Appetite Regulation of TLR4-Induced Inflammatory Signaling

Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite,...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Yongxiang Li [verfasserIn] Qingyan Jiang [verfasserIn] Lina Wang [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2021

Schlagwörter:	TLR4 inflammation hypothalamus dopamine system SFAs

Übergeordnetes Werk:	In: Frontiers in Endocrinology - Frontiers Media S.A., 2011, 12(2021)
Übergeordnetes Werk:	volume:12 ; year:2021

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.3389/fendo.2021.777997

Katalog-ID:	DOAJ062944185

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520			\|a Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions.
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spelling	10.3389/fendo.2021.777997 doi (DE-627)DOAJ062944185 (DE-599)DOAJdb856b9176084a1aa63bd6ade7f44188 DE-627 ger DE-627 rakwb eng RC648-665 Yongxiang Li verfasserin aut Appetite Regulation of TLR4-Induced Inflammatory Signaling 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions. TLR4 inflammation hypothalamus dopamine system SFAs Diseases of the endocrine glands. Clinical endocrinology Yongxiang Li verfasserin aut Yongxiang Li verfasserin aut Qingyan Jiang verfasserin aut Qingyan Jiang verfasserin aut Lina Wang verfasserin aut Lina Wang verfasserin aut In Frontiers in Endocrinology Frontiers Media S.A., 2011 12(2021) (DE-627)645090948 (DE-600)2592084-4 16642392 nnns volume:12 year:2021 https://doi.org/10.3389/fendo.2021.777997 kostenfrei https://doaj.org/article/db856b9176084a1aa63bd6ade7f44188 kostenfrei https://www.frontiersin.org/articles/10.3389/fendo.2021.777997/full kostenfrei https://doaj.org/toc/1664-2392 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2021
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allfieldsGer	10.3389/fendo.2021.777997 doi (DE-627)DOAJ062944185 (DE-599)DOAJdb856b9176084a1aa63bd6ade7f44188 DE-627 ger DE-627 rakwb eng RC648-665 Yongxiang Li verfasserin aut Appetite Regulation of TLR4-Induced Inflammatory Signaling 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions. TLR4 inflammation hypothalamus dopamine system SFAs Diseases of the endocrine glands. Clinical endocrinology Yongxiang Li verfasserin aut Yongxiang Li verfasserin aut Qingyan Jiang verfasserin aut Qingyan Jiang verfasserin aut Lina Wang verfasserin aut Lina Wang verfasserin aut In Frontiers in Endocrinology Frontiers Media S.A., 2011 12(2021) (DE-627)645090948 (DE-600)2592084-4 16642392 nnns volume:12 year:2021 https://doi.org/10.3389/fendo.2021.777997 kostenfrei https://doaj.org/article/db856b9176084a1aa63bd6ade7f44188 kostenfrei https://www.frontiersin.org/articles/10.3389/fendo.2021.777997/full kostenfrei https://doaj.org/toc/1664-2392 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2021
allfieldsSound	10.3389/fendo.2021.777997 doi (DE-627)DOAJ062944185 (DE-599)DOAJdb856b9176084a1aa63bd6ade7f44188 DE-627 ger DE-627 rakwb eng RC648-665 Yongxiang Li verfasserin aut Appetite Regulation of TLR4-Induced Inflammatory Signaling 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions. TLR4 inflammation hypothalamus dopamine system SFAs Diseases of the endocrine glands. Clinical endocrinology Yongxiang Li verfasserin aut Yongxiang Li verfasserin aut Qingyan Jiang verfasserin aut Qingyan Jiang verfasserin aut Lina Wang verfasserin aut Lina Wang verfasserin aut In Frontiers in Endocrinology Frontiers Media S.A., 2011 12(2021) (DE-627)645090948 (DE-600)2592084-4 16642392 nnns volume:12 year:2021 https://doi.org/10.3389/fendo.2021.777997 kostenfrei https://doaj.org/article/db856b9176084a1aa63bd6ade7f44188 kostenfrei https://www.frontiersin.org/articles/10.3389/fendo.2021.777997/full kostenfrei https://doaj.org/toc/1664-2392 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2021
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callnumber-first	R - Medicine
author	Yongxiang Li
spellingShingle	Yongxiang Li misc RC648-665 misc TLR4 misc inflammation misc hypothalamus misc dopamine system misc SFAs misc Diseases of the endocrine glands. Clinical endocrinology Appetite Regulation of TLR4-Induced Inflammatory Signaling
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topic_title	RC648-665 Appetite Regulation of TLR4-Induced Inflammatory Signaling TLR4 inflammation hypothalamus dopamine system SFAs
topic	misc RC648-665 misc TLR4 misc inflammation misc hypothalamus misc dopamine system misc SFAs misc Diseases of the endocrine glands. Clinical endocrinology
topic_unstemmed	misc RC648-665 misc TLR4 misc inflammation misc hypothalamus misc dopamine system misc SFAs misc Diseases of the endocrine glands. Clinical endocrinology
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title	Appetite Regulation of TLR4-Induced Inflammatory Signaling
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title_full	Appetite Regulation of TLR4-Induced Inflammatory Signaling
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title_sort	appetite regulation of tlr4-induced inflammatory signaling
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title_auth	Appetite Regulation of TLR4-Induced Inflammatory Signaling
abstract	Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions.
abstractGer	Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions.
abstract_unstemmed	Appetite is the basis for obtaining food and maintaining normal metabolism. Toll-like receptor 4 (TLR4) is an important receptor expressed in the brain that induces inflammatory signaling after activation. Inflammation is considered to affect the homeostatic and non-homeostatic systems of appetite, which are dominated by hypothalamic and mesolimbic dopamine signaling. Although the pathological features of many types of inflammation are known, their physiological functions in appetite are largely unknown. This review mainly addresses several key issues, including the structures of the homeostatic and non-homeostatic systems. In addition, the mechanism by which TLR4-induced inflammatory signaling contributes to these two systems to regulate appetite is also discussed. This review will provide potential opportunities to develop new therapeutic interventions that control appetite under inflammatory conditions.
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title_short	Appetite Regulation of TLR4-Induced Inflammatory Signaling
url	https://doi.org/10.3389/fendo.2021.777997 https://doaj.org/article/db856b9176084a1aa63bd6ade7f44188 https://www.frontiersin.org/articles/10.3389/fendo.2021.777997/full https://doaj.org/toc/1664-2392
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Appetite Regulation of TLR4-Induced Inflammatory Signaling

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