Epithelial maturation and molecular biology of oral HPV

<p<Abstract</p< <p<Human papillomavirus (HPV) is widespread and can cause latent infection in basal cells, with low HPV DNA copy-number insufficient for transmission of infection; can cause subclinical infection that is active but without clinical signs; or can cause clinical infec...
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Autor*in:	Wood Neil H [verfasserIn] Khammissa Razia AG [verfasserIn] Feller Liviu [verfasserIn] Lemmer Johan [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2009

Übergeordnetes Werk:	In: Infectious Agents and Cancer - BMC, 2006, 4(2009), 1, p 16
Übergeordnetes Werk:	volume:4 ; year:2009 ; number:1, p 16

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.1186/1750-9378-4-16

Katalog-ID:	DOAJ075485036

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allfieldsSound	10.1186/1750-9378-4-16 doi (DE-627)DOAJ075485036 (DE-599)DOAJb604d7a01bbb4876933f34229711be0b DE-627 ger DE-627 rakwb eng RC254-282 RC109-216 Wood Neil H verfasserin aut Epithelial maturation and molecular biology of oral HPV 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier <p<Abstract</p< <p<Human papillomavirus (HPV) is widespread and can cause latent infection in basal cells, with low HPV DNA copy-number insufficient for transmission of infection; can cause subclinical infection that is active but without clinical signs; or can cause clinical infection leading to benign, potentially malignant or malignant lesions. The HPV cycle is influenced by the stage of maturation of the infected keratinocytes, and the production of virions is restricted to the post-mitotic suprabasal epithelial cells where all the virus genes are expressed.</p< <p<Low-risk HPV genotypes are associated with the development of benign oral lesions, whereas high-risk HPV genotypes are implicated in the development of malignant epithelial neoplasms. The rôle of high-risk HPV as a causative agent in epithelial malignancy is different at different anatomical sites: it is almost invariably implicated in squamous cell carcinoma of the uterine cervix, fairly frequently implicated in squamous cell carcinoma of the oropharynx, and it is seldom implicated in squamous cell carcinoma of the mouth.</p< Neoplasms. Tumors. Oncology. Including cancer and carcinogens Infectious and parasitic diseases Khammissa Razia AG verfasserin aut Feller Liviu verfasserin aut Lemmer Johan verfasserin aut In Infectious Agents and Cancer BMC, 2006 4(2009), 1, p 16 (DE-627)51781403X (DE-600)2251117-9 17509378 nnns volume:4 year:2009 number:1, p 16 https://doi.org/10.1186/1750-9378-4-16 kostenfrei https://doaj.org/article/b604d7a01bbb4876933f34229711be0b kostenfrei http://www.infectagentscancer.com/content/4/1/16 kostenfrei https://doaj.org/toc/1750-9378 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 4 2009 1, p 16
language	English
source	In Infectious Agents and Cancer 4(2009), 1, p 16 volume:4 year:2009 number:1, p 16
sourceStr	In Infectious Agents and Cancer 4(2009), 1, p 16 volume:4 year:2009 number:1, p 16
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topic_facet	Neoplasms. Tumors. Oncology. Including cancer and carcinogens Infectious and parasitic diseases
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container_title	Infectious Agents and Cancer
authorswithroles_txt_mv	Wood Neil H @@aut@@ Khammissa Razia AG @@aut@@ Feller Liviu @@aut@@ Lemmer Johan @@aut@@
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title	Epithelial maturation and molecular biology of oral HPV
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abstract	<p<Abstract</p< <p<Human papillomavirus (HPV) is widespread and can cause latent infection in basal cells, with low HPV DNA copy-number insufficient for transmission of infection; can cause subclinical infection that is active but without clinical signs; or can cause clinical infection leading to benign, potentially malignant or malignant lesions. The HPV cycle is influenced by the stage of maturation of the infected keratinocytes, and the production of virions is restricted to the post-mitotic suprabasal epithelial cells where all the virus genes are expressed.</p< <p<Low-risk HPV genotypes are associated with the development of benign oral lesions, whereas high-risk HPV genotypes are implicated in the development of malignant epithelial neoplasms. The rôle of high-risk HPV as a causative agent in epithelial malignancy is different at different anatomical sites: it is almost invariably implicated in squamous cell carcinoma of the uterine cervix, fairly frequently implicated in squamous cell carcinoma of the oropharynx, and it is seldom implicated in squamous cell carcinoma of the mouth.</p<
abstractGer	<p<Abstract</p< <p<Human papillomavirus (HPV) is widespread and can cause latent infection in basal cells, with low HPV DNA copy-number insufficient for transmission of infection; can cause subclinical infection that is active but without clinical signs; or can cause clinical infection leading to benign, potentially malignant or malignant lesions. The HPV cycle is influenced by the stage of maturation of the infected keratinocytes, and the production of virions is restricted to the post-mitotic suprabasal epithelial cells where all the virus genes are expressed.</p< <p<Low-risk HPV genotypes are associated with the development of benign oral lesions, whereas high-risk HPV genotypes are implicated in the development of malignant epithelial neoplasms. The rôle of high-risk HPV as a causative agent in epithelial malignancy is different at different anatomical sites: it is almost invariably implicated in squamous cell carcinoma of the uterine cervix, fairly frequently implicated in squamous cell carcinoma of the oropharynx, and it is seldom implicated in squamous cell carcinoma of the mouth.</p<
abstract_unstemmed	<p<Abstract</p< <p<Human papillomavirus (HPV) is widespread and can cause latent infection in basal cells, with low HPV DNA copy-number insufficient for transmission of infection; can cause subclinical infection that is active but without clinical signs; or can cause clinical infection leading to benign, potentially malignant or malignant lesions. The HPV cycle is influenced by the stage of maturation of the infected keratinocytes, and the production of virions is restricted to the post-mitotic suprabasal epithelial cells where all the virus genes are expressed.</p< <p<Low-risk HPV genotypes are associated with the development of benign oral lesions, whereas high-risk HPV genotypes are implicated in the development of malignant epithelial neoplasms. The rôle of high-risk HPV as a causative agent in epithelial malignancy is different at different anatomical sites: it is almost invariably implicated in squamous cell carcinoma of the uterine cervix, fairly frequently implicated in squamous cell carcinoma of the oropharynx, and it is seldom implicated in squamous cell carcinoma of the mouth.</p<
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title_short	Epithelial maturation and molecular biology of oral HPV
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