Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction

The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS)...
Ausführliche Beschreibung

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Autor*in:	Ting Wu [verfasserIn] Hua Li [verfasserIn] Bing Wu [verfasserIn] Lei Zhang [verfasserIn] San-Wu Wu [verfasserIn] Jia-Ning Wang [verfasserIn] You-En Zhang Ph.D. [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2017

Übergeordnetes Werk:	In: Cell Transplantation - SAGE Publishing, 2017, 26(2017)
Übergeordnetes Werk:	volume:26 ; year:2017

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc Journal toc

DOI / URN:	10.3727/096368917X695029

Katalog-ID:	DOAJ017563313

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520			\|a The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure.
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allfields	10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017
spelling	10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017
allfields_unstemmed	10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017
allfieldsGer	10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017
allfieldsSound	10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017
language	English
source	In Cell Transplantation 26(2017) volume:26 year:2017
sourceStr	In Cell Transplantation 26(2017) volume:26 year:2017
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container_title	Cell Transplantation
authorswithroles_txt_mv	Ting Wu @@aut@@ Hua Li @@aut@@ Bing Wu @@aut@@ Lei Zhang @@aut@@ San-Wu Wu @@aut@@ Jia-Ning Wang @@aut@@ You-En Zhang Ph.D. @@aut@@
publishDateDaySort_date	2017-01-01T00:00:00Z
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author	Ting Wu
spellingShingle	Ting Wu misc Medicine misc R Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
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topic_title	Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
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title	Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
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title_full	Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
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author_browse	Ting Wu Hua Li Bing Wu Lei Zhang San-Wu Wu Jia-Ning Wang You-En Zhang Ph.D.
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title_sort	hydrogen sulfide reduces recruitment of cd11bdg gr-1dg cells in mice with myocardial infarction
title_auth	Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
abstract	The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure.
abstractGer	The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure.
abstract_unstemmed	The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure.
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title_short	Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
url	https://doi.org/10.3727/096368917X695029 https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 https://doaj.org/toc/0963-6897 https://doaj.org/toc/1555-3892
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