Pain modulation by nitric oxide in the spinal cord.

Nitric oxide (NO) is a versatile messenger molecule first associated with endothelial relaxing effects. In the central nervous system (CNS), NO synthesis is primarily triggered by activation of N-methyl-D-aspartate (NMDA) receptors and has a Janus face, with both beneficial and harmful properties, d...
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Gespeichert in:

Autor*in:	Marco Aurelio M Freire [verfasserIn] Joanilson S Guimarães [verfasserIn] Walace Gomes-Leal [verfasserIn] Antonio Pereira [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2009

Schlagwörter:	Hyperalgesia Nitric Oxide Pain Renshaw cell Spinal Cord

Übergeordnetes Werk:	In: Frontiers in Neuroscience - Frontiers Media S.A., 2008, 3(2009)
Übergeordnetes Werk:	volume:3 ; year:2009

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.3389/neuro.01.024.2009

Katalog-ID:	DOAJ018946755

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allfieldsGer	10.3389/neuro.01.024.2009 doi (DE-627)DOAJ018946755 (DE-599)DOAJ04c68009c9cd4b369e94213b23bfd163 DE-627 ger DE-627 rakwb eng RC321-571 Marco Aurelio M Freire verfasserin aut Pain modulation by nitric oxide in the spinal cord. 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Nitric oxide (NO) is a versatile messenger molecule first associated with endothelial relaxing effects. In the central nervous system (CNS), NO synthesis is primarily triggered by activation of N-methyl-D-aspartate (NMDA) receptors and has a Janus face, with both beneficial and harmful properties, depending on concentration and the identity of its synthetic enzyme isoform. There are three isoforms of the NO synthesizing enzyme nitric oxide synthase (NOS): neuronal (nNOS), endothelial (eNOS), and inducible nitric oxide synthase (iNOS), each one involved with specific events in the brain. In CNS, nNOS is involved with modulation of synaptic transmission through long-term potentiation in several regions, including nociceptive circuits in the spinal cord. Here, we review the role played by NO on central pain sensitization. Hyperalgesia Nitric Oxide Pain Renshaw cell Spinal Cord Neurosciences. Biological psychiatry. Neuropsychiatry Joanilson S Guimarães verfasserin aut Walace Gomes-Leal verfasserin aut Antonio Pereira verfasserin aut Antonio Pereira verfasserin aut In Frontiers in Neuroscience Frontiers Media S.A., 2008 3(2009) (DE-627)55908109X (DE-600)2411902-7 1662453X nnns volume:3 year:2009 https://doi.org/10.3389/neuro.01.024.2009 kostenfrei https://doaj.org/article/04c68009c9cd4b369e94213b23bfd163 kostenfrei http://journal.frontiersin.org/Journal/10.3389/neuro.01.024.2009/full kostenfrei https://doaj.org/toc/1662-453X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009
allfieldsSound	10.3389/neuro.01.024.2009 doi (DE-627)DOAJ018946755 (DE-599)DOAJ04c68009c9cd4b369e94213b23bfd163 DE-627 ger DE-627 rakwb eng RC321-571 Marco Aurelio M Freire verfasserin aut Pain modulation by nitric oxide in the spinal cord. 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Nitric oxide (NO) is a versatile messenger molecule first associated with endothelial relaxing effects. In the central nervous system (CNS), NO synthesis is primarily triggered by activation of N-methyl-D-aspartate (NMDA) receptors and has a Janus face, with both beneficial and harmful properties, depending on concentration and the identity of its synthetic enzyme isoform. There are three isoforms of the NO synthesizing enzyme nitric oxide synthase (NOS): neuronal (nNOS), endothelial (eNOS), and inducible nitric oxide synthase (iNOS), each one involved with specific events in the brain. In CNS, nNOS is involved with modulation of synaptic transmission through long-term potentiation in several regions, including nociceptive circuits in the spinal cord. Here, we review the role played by NO on central pain sensitization. Hyperalgesia Nitric Oxide Pain Renshaw cell Spinal Cord Neurosciences. Biological psychiatry. Neuropsychiatry Joanilson S Guimarães verfasserin aut Walace Gomes-Leal verfasserin aut Antonio Pereira verfasserin aut Antonio Pereira verfasserin aut In Frontiers in Neuroscience Frontiers Media S.A., 2008 3(2009) (DE-627)55908109X (DE-600)2411902-7 1662453X nnns volume:3 year:2009 https://doi.org/10.3389/neuro.01.024.2009 kostenfrei https://doaj.org/article/04c68009c9cd4b369e94213b23bfd163 kostenfrei http://journal.frontiersin.org/Journal/10.3389/neuro.01.024.2009/full kostenfrei https://doaj.org/toc/1662-453X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009
