Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction
The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS)...
Ausführliche Beschreibung
Autor*in: |
Ting Wu [verfasserIn] Hua Li [verfasserIn] Bing Wu [verfasserIn] Lei Zhang [verfasserIn] San-Wu Wu [verfasserIn] Jia-Ning Wang [verfasserIn] You-En Zhang Ph.D. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2017 |
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Übergeordnetes Werk: |
In: Cell Transplantation - SAGE Publishing, 2017, 26(2017) |
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Übergeordnetes Werk: |
volume:26 ; year:2017 |
Links: |
Link aufrufen |
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DOI / URN: |
10.3727/096368917X695029 |
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Katalog-ID: |
DOAJ017563313 |
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10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017 |
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10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017 |
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10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017 |
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10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017 |
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10.3727/096368917X695029 doi (DE-627)DOAJ017563313 (DE-599)DOAJ57301dd0ad34443b8ddbb14e480fe1f2 DE-627 ger DE-627 rakwb eng Ting Wu verfasserin aut Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. Medicine R Hua Li verfasserin aut Bing Wu verfasserin aut Lei Zhang verfasserin aut San-Wu Wu verfasserin aut Jia-Ning Wang verfasserin aut You-En Zhang Ph.D. verfasserin aut In Cell Transplantation SAGE Publishing, 2017 26(2017) (DE-627)320603504 (DE-600)2020466-8 15553892 nnns volume:26 year:2017 https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/article/57301dd0ad34443b8ddbb14e480fe1f2 kostenfrei https://doi.org/10.3727/096368917X695029 kostenfrei https://doaj.org/toc/0963-6897 Journal toc kostenfrei https://doaj.org/toc/1555-3892 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_374 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_2704 GBV_ILN_2707 GBV_ILN_2890 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 26 2017 |
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Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction |
abstract |
The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. |
abstractGer |
The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. |
abstract_unstemmed |
The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H 2 S) attenuates left ventricular remodeling after myocardial infarction (MI). MI was created in mice by left coronary artery ligation. One group of mice received injections of the H 2 S donor sodium hydrosulfide (NaHS) immediately before and 1 h after ligation, while the control group received saline alone. During both the subacute and chronic stages (1 and 4 weeks postinfarction, respectively), NaHS-treated mice demonstrated attenuation of cardiac dilation in the infarcted myocardium. Furthermore, fewer CD11b + Gr-1 + myeloid cells were detected in the infarct myocardium and peripheral blood from NaHS-treated mice, while more CD11b + Gr-1 + cells remained in the spleen and bone marrow in these animals. NaHS-treated mice also exhibited reduction in cardiomyocyte apoptosis, interstitial fibrosis, cardiac hypertrophy, and pulmonary edema, as well as overall better survival rates, when compared to controls. Thus, exogenous H 2 S has favorable effects on cardiac remodeling after MI. These observations further support the emerging concept that H 2 S treatment might have therapeutic benefits in the setting of ischemia-induced heart failure. |
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Hydrogen Sulfide Reduces Recruitment of CD11bDG Gr-1DG Cells in Mice with Myocardial Infarction |
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