Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice

Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Nava...
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Autor*in:	Miguel Hueso [verfasserIn] Laura De Ramon [verfasserIn] Estanislao Navarro [verfasserIn] Elia Ripoll [verfasserIn] Josep M. Cruzado [verfasserIn] Josep M. Grinyo [verfasserIn] Joan Torras [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2016

Schlagwörter:	CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr

Übergeordnetes Werk:	In: Data in Brief - Elsevier, 2015, 9(2016), C, Seite 1105-1112
Übergeordnetes Werk:	volume:9 ; year:2016 ; number:C ; pages:1105-1112

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.1016/j.dib.2016.11.045

Katalog-ID:	DOAJ046548246

Internformat


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700	0		\|a Josep M. Grinyo \|e verfasserin \|4 aut
700	0		\|a Joan Torras \|e verfasserin \|4 aut
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allfields	10.1016/j.dib.2016.11.045 doi (DE-627)DOAJ046548246 (DE-599)DOAJ035470eff9b3476683396c75ec8c0f67 DE-627 ger DE-627 rakwb eng R858-859.7 Q1-390 Miguel Hueso verfasserin aut Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing. CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr Computer applications to medicine. Medical informatics Science (General) Laura De Ramon verfasserin aut Estanislao Navarro verfasserin aut Elia Ripoll verfasserin aut Josep M. Cruzado verfasserin aut Josep M. Grinyo verfasserin aut Joan Torras verfasserin aut In Data in Brief Elsevier, 2015 9(2016), C, Seite 1105-1112 (DE-627)797838937 (DE-600)2786545-9 23523409 nnns volume:9 year:2016 number:C pages:1105-1112 https://doi.org/10.1016/j.dib.2016.11.045 kostenfrei https://doaj.org/article/035470eff9b3476683396c75ec8c0f67 kostenfrei http://www.sciencedirect.com/science/article/pii/S2352340916307053 kostenfrei https://doaj.org/toc/2352-3409 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4393 GBV_ILN_4700 AR 9 2016 C 1105-1112
spelling	10.1016/j.dib.2016.11.045 doi (DE-627)DOAJ046548246 (DE-599)DOAJ035470eff9b3476683396c75ec8c0f67 DE-627 ger DE-627 rakwb eng R858-859.7 Q1-390 Miguel Hueso verfasserin aut Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing. CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr Computer applications to medicine. Medical informatics Science (General) Laura De Ramon verfasserin aut Estanislao Navarro verfasserin aut Elia Ripoll verfasserin aut Josep M. Cruzado verfasserin aut Josep M. Grinyo verfasserin aut Joan Torras verfasserin aut In Data in Brief Elsevier, 2015 9(2016), C, Seite 1105-1112 (DE-627)797838937 (DE-600)2786545-9 23523409 nnns volume:9 year:2016 number:C pages:1105-1112 https://doi.org/10.1016/j.dib.2016.11.045 kostenfrei https://doaj.org/article/035470eff9b3476683396c75ec8c0f67 kostenfrei http://www.sciencedirect.com/science/article/pii/S2352340916307053 kostenfrei https://doaj.org/toc/2352-3409 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4393 GBV_ILN_4700 AR 9 2016 C 1105-1112
allfields_unstemmed	10.1016/j.dib.2016.11.045 doi (DE-627)DOAJ046548246 (DE-599)DOAJ035470eff9b3476683396c75ec8c0f67 DE-627 ger DE-627 rakwb eng R858-859.7 Q1-390 Miguel Hueso verfasserin aut Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing. CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr Computer applications to medicine. Medical informatics Science (General) Laura De Ramon verfasserin aut Estanislao Navarro verfasserin aut Elia Ripoll verfasserin aut Josep M. Cruzado verfasserin aut Josep M. Grinyo verfasserin aut Joan Torras verfasserin aut In Data in Brief Elsevier, 2015 9(2016), C, Seite 1105-1112 (DE-627)797838937 (DE-600)2786545-9 23523409 nnns volume:9 year:2016 number:C pages:1105-1112 https://doi.org/10.1016/j.dib.2016.11.045 kostenfrei https://doaj.org/article/035470eff9b3476683396c75ec8c0f67 kostenfrei http://www.sciencedirect.com/science/article/pii/S2352340916307053 kostenfrei https://doaj.org/toc/2352-3409 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4393 GBV_ILN_4700 AR 9 2016 C 1105-1112
allfieldsGer	10.1016/j.dib.2016.11.045 doi (DE-627)DOAJ046548246 (DE-599)DOAJ035470eff9b3476683396c75ec8c0f67 DE-627 ger DE-627 rakwb eng R858-859.7 Q1-390 Miguel Hueso verfasserin aut Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing. CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr Computer applications to medicine. Medical informatics Science (General) Laura De Ramon verfasserin aut Estanislao Navarro verfasserin aut Elia Ripoll verfasserin aut Josep M. Cruzado verfasserin aut Josep M. Grinyo verfasserin aut Joan Torras verfasserin aut In Data in Brief Elsevier, 2015 9(2016), C, Seite 1105-1112 (DE-627)797838937 (DE-600)2786545-9 23523409 nnns volume:9 year:2016 number:C pages:1105-1112 https://doi.org/10.1016/j.dib.2016.11.045 kostenfrei https://doaj.org/article/035470eff9b3476683396c75ec8c0f67 kostenfrei http://www.sciencedirect.com/science/article/pii/S2352340916307053 kostenfrei https://doaj.org/toc/2352-3409 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4393 GBV_ILN_4700 AR 9 2016 C 1105-1112
