TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC
Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting im...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
DANILA Tatiana [verfasserIn] MUNTEANU Mihaela [verfasserIn] CIOBANU Lucia [verfasserIn] POPOVICI Mihail [verfasserIn] COBEȚ VALERIU [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch ; Französisch ; Rumänisch ; Russisch |
| Erschienen: |
2022 |
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| Schlagwörter: |
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| Übergeordnetes Werk: |
In: Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale - Academy of Sciences of Moldova, 2022, 72(2022), 1, Seite 97-105 |
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| Übergeordnetes Werk: |
volume:72 ; year:2022 ; number:1 ; pages:97-105 |
| Links: |
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| DOI / URN: |
10.52692/1857-0011.2022.1-72.15 |
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| Katalog-ID: |
DOAJ043397182 |
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| 520 | |a Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. | ||
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10.52692/1857-0011.2022.1-72.15 doi (DE-627)DOAJ043397182 (DE-599)DOAJ8566fc5b92234c0bb701f40633b9812c DE-627 ger DE-627 rakwb eng fre rum rus R5-920 RC31-1245 RZ201-999 RA1-1270 DANILA Tatiana verfasserin aut TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. nstemi dereglarea hemostazei markeri procoagulanți markerii ai sistemului antiocoagulant markeri ai fibrinolizei Medicine (General) Internal medicine Other systems of medicine Public aspects of medicine MUNTEANU Mihaela verfasserin aut CIOBANU Lucia verfasserin aut POPOVICI Mihail verfasserin aut COBEȚ VALERIU verfasserin aut In Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale Academy of Sciences of Moldova, 2022 72(2022), 1, Seite 97-105 (DE-627)DOAJ078602351 18570011 nnns volume:72 year:2022 number:1 pages:97-105 https://doi.org/10.52692/1857-0011.2022.1-72.15 kostenfrei https://doaj.org/article/8566fc5b92234c0bb701f40633b9812c kostenfrei https://bulmed.md/bulmed/article/view/3409 kostenfrei https://doaj.org/toc/1857-0011 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ AR 72 2022 1 97-105 |
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10.52692/1857-0011.2022.1-72.15 doi (DE-627)DOAJ043397182 (DE-599)DOAJ8566fc5b92234c0bb701f40633b9812c DE-627 ger DE-627 rakwb eng fre rum rus R5-920 RC31-1245 RZ201-999 RA1-1270 DANILA Tatiana verfasserin aut TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. nstemi dereglarea hemostazei markeri procoagulanți markerii ai sistemului antiocoagulant markeri ai fibrinolizei Medicine (General) Internal medicine Other systems of medicine Public aspects of medicine MUNTEANU Mihaela verfasserin aut CIOBANU Lucia verfasserin aut POPOVICI Mihail verfasserin aut COBEȚ VALERIU verfasserin aut In Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale Academy of Sciences of Moldova, 2022 72(2022), 1, Seite 97-105 (DE-627)DOAJ078602351 18570011 nnns volume:72 year:2022 number:1 pages:97-105 https://doi.org/10.52692/1857-0011.2022.1-72.15 kostenfrei https://doaj.org/article/8566fc5b92234c0bb701f40633b9812c kostenfrei https://bulmed.md/bulmed/article/view/3409 kostenfrei https://doaj.org/toc/1857-0011 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ AR 72 2022 1 97-105 |
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10.52692/1857-0011.2022.1-72.15 doi (DE-627)DOAJ043397182 (DE-599)DOAJ8566fc5b92234c0bb701f40633b9812c DE-627 ger DE-627 rakwb eng fre rum rus R5-920 RC31-1245 RZ201-999 RA1-1270 DANILA Tatiana verfasserin aut TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. nstemi dereglarea hemostazei markeri procoagulanți markerii ai sistemului antiocoagulant markeri ai fibrinolizei Medicine (General) Internal medicine Other systems of medicine Public aspects of medicine MUNTEANU Mihaela verfasserin aut CIOBANU Lucia verfasserin aut POPOVICI Mihail verfasserin aut COBEȚ VALERIU verfasserin aut In Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale Academy of Sciences of Moldova, 2022 72(2022), 1, Seite 97-105 (DE-627)DOAJ078602351 18570011 nnns volume:72 year:2022 number:1 pages:97-105 https://doi.org/10.52692/1857-0011.2022.1-72.15 kostenfrei https://doaj.org/article/8566fc5b92234c0bb701f40633b9812c kostenfrei https://bulmed.md/bulmed/article/view/3409 kostenfrei https://doaj.org/toc/1857-0011 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ AR 72 2022 1 97-105 |
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10.52692/1857-0011.2022.1-72.15 doi (DE-627)DOAJ043397182 (DE-599)DOAJ8566fc5b92234c0bb701f40633b9812c DE-627 ger DE-627 rakwb eng fre rum rus R5-920 RC31-1245 RZ201-999 RA1-1270 DANILA Tatiana verfasserin aut TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. nstemi dereglarea hemostazei markeri procoagulanți markerii ai sistemului antiocoagulant markeri ai fibrinolizei Medicine (General) Internal medicine Other systems of medicine Public aspects of medicine MUNTEANU Mihaela verfasserin aut CIOBANU Lucia verfasserin aut POPOVICI Mihail verfasserin aut COBEȚ VALERIU verfasserin aut In Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale Academy of Sciences of Moldova, 2022 72(2022), 1, Seite 97-105 (DE-627)DOAJ078602351 18570011 nnns volume:72 year:2022 number:1 pages:97-105 https://doi.org/10.52692/1857-0011.2022.1-72.15 kostenfrei https://doaj.org/article/8566fc5b92234c0bb701f40633b9812c kostenfrei https://bulmed.md/bulmed/article/view/3409 kostenfrei https://doaj.org/toc/1857-0011 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ AR 72 2022 1 97-105 |
