Cardiac Amyloidosis Therapy: A Systematic Review
Heart involvement in Cardiac Amyloidosis (CA) results in a worsening of the prognosis in almost all patients with both light-chain (AL) and transthyretin amyloidosis (ATTR). The mainstream CA is a restrictive cardiomyopathy with hypertrophic phenotype at cardiac imaging that clinically leads to hear...
Ausführliche Beschreibung
Autor*in: |
Franco Iodice [verfasserIn] Marco Di Mauro [verfasserIn] Marco Giuseppe Migliaccio [verfasserIn] Angela Iannuzzi [verfasserIn] Roberta Pacileo [verfasserIn] Martina Caiazza [verfasserIn] Augusto Esposito [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2021 |
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Schlagwörter: |
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Übergeordnetes Werk: |
In: Cardiogenetics - MDPI AG, 2011, 11(2021), 1, Seite 10-17 |
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Übergeordnetes Werk: |
volume:11 ; year:2021 ; number:1 ; pages:10-17 |
Links: |
Link aufrufen |
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DOI / URN: |
10.3390/cardiogenetics11010002 |
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Katalog-ID: |
DOAJ050254189 |
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10.3390/cardiogenetics11010002 doi (DE-627)DOAJ050254189 (DE-599)DOAJc5b40b8f444c48a98585bf141cc68f63 DE-627 ger DE-627 rakwb eng RC666-701 Franco Iodice verfasserin aut Cardiac Amyloidosis Therapy: A Systematic Review 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Heart involvement in Cardiac Amyloidosis (CA) results in a worsening of the prognosis in almost all patients with both light-chain (AL) and transthyretin amyloidosis (ATTR). The mainstream CA is a restrictive cardiomyopathy with hypertrophic phenotype at cardiac imaging that clinically leads to heart failure with preserved ejection fraction (HFpEF). An early diagnosis is essential to reduce cardiac damage and to improve the prognosis. Many therapies are available, but most of them have late benefits to cardiac function; for this reason, novel therapies are going to come soon. cardiac amyloidosis therapies amyloidosis Diseases of the circulatory (Cardiovascular) system Marco Di Mauro verfasserin aut Marco Giuseppe Migliaccio verfasserin aut Angela Iannuzzi verfasserin aut Roberta Pacileo verfasserin aut Martina Caiazza verfasserin aut Augusto Esposito verfasserin aut In Cardiogenetics MDPI AG, 2011 11(2021), 1, Seite 10-17 (DE-627)668713992 (DE-600)2628818-7 20358148 nnns volume:11 year:2021 number:1 pages:10-17 https://doi.org/10.3390/cardiogenetics11010002 kostenfrei https://doaj.org/article/c5b40b8f444c48a98585bf141cc68f63 kostenfrei https://www.mdpi.com/2035-8148/11/1/2 kostenfrei https://doaj.org/toc/2035-8253 Journal toc kostenfrei https://doaj.org/toc/2035-8148 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2031 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2061 GBV_ILN_2111 GBV_ILN_2113 GBV_ILN_2190 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 11 2021 1 10-17 |
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Heart involvement in Cardiac Amyloidosis (CA) results in a worsening of the prognosis in almost all patients with both light-chain (AL) and transthyretin amyloidosis (ATTR). The mainstream CA is a restrictive cardiomyopathy with hypertrophic phenotype at cardiac imaging that clinically leads to heart failure with preserved ejection fraction (HFpEF). An early diagnosis is essential to reduce cardiac damage and to improve the prognosis. Many therapies are available, but most of them have late benefits to cardiac function; for this reason, novel therapies are going to come soon. |
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Heart involvement in Cardiac Amyloidosis (CA) results in a worsening of the prognosis in almost all patients with both light-chain (AL) and transthyretin amyloidosis (ATTR). The mainstream CA is a restrictive cardiomyopathy with hypertrophic phenotype at cardiac imaging that clinically leads to heart failure with preserved ejection fraction (HFpEF). An early diagnosis is essential to reduce cardiac damage and to improve the prognosis. Many therapies are available, but most of them have late benefits to cardiac function; for this reason, novel therapies are going to come soon. |
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Heart involvement in Cardiac Amyloidosis (CA) results in a worsening of the prognosis in almost all patients with both light-chain (AL) and transthyretin amyloidosis (ATTR). The mainstream CA is a restrictive cardiomyopathy with hypertrophic phenotype at cardiac imaging that clinically leads to heart failure with preserved ejection fraction (HFpEF). An early diagnosis is essential to reduce cardiac damage and to improve the prognosis. Many therapies are available, but most of them have late benefits to cardiac function; for this reason, novel therapies are going to come soon. |
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