			\|a Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy.
650		4	\|a interleukin-15
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650		4	\|a Parkinson's disease
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912			\|a GBV_ILN_2004
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952			\|d 12 \|j 2003 \|e 4 \|h 251-253

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publishDate	2003
allfields	10.1080/09629350310001599701 doi (DE-627)DOAJ005387655 (DE-599)DOAJf047e65592ef49859564496c23891399 DE-627 ger DE-627 rakwb eng RB1-214 Sebastiano Gangemi verfasserin aut Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease 2003 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy. interleukin-15 RANTES Parkinson's disease levodopa. Pathology Giorgio Basile verfasserin aut Rosaria Alba Merendino verfasserin aut Antonio Epifanio verfasserin aut Giuseppe Di Pasquale verfasserin aut Benito Ferlazzo verfasserin aut Vittorio Nicita-mauro verfasserin aut Letterio Morgante verfasserin aut In Mediators of Inflammation Hindawi Limited, 2002 12(2003), 4, Seite 251-253 (DE-627)320467384 (DE-600)2008065-7 14661861 nnns volume:12 year:2003 number:4 pages:251-253 https://doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/article/f047e65592ef49859564496c23891399 kostenfrei http://dx.doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/toc/0962-9351 Journal toc kostenfrei https://doaj.org/toc/1466-1861 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2003 4 251-253
spelling	10.1080/09629350310001599701 doi (DE-627)DOAJ005387655 (DE-599)DOAJf047e65592ef49859564496c23891399 DE-627 ger DE-627 rakwb eng RB1-214 Sebastiano Gangemi verfasserin aut Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease 2003 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy. interleukin-15 RANTES Parkinson's disease levodopa. Pathology Giorgio Basile verfasserin aut Rosaria Alba Merendino verfasserin aut Antonio Epifanio verfasserin aut Giuseppe Di Pasquale verfasserin aut Benito Ferlazzo verfasserin aut Vittorio Nicita-mauro verfasserin aut Letterio Morgante verfasserin aut In Mediators of Inflammation Hindawi Limited, 2002 12(2003), 4, Seite 251-253 (DE-627)320467384 (DE-600)2008065-7 14661861 nnns volume:12 year:2003 number:4 pages:251-253 https://doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/article/f047e65592ef49859564496c23891399 kostenfrei http://dx.doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/toc/0962-9351 Journal toc kostenfrei https://doaj.org/toc/1466-1861 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2003 4 251-253
allfields_unstemmed	10.1080/09629350310001599701 doi (DE-627)DOAJ005387655 (DE-599)DOAJf047e65592ef49859564496c23891399 DE-627 ger DE-627 rakwb eng RB1-214 Sebastiano Gangemi verfasserin aut Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease 2003 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy. interleukin-15 RANTES Parkinson's disease levodopa. Pathology Giorgio Basile verfasserin aut Rosaria Alba Merendino verfasserin aut Antonio Epifanio verfasserin aut Giuseppe Di Pasquale verfasserin aut Benito Ferlazzo verfasserin aut Vittorio Nicita-mauro verfasserin aut Letterio Morgante verfasserin aut In Mediators of Inflammation Hindawi Limited, 2002 12(2003), 4, Seite 251-253 (DE-627)320467384 (DE-600)2008065-7 14661861 nnns volume:12 year:2003 number:4 pages:251-253 https://doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/article/f047e65592ef49859564496c23891399 kostenfrei http://dx.doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/toc/0962-9351 Journal toc kostenfrei https://doaj.org/toc/1466-1861 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2003 4 251-253
allfieldsGer	10.1080/09629350310001599701 doi (DE-627)DOAJ005387655 (DE-599)DOAJf047e65592ef49859564496c23891399 DE-627 ger DE-627 rakwb eng RB1-214 Sebastiano Gangemi verfasserin aut Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease 2003 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy. interleukin-15 RANTES Parkinson's disease levodopa. Pathology Giorgio Basile verfasserin aut Rosaria Alba Merendino verfasserin aut Antonio Epifanio verfasserin aut Giuseppe Di Pasquale verfasserin aut Benito Ferlazzo verfasserin aut Vittorio Nicita-mauro verfasserin aut Letterio Morgante verfasserin aut In Mediators of Inflammation Hindawi Limited, 2002 12(2003), 4, Seite 251-253 (DE-627)320467384 (DE-600)2008065-7 14661861 nnns volume:12 year:2003 number:4 pages:251-253 https://doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/article/f047e65592ef49859564496c23891399 kostenfrei http://dx.doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/toc/0962-9351 Journal toc kostenfrei https://doaj.org/toc/1466-1861 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2003 4 251-253
