Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children

Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine...
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Autor*in:	Xiao-jun KUANG [verfasserIn] Ze-shu NING [verfasserIn] Li-ming YANG [verfasserIn] Bo CHEN [verfasserIn] Qin ZHANG [verfasserIn] Zhao WU [verfasserIn] Hong-mei LIAO [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch ; Chinesisch

Erschienen:	2018

Schlagwörter:	Movement disorders Electroencephalography Child

Übergeordnetes Werk:	In: Chinese Journal of Contemporary Neurology and Neurosurgery - Tianjin Huanhu Hospital, 2013, 18(2018), 1, Seite 60-64
Übergeordnetes Werk:	volume:18 ; year:2018 ; number:1 ; pages:60-64

Links:	Link aufrufen Link aufrufen Journal toc

Katalog-ID:	DOAJ074659103

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520			\|a Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010
650		4	\|a Movement disorders
650		4	\|a Electroencephalography
650		4	\|a Child
653		0	\|a Neurology. Diseases of the nervous system
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allfields	(DE-627)DOAJ074659103 (DE-599)DOAJ6313f06176074eceb83bf1daf73713b6 DE-627 ger DE-627 rakwb eng chi RC346-429 Xiao-jun KUANG verfasserin aut Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children 2018 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010 Movement disorders Electroencephalography Child Neurology. Diseases of the nervous system Ze-shu NING verfasserin aut Li-ming YANG verfasserin aut Bo CHEN verfasserin aut Qin ZHANG verfasserin aut Zhao WU verfasserin aut Hong-mei LIAO verfasserin aut In Chinese Journal of Contemporary Neurology and Neurosurgery Tianjin Huanhu Hospital, 2013 18(2018), 1, Seite 60-64 (DE-627)741171848 (DE-600)2710763-2 16726731 nnns volume:18 year:2018 number:1 pages:60-64 https://doaj.org/article/6313f06176074eceb83bf1daf73713b6 kostenfrei http://www.cjcnn.org/index.php/cjcnn/article/view/1722 kostenfrei https://doaj.org/toc/1672-6731 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2014 GBV_ILN_2106 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 18 2018 1 60-64
spelling	(DE-627)DOAJ074659103 (DE-599)DOAJ6313f06176074eceb83bf1daf73713b6 DE-627 ger DE-627 rakwb eng chi RC346-429 Xiao-jun KUANG verfasserin aut Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children 2018 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010 Movement disorders Electroencephalography Child Neurology. Diseases of the nervous system Ze-shu NING verfasserin aut Li-ming YANG verfasserin aut Bo CHEN verfasserin aut Qin ZHANG verfasserin aut Zhao WU verfasserin aut Hong-mei LIAO verfasserin aut In Chinese Journal of Contemporary Neurology and Neurosurgery Tianjin Huanhu Hospital, 2013 18(2018), 1, Seite 60-64 (DE-627)741171848 (DE-600)2710763-2 16726731 nnns volume:18 year:2018 number:1 pages:60-64 https://doaj.org/article/6313f06176074eceb83bf1daf73713b6 kostenfrei http://www.cjcnn.org/index.php/cjcnn/article/view/1722 kostenfrei https://doaj.org/toc/1672-6731 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2014 GBV_ILN_2106 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 18 2018 1 60-64
allfields_unstemmed	(DE-627)DOAJ074659103 (DE-599)DOAJ6313f06176074eceb83bf1daf73713b6 DE-627 ger DE-627 rakwb eng chi RC346-429 Xiao-jun KUANG verfasserin aut Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children 2018 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010 Movement disorders Electroencephalography Child Neurology. Diseases of the nervous system Ze-shu NING verfasserin aut Li-ming YANG verfasserin aut Bo CHEN verfasserin aut Qin ZHANG verfasserin aut Zhao WU verfasserin aut Hong-mei LIAO verfasserin aut In Chinese Journal of Contemporary Neurology and Neurosurgery Tianjin Huanhu Hospital, 2013 18(2018), 1, Seite 60-64 (DE-627)741171848 (DE-600)2710763-2 16726731 nnns volume:18 year:2018 number:1 pages:60-64 https://doaj.org/article/6313f06176074eceb83bf1daf73713b6 kostenfrei http://www.cjcnn.org/index.php/cjcnn/article/view/1722 kostenfrei https://doaj.org/toc/1672-6731 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2014 GBV_ILN_2106 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 18 2018 1 60-64
allfieldsGer	(DE-627)DOAJ074659103 (DE-599)DOAJ6313f06176074eceb83bf1daf73713b6 DE-627 ger DE-627 rakwb eng chi RC346-429 Xiao-jun KUANG verfasserin aut Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children 2018 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010 Movement disorders Electroencephalography Child Neurology. Diseases of the nervous system Ze-shu NING verfasserin aut Li-ming YANG verfasserin aut Bo CHEN verfasserin aut Qin ZHANG verfasserin aut Zhao WU verfasserin aut Hong-mei LIAO verfasserin aut In Chinese Journal of Contemporary Neurology and Neurosurgery Tianjin Huanhu Hospital, 2013 18(2018), 1, Seite 60-64 (DE-627)741171848 (DE-600)2710763-2 16726731 nnns volume:18 year:2018 number:1 pages:60-64 https://doaj.org/article/6313f06176074eceb83bf1daf73713b6 kostenfrei http://www.cjcnn.org/index.php/cjcnn/article/view/1722 kostenfrei https://doaj.org/toc/1672-6731 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2014 GBV_ILN_2106 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 18 2018 1 60-64