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10.52692/1857-0011.2022.1-72.15 doi (DE-627)DOAJ043397182 (DE-599)DOAJ8566fc5b92234c0bb701f40633b9812c DE-627 ger DE-627 rakwb eng fre rum rus R5-920 RC31-1245 RZ201-999 RA1-1270 DANILA Tatiana verfasserin aut TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. nstemi dereglarea hemostazei markeri procoagulanți markerii ai sistemului antiocoagulant markeri ai fibrinolizei Medicine (General) Internal medicine Other systems of medicine Public aspects of medicine MUNTEANU Mihaela verfasserin aut CIOBANU Lucia verfasserin aut POPOVICI Mihail verfasserin aut COBEȚ VALERIU verfasserin aut In Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale Academy of Sciences of Moldova, 2022 72(2022), 1, Seite 97-105 (DE-627)DOAJ078602351 18570011 nnns volume:72 year:2022 number:1 pages:97-105 https://doi.org/10.52692/1857-0011.2022.1-72.15 kostenfrei https://doaj.org/article/8566fc5b92234c0bb701f40633b9812c kostenfrei https://bulmed.md/bulmed/article/view/3409 kostenfrei https://doaj.org/toc/1857-0011 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ AR 72 2022 1 97-105 |
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Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">nstemi</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">dereglarea hemostazei</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">markeri procoagulanți</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">markerii ai sistemului antiocoagulant</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">markeri ai fibrinolizei</subfield></datafield><datafield tag="653" ind1=" " ind2="0"><subfield code="a">Medicine (General)</subfield></datafield><datafield tag="653" ind1=" " ind2="0"><subfield code="a">Internal medicine</subfield></datafield><datafield tag="653" ind1=" " ind2="0"><subfield code="a">Other systems of medicine</subfield></datafield><datafield tag="653" ind1=" " ind2="0"><subfield code="a">Public aspects of medicine</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">MUNTEANU Mihaela</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">CIOBANU Lucia</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">POPOVICI Mihail</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">COBEȚ VALERIU</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">In</subfield><subfield code="t">Buletinul Academiei de Ştiinţe a Moldovei: Ştiinţe Medicale</subfield><subfield code="d">Academy of Sciences of Moldova, 2022</subfield><subfield code="g">72(2022), 1, Seite 97-105</subfield><subfield code="w">(DE-627)DOAJ078602351</subfield><subfield code="x">18570011</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:72</subfield><subfield code="g">year:2022</subfield><subfield code="g">number:1</subfield><subfield code="g">pages:97-105</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.52692/1857-0011.2022.1-72.15</subfield><subfield code="z">kostenfrei</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doaj.org/article/8566fc5b92234c0bb701f40633b9812c</subfield><subfield code="z">kostenfrei</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://bulmed.md/bulmed/article/view/3409</subfield><subfield code="z">kostenfrei</subfield></datafield><datafield tag="856" ind1="4" ind2="2"><subfield code="u">https://doaj.org/toc/1857-0011</subfield><subfield code="y">Journal toc</subfield><subfield code="z">kostenfrei</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_DOAJ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">72</subfield><subfield code="j">2022</subfield><subfield code="e">1</subfield><subfield code="h">97-105</subfield></datafield></record></collection>
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TULBURĂRILE HEMOSTAZEI LA PACIENȚII CU NSTEMI ȘI MARKERII DE DIAGNOSTIC |
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Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. |
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Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. |
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Background. Hemostasis disorder is an important mechanism of cardiovascular disease, and its pathogenetic contribution to the evolution of NSTEMI is particularly notable in the context of the significance of the pathophysiological pattern caused by coronary microcirculation disorder. Highlighting imminent markers will facilitate the diagnosis and prognosis of NSTEMI. Object. Reporting literature summary data on markers with definite predictive value on proven hemostasis disorder in patients with NSTEMI. Results. The main markers of hemostasis disorder in NSTEMI and with predictive value on the diagnosis and evolution of post-infarction are:opposite the procoagulant system: elevation of microparticles derived from endotheliocytes and platelets expressing phosphotidylserine, activation of the complex of coagulation factors II, VII and X, formation of aggregates between platelets and leukocytes;opposite the anticoagulant system: increase in soluble thrombomodulin in combination with plasma decrease in antithrombin and C and S proteins;opposite the fibrinolytic system: elevation of the plasma levels of fibrin monomers and D-dimers of the plasminogen activator inhibitor in association with quantitative impairment of the plasminogen activator. |
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