allfieldsSound	10.1016/j.dib.2016.11.045 doi (DE-627)DOAJ046548246 (DE-599)DOAJ035470eff9b3476683396c75ec8c0f67 DE-627 ger DE-627 rakwb eng R858-859.7 Q1-390 Miguel Hueso verfasserin aut Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing. CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr Computer applications to medicine. Medical informatics Science (General) Laura De Ramon verfasserin aut Estanislao Navarro verfasserin aut Elia Ripoll verfasserin aut Josep M. Cruzado verfasserin aut Josep M. Grinyo verfasserin aut Joan Torras verfasserin aut In Data in Brief Elsevier, 2015 9(2016), C, Seite 1105-1112 (DE-627)797838937 (DE-600)2786545-9 23523409 nnns volume:9 year:2016 number:C pages:1105-1112 https://doi.org/10.1016/j.dib.2016.11.045 kostenfrei https://doaj.org/article/035470eff9b3476683396c75ec8c0f67 kostenfrei http://www.sciencedirect.com/science/article/pii/S2352340916307053 kostenfrei https://doaj.org/toc/2352-3409 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4393 GBV_ILN_4700 AR 9 2016 C 1105-1112
language	English
source	In Data in Brief 9(2016), C, Seite 1105-1112 volume:9 year:2016 number:C pages:1105-1112
sourceStr	In Data in Brief 9(2016), C, Seite 1105-1112 volume:9 year:2016 number:C pages:1105-1112
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr Computer applications to medicine. Medical informatics Science (General)
isfreeaccess_bool	true
container_title	Data in Brief
authorswithroles_txt_mv	Miguel Hueso @@aut@@ Laura De Ramon @@aut@@ Estanislao Navarro @@aut@@ Elia Ripoll @@aut@@ Josep M. Cruzado @@aut@@ Josep M. Grinyo @@aut@@ Joan Torras @@aut@@
publishDateDaySort_date	2016-01-01T00:00:00Z
hierarchy_top_id	797838937
id	DOAJ046548246
language_de	englisch
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callnumber-first	R - Medicine
author	Miguel Hueso
spellingShingle	Miguel Hueso misc R858-859.7 misc Q1-390 misc CD40 misc siRNA misc Macrophages misc Atherosclerosis misc GO analysis misc NF-kB misc miR-125b misc Clec/Klr misc Computer applications to medicine. Medical informatics misc Science (General) Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice
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topic_title	R858-859.7 Q1-390 Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice CD40 siRNA Macrophages Atherosclerosis GO analysis NF-kB miR-125b Clec/Klr
topic	misc R858-859.7 misc Q1-390 misc CD40 misc siRNA misc Macrophages misc Atherosclerosis misc GO analysis misc NF-kB misc miR-125b misc Clec/Klr misc Computer applications to medicine. Medical informatics misc Science (General)
topic_unstemmed	misc R858-859.7 misc Q1-390 misc CD40 misc siRNA misc Macrophages misc Atherosclerosis misc GO analysis misc NF-kB misc miR-125b misc Clec/Klr misc Computer applications to medicine. Medical informatics misc Science (General)
topic_browse	misc R858-859.7 misc Q1-390 misc CD40 misc siRNA misc Macrophages misc Atherosclerosis misc GO analysis misc NF-kB misc miR-125b misc Clec/Klr misc Computer applications to medicine. Medical informatics misc Science (General)
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title	Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice
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title_full	Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice
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author_browse	Miguel Hueso Laura De Ramon Estanislao Navarro Elia Ripoll Josep M. Cruzado Josep M. Grinyo Joan Torras
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title_sort	datasets for the validation of the "in vivo" sirna-silencing of cd40 and for the detection of new markers of atherosclerosis progression in apoe-deficient mice
callnumber	R858-859.7
title_auth	Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice
abstract	Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing.
abstractGer	Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing.
abstract_unstemmed	Data presented in this Data in Brief article correspond to the article "in vivo" silencing of CD40 reduces progression of experimental atherogenesis through a NFκB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis" (M. Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. We provide the scientific community with a set of genes whose expression is increased during atherosclerosis progression but downregulated upon CD40 silencing.
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title_short	Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice
url	https://doi.org/10.1016/j.dib.2016.11.045 https://doaj.org/article/035470eff9b3476683396c75ec8c0f67 http://www.sciencedirect.com/science/article/pii/S2352340916307053 https://doaj.org/toc/2352-3409
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up_date	2024-07-03T21:16:15.648Z
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Hueso, L. De Ramon, E. Navarro, E. Ripoll, J.M. Cruzado, J.M. Grinyo, J. Torras, 2016) [1]. Here, we describe the validation of the silencing of CD40 expression with a specific siRNA in ApoE−/− mouse aortas, and its systemic effects on splenic lymphocytic subpopulations as well as on the infiltration of aortic intima by F4/80+, galectin-3+ macrophages or by NF-κB+ cells. We also show the output of a Gene Ontology and TLDA analysis which allowed the detection of potential mediators of atherosclerosis progression. 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Datasets for the validation of the "in vivo" siRNA-silencing of CD40 and for the detection of new markers of atherosclerosis progression in ApoE-deficient mice

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