p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
Ageing normal oesophagus epithelium contains p53 mutant clones. Here the authors use transgenic mice to show how these clones form and contribute to cancer development.
Autor*in: |
Kasumi Murai [verfasserIn] Stefan Dentro [verfasserIn] Swee Hoe Ong [verfasserIn] Roshan Sood [verfasserIn] David Fernandez-Antoran [verfasserIn] Albert Herms [verfasserIn] Vasiliki Kostiou [verfasserIn] Irina Abnizova [verfasserIn] Benjamin A. Hall [verfasserIn] Moritz Gerstung [verfasserIn] Philip H. Jones [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2022 |
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Übergeordnetes Werk: |
In: Nature Communications - Nature Portfolio, 2016, 13(2022), 1, Seite 17 |
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Übergeordnetes Werk: |
volume:13 ; year:2022 ; number:1 ; pages:17 |
Links: |
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DOI / URN: |
10.1038/s41467-022-33945-y |
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Katalog-ID: |
DOAJ021952647 |
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p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition |
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Ageing normal oesophagus epithelium contains p53 mutant clones. Here the authors use transgenic mice to show how these clones form and contribute to cancer development. |
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