allfieldsSound	10.1080/09629350310001599701 doi (DE-627)DOAJ005387655 (DE-599)DOAJf047e65592ef49859564496c23891399 DE-627 ger DE-627 rakwb eng RB1-214 Sebastiano Gangemi verfasserin aut Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease 2003 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy. interleukin-15 RANTES Parkinson's disease levodopa. Pathology Giorgio Basile verfasserin aut Rosaria Alba Merendino verfasserin aut Antonio Epifanio verfasserin aut Giuseppe Di Pasquale verfasserin aut Benito Ferlazzo verfasserin aut Vittorio Nicita-mauro verfasserin aut Letterio Morgante verfasserin aut In Mediators of Inflammation Hindawi Limited, 2002 12(2003), 4, Seite 251-253 (DE-627)320467384 (DE-600)2008065-7 14661861 nnns volume:12 year:2003 number:4 pages:251-253 https://doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/article/f047e65592ef49859564496c23891399 kostenfrei http://dx.doi.org/10.1080/09629350310001599701 kostenfrei https://doaj.org/toc/0962-9351 Journal toc kostenfrei https://doaj.org/toc/1466-1861 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2003 4 251-253
language	English
source	In Mediators of Inflammation 12(2003), 4, Seite 251-253 volume:12 year:2003 number:4 pages:251-253
sourceStr	In Mediators of Inflammation 12(2003), 4, Seite 251-253 volume:12 year:2003 number:4 pages:251-253
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	interleukin-15 RANTES Parkinson's disease levodopa. Pathology
isfreeaccess_bool	true
container_title	Mediators of Inflammation
authorswithroles_txt_mv	Sebastiano Gangemi @@aut@@ Giorgio Basile @@aut@@ Rosaria Alba Merendino @@aut@@ Antonio Epifanio @@aut@@ Giuseppe Di Pasquale @@aut@@ Benito Ferlazzo @@aut@@ Vittorio Nicita-mauro @@aut@@ Letterio Morgante @@aut@@
publishDateDaySort_date	2003-01-01T00:00:00Z
hierarchy_top_id	320467384
id	DOAJ005387655
language_de	englisch
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callnumber-first	R - Medicine
author	Sebastiano Gangemi
spellingShingle	Sebastiano Gangemi misc RB1-214 misc interleukin-15 misc RANTES misc Parkinson's disease misc levodopa. misc Pathology Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease
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topic_title	RB1-214 Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease interleukin-15 RANTES Parkinson's disease levodopa
topic	misc RB1-214 misc interleukin-15 misc RANTES misc Parkinson's disease misc levodopa. misc Pathology
topic_unstemmed	misc RB1-214 misc interleukin-15 misc RANTES misc Parkinson's disease misc levodopa. misc Pathology
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title	Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease
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title_full	Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease
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author_browse	Sebastiano Gangemi Giorgio Basile Rosaria Alba Merendino Antonio Epifanio Giuseppe Di Pasquale Benito Ferlazzo Vittorio Nicita-mauro Letterio Morgante
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title_sort	effect of levodopa on interleukin-15 and rantes circulating levels in patients affected by parkinson's disease
callnumber	RB1-214
title_auth	Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease
abstract	Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy.
abstractGer	Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy.
abstract_unstemmed	Parkinson's disease (PD) is an extra-pyramidal neurodegenerative disorder, in which alterations of the immune system are involved. Interleukin (IL)-15 stimulates cellular immune response and induces growth and differentiation of various immune cells. RANTES, promoting leukocyte infiltration to sites of inflammation, mediates the trafficking and homing of immune cells. To clarify the potential effect of levodopa on the immunological network of PD, we analyzed IL-15 and RANTES serum levels in PD patients, treated or not with levodopa, and in healthy donors. Levodopa-treated patients showed significantly higher IL-15 and RANTES circulating levels with respect to healthy controls and higher, although not significantly, levels with respect to untreated patients. So, we hypothesize that the immunological alterations found in PD may be linked, at least in part, to levodopa therapy.
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container_issue	4
title_short	Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease
url	https://doi.org/10.1080/09629350310001599701 https://doaj.org/article/f047e65592ef49859564496c23891399 http://dx.doi.org/10.1080/09629350310001599701 https://doaj.org/toc/0962-9351 https://doaj.org/toc/1466-1861
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up_date	2024-07-03T14:43:02.135Z
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Effect of levodopa on interleukin-15 and RANTES circulating levels in patients affected by Parkinson's disease

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