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callnumber-first	R - Medicine
author	Marco Aurelio M Freire
spellingShingle	Marco Aurelio M Freire misc RC321-571 misc Hyperalgesia misc Nitric Oxide misc Pain misc Renshaw cell misc Spinal Cord misc Neurosciences. Biological psychiatry. Neuropsychiatry Pain modulation by nitric oxide in the spinal cord.
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topic_title	RC321-571 Pain modulation by nitric oxide in the spinal cord Hyperalgesia Nitric Oxide Pain Renshaw cell Spinal Cord
topic	misc RC321-571 misc Hyperalgesia misc Nitric Oxide misc Pain misc Renshaw cell misc Spinal Cord misc Neurosciences. Biological psychiatry. Neuropsychiatry
topic_unstemmed	misc RC321-571 misc Hyperalgesia misc Nitric Oxide misc Pain misc Renshaw cell misc Spinal Cord misc Neurosciences. Biological psychiatry. Neuropsychiatry
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title	Pain modulation by nitric oxide in the spinal cord.
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title_sort	pain modulation by nitric oxide in the spinal cord
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title_auth	Pain modulation by nitric oxide in the spinal cord.
abstract	Nitric oxide (NO) is a versatile messenger molecule first associated with endothelial relaxing effects. In the central nervous system (CNS), NO synthesis is primarily triggered by activation of N-methyl-D-aspartate (NMDA) receptors and has a Janus face, with both beneficial and harmful properties, depending on concentration and the identity of its synthetic enzyme isoform. There are three isoforms of the NO synthesizing enzyme nitric oxide synthase (NOS): neuronal (nNOS), endothelial (eNOS), and inducible nitric oxide synthase (iNOS), each one involved with specific events in the brain. In CNS, nNOS is involved with modulation of synaptic transmission through long-term potentiation in several regions, including nociceptive circuits in the spinal cord. Here, we review the role played by NO on central pain sensitization.
abstractGer	Nitric oxide (NO) is a versatile messenger molecule first associated with endothelial relaxing effects. In the central nervous system (CNS), NO synthesis is primarily triggered by activation of N-methyl-D-aspartate (NMDA) receptors and has a Janus face, with both beneficial and harmful properties, depending on concentration and the identity of its synthetic enzyme isoform. There are three isoforms of the NO synthesizing enzyme nitric oxide synthase (NOS): neuronal (nNOS), endothelial (eNOS), and inducible nitric oxide synthase (iNOS), each one involved with specific events in the brain. In CNS, nNOS is involved with modulation of synaptic transmission through long-term potentiation in several regions, including nociceptive circuits in the spinal cord. Here, we review the role played by NO on central pain sensitization.
abstract_unstemmed	Nitric oxide (NO) is a versatile messenger molecule first associated with endothelial relaxing effects. In the central nervous system (CNS), NO synthesis is primarily triggered by activation of N-methyl-D-aspartate (NMDA) receptors and has a Janus face, with both beneficial and harmful properties, depending on concentration and the identity of its synthetic enzyme isoform. There are three isoforms of the NO synthesizing enzyme nitric oxide synthase (NOS): neuronal (nNOS), endothelial (eNOS), and inducible nitric oxide synthase (iNOS), each one involved with specific events in the brain. In CNS, nNOS is involved with modulation of synaptic transmission through long-term potentiation in several regions, including nociceptive circuits in the spinal cord. Here, we review the role played by NO on central pain sensitization.
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title_short	Pain modulation by nitric oxide in the spinal cord.
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