allfieldsSound	(DE-627)DOAJ074659103 (DE-599)DOAJ6313f06176074eceb83bf1daf73713b6 DE-627 ger DE-627 rakwb eng chi RC346-429 Xiao-jun KUANG verfasserin aut Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children 2018 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010 Movement disorders Electroencephalography Child Neurology. Diseases of the nervous system Ze-shu NING verfasserin aut Li-ming YANG verfasserin aut Bo CHEN verfasserin aut Qin ZHANG verfasserin aut Zhao WU verfasserin aut Hong-mei LIAO verfasserin aut In Chinese Journal of Contemporary Neurology and Neurosurgery Tianjin Huanhu Hospital, 2013 18(2018), 1, Seite 60-64 (DE-627)741171848 (DE-600)2710763-2 16726731 nnns volume:18 year:2018 number:1 pages:60-64 https://doaj.org/article/6313f06176074eceb83bf1daf73713b6 kostenfrei http://www.cjcnn.org/index.php/cjcnn/article/view/1722 kostenfrei https://doaj.org/toc/1672-6731 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2005 GBV_ILN_2014 GBV_ILN_2106 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 18 2018 1 60-64
language	English Chinese
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title	Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children
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title_auth	Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children
abstract	Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010
abstractGer	Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010
abstract_unstemmed	Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. DOI: 10.3969/j.issn.1672-6731.2018.01.010
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fullrecord_marcxml	<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">DOAJ074659103</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230502123932.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">230228s2018 xx \|\|\|\|\|o 00\| \|\|eng c</controlfield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)DOAJ074659103</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-599)DOAJ6313f06176074eceb83bf1daf73713b6</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield><subfield code="a">chi</subfield></datafield><datafield tag="050" ind1=" " ind2="0"><subfield code="a">RC346-429</subfield></datafield><datafield tag="100" ind1="0" ind2=" "><subfield code="a">Xiao-jun KUANG</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2018</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Objective To investigate the features and clinical significance of video electroencephalogram (VEEG) about paroxysmal kinesigenic dyskinesia (PKD) in children. Methods The clinical data of 9 children with PKD in our hospital from January 2015 to January 2017 were reviewed and analyzed. Results Nine patients were 8 boys and one girl, with age of 6.25-15.17 years, average (7.10 ± 3.24) years, and duration 1-12 months, average (6.12 ± 2.58) months. All patients were monitored 45 times of clinical paroxysms, with duration lasting for 5-35 s, average (9.21 ± 4.35) s. Clinical manifestations included 10 times of simple choreoathetosis, 6 times of dystonia and unsteady stand, 29 times of choreoathetosis, dystonia and unsteady stand. Clinical paroxysms of all patients were induced by sudden movement, among whom 2 cases (2/9) were induced by hyperventilation and one case (1/9) by starle. Four cases (4/9) had aura such as rigidity and numbness of limbs and paraesthesia before the onset. The ictal VEEG showed normal background rhythm or was covered by a large number of motion artifacts. Abnormal signs such as epileptic discharges, background rhythm changes or local slow waves were not seen. During interictal phase, background activities were normal. Two cases (2/9) showed nonspecific abnormalities, including frontal or occipital discontinuous slow waves, one case (1/9) showed centro?temporal (Rolandic area) spikes. Five cases (5/9) were positive for proline-rich transmembrane protein 2 (PRRT2) gene. Conclusions There may be interictal nonspecific abnormalities or epileptic discharges in VEEG of children with PKD. The clinical manifestations and synchronous VEEG during the attack period are of great significance to make clear diagnosises. 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Video electroencephalogram features of paroxysmal kinesigenic dyskinesia in children

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