Ginsenoside and Its Therapeutic Potential for Cognitive Impairment

Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its en...
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Autor*in:	Hui Feng [verfasserIn] Mei Xue [verfasserIn] Hao Deng [verfasserIn] Shiqi Cheng [verfasserIn] Yue Hu [verfasserIn] Chunxiang Zhou [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2022

Schlagwörter:	ginsenosides cognitive impairment pharmacological properties apoptosis inflammation

Übergeordnetes Werk:	In: Biomolecules - MDPI AG, 2013, 12(2022), 9, p 1310
Übergeordnetes Werk:	volume:12 ; year:2022 ; number:9, p 1310

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.3390/biom12091310

Katalog-ID:	DOAJ034094245

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520			\|a Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides.
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allfields	10.3390/biom12091310 doi (DE-627)DOAJ034094245 (DE-599)DOAJ66fb92e8041f4cf292974576451f18d0 DE-627 ger DE-627 rakwb eng QR1-502 Hui Feng verfasserin aut Ginsenoside and Its Therapeutic Potential for Cognitive Impairment 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides. ginsenosides cognitive impairment pharmacological properties apoptosis inflammation Microbiology Mei Xue verfasserin aut Hao Deng verfasserin aut Shiqi Cheng verfasserin aut Yue Hu verfasserin aut Chunxiang Zhou verfasserin aut In Biomolecules MDPI AG, 2013 12(2022), 9, p 1310 (DE-627)735688915 (DE-600)2701262-1 2218273X nnns volume:12 year:2022 number:9, p 1310 https://doi.org/10.3390/biom12091310 kostenfrei https://doaj.org/article/66fb92e8041f4cf292974576451f18d0 kostenfrei https://www.mdpi.com/2218-273X/12/9/1310 kostenfrei https://doaj.org/toc/2218-273X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2022 9, p 1310
spelling	10.3390/biom12091310 doi (DE-627)DOAJ034094245 (DE-599)DOAJ66fb92e8041f4cf292974576451f18d0 DE-627 ger DE-627 rakwb eng QR1-502 Hui Feng verfasserin aut Ginsenoside and Its Therapeutic Potential for Cognitive Impairment 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides. ginsenosides cognitive impairment pharmacological properties apoptosis inflammation Microbiology Mei Xue verfasserin aut Hao Deng verfasserin aut Shiqi Cheng verfasserin aut Yue Hu verfasserin aut Chunxiang Zhou verfasserin aut In Biomolecules MDPI AG, 2013 12(2022), 9, p 1310 (DE-627)735688915 (DE-600)2701262-1 2218273X nnns volume:12 year:2022 number:9, p 1310 https://doi.org/10.3390/biom12091310 kostenfrei https://doaj.org/article/66fb92e8041f4cf292974576451f18d0 kostenfrei https://www.mdpi.com/2218-273X/12/9/1310 kostenfrei https://doaj.org/toc/2218-273X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2022 9, p 1310
allfields_unstemmed	10.3390/biom12091310 doi (DE-627)DOAJ034094245 (DE-599)DOAJ66fb92e8041f4cf292974576451f18d0 DE-627 ger DE-627 rakwb eng QR1-502 Hui Feng verfasserin aut Ginsenoside and Its Therapeutic Potential for Cognitive Impairment 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides. ginsenosides cognitive impairment pharmacological properties apoptosis inflammation Microbiology Mei Xue verfasserin aut Hao Deng verfasserin aut Shiqi Cheng verfasserin aut Yue Hu verfasserin aut Chunxiang Zhou verfasserin aut In Biomolecules MDPI AG, 2013 12(2022), 9, p 1310 (DE-627)735688915 (DE-600)2701262-1 2218273X nnns volume:12 year:2022 number:9, p 1310 https://doi.org/10.3390/biom12091310 kostenfrei https://doaj.org/article/66fb92e8041f4cf292974576451f18d0 kostenfrei https://www.mdpi.com/2218-273X/12/9/1310 kostenfrei https://doaj.org/toc/2218-273X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2022 9, p 1310
