p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition

Ageing normal oesophagus epithelium contains p53 mutant clones. Here the authors use transgenic mice to show how these clones form and contribute to cancer development.

Gespeichert in:

Autor*in:	Kasumi Murai [verfasserIn] Stefan Dentro [verfasserIn] Swee Hoe Ong [verfasserIn] Roshan Sood [verfasserIn] David Fernandez-Antoran [verfasserIn] Albert Herms [verfasserIn] Vasiliki Kostiou [verfasserIn] Irina Abnizova [verfasserIn] Benjamin A. Hall [verfasserIn] Moritz Gerstung [verfasserIn] Philip H. Jones [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2022

Übergeordnetes Werk:	In: Nature Communications - Nature Portfolio, 2016, 13(2022), 1, Seite 17
Übergeordnetes Werk:	volume:13 ; year:2022 ; number:1 ; pages:17

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.1038/s41467-022-33945-y

Katalog-ID:	DOAJ021952647

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author	Kasumi Murai
spellingShingle	Kasumi Murai misc Science misc Q p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
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topic_title	p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
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title	p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
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title_full	p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
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author_browse	Kasumi Murai Stefan Dentro Swee Hoe Ong Roshan Sood David Fernandez-Antoran Albert Herms Vasiliki Kostiou Irina Abnizova Benjamin A. Hall Moritz Gerstung Philip H. Jones
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title_sort	p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
title_auth	p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
abstract	Ageing normal oesophagus epithelium contains p53 mutant clones. Here the authors use transgenic mice to show how these clones form and contribute to cancer development.
abstractGer	Ageing normal oesophagus epithelium contains p53 mutant clones. Here the authors use transgenic mice to show how these clones form and contribute to cancer development.
abstract_unstemmed	Ageing normal oesophagus epithelium contains p53 mutant clones. Here the authors use transgenic mice to show how these clones form and contribute to cancer development.
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title_short	p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition
url	https://doi.org/10.1038/s41467-022-33945-y https://doaj.org/article/1dd89c9fd24a483699e9a724ff044c1c https://doaj.org/toc/2041-1723
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