allfieldsGer	10.3390/biom12091310 doi (DE-627)DOAJ034094245 (DE-599)DOAJ66fb92e8041f4cf292974576451f18d0 DE-627 ger DE-627 rakwb eng QR1-502 Hui Feng verfasserin aut Ginsenoside and Its Therapeutic Potential for Cognitive Impairment 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides. ginsenosides cognitive impairment pharmacological properties apoptosis inflammation Microbiology Mei Xue verfasserin aut Hao Deng verfasserin aut Shiqi Cheng verfasserin aut Yue Hu verfasserin aut Chunxiang Zhou verfasserin aut In Biomolecules MDPI AG, 2013 12(2022), 9, p 1310 (DE-627)735688915 (DE-600)2701262-1 2218273X nnns volume:12 year:2022 number:9, p 1310 https://doi.org/10.3390/biom12091310 kostenfrei https://doaj.org/article/66fb92e8041f4cf292974576451f18d0 kostenfrei https://www.mdpi.com/2218-273X/12/9/1310 kostenfrei https://doaj.org/toc/2218-273X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2022 9, p 1310
allfieldsSound	10.3390/biom12091310 doi (DE-627)DOAJ034094245 (DE-599)DOAJ66fb92e8041f4cf292974576451f18d0 DE-627 ger DE-627 rakwb eng QR1-502 Hui Feng verfasserin aut Ginsenoside and Its Therapeutic Potential for Cognitive Impairment 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides. ginsenosides cognitive impairment pharmacological properties apoptosis inflammation Microbiology Mei Xue verfasserin aut Hao Deng verfasserin aut Shiqi Cheng verfasserin aut Yue Hu verfasserin aut Chunxiang Zhou verfasserin aut In Biomolecules MDPI AG, 2013 12(2022), 9, p 1310 (DE-627)735688915 (DE-600)2701262-1 2218273X nnns volume:12 year:2022 number:9, p 1310 https://doi.org/10.3390/biom12091310 kostenfrei https://doaj.org/article/66fb92e8041f4cf292974576451f18d0 kostenfrei https://www.mdpi.com/2218-273X/12/9/1310 kostenfrei https://doaj.org/toc/2218-273X Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2022 9, p 1310
language	English
source	In Biomolecules 12(2022), 9, p 1310 volume:12 year:2022 number:9, p 1310
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topic_facet	ginsenosides cognitive impairment pharmacological properties apoptosis inflammation Microbiology
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author	Hui Feng
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topic	misc QR1-502 misc ginsenosides misc cognitive impairment misc pharmacological properties misc apoptosis misc inflammation misc Microbiology
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title	Ginsenoside and Its Therapeutic Potential for Cognitive Impairment
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title_auth	Ginsenoside and Its Therapeutic Potential for Cognitive Impairment
abstract	Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides.
abstractGer	Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides.
abstract_unstemmed	Cognitive impairment (CI) is one of the major clinical features of many neurodegenerative diseases. It can be aging-related or even appear in non-central nerve system (CNS) diseases. CI has a wide spectrum that ranges from the cognitive complaint with normal screening tests to mild CI and, at its end, dementia. Ginsenosides, agents extracted from a key Chinese herbal medicine (ginseng), show great promise as a new therapeutic option for treating CI. This review covered both clinical trials and preclinical studies to summarize the possible mechanisms of how ginsenosides affect CI in different diseases. It shows that ginsenosides can modulate signaling pathways associated with oxidative stress, apoptosis, inflammation, synaptic plasticity, and neurogenesis. The involved signaling pathways mainly include the PI3K/Akt, CREB/BDNF, Keap1/Nrf2 signaling, and NF-κB/NLRP3 inflammasome pathways. We hope to provide a theoretical basis for the treatment of CI for related diseases by ginsenosides.
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title_short	Ginsenoside and Its Therapeutic Potential for Cognitive Impairment
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