The pathogenesis of idiopathic normal pressure hydrocephalus based on the understanding of AQP1 and AQP4
Idiopathic normal pressure hydrocephalus (iNPH) is a neurological disorder without a recognized cause. Aquaporins (AQPs) are transmembrane channels that carry water through cell membranes and are critical for cerebrospinal fluid circulation and cerebral water balance. The function of AQPs in develop...
Ausführliche Beschreibung
Autor*in: |
Zitong Zhao [verfasserIn] Jian He [verfasserIn] Yibing Chen [verfasserIn] Yuchang Wang [verfasserIn] Chuansen Wang [verfasserIn] Changwu Tan [verfasserIn] Junbo Liao [verfasserIn] Gelei Xiao [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2022 |
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Schlagwörter: |
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Übergeordnetes Werk: |
In: Frontiers in Molecular Neuroscience - Frontiers Media S.A., 2008, 15(2022) |
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Übergeordnetes Werk: |
volume:15 ; year:2022 |
Links: |
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DOI / URN: |
10.3389/fnmol.2022.952036 |
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Katalog-ID: |
DOAJ084415835 |
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10.3389/fnmol.2022.952036 doi (DE-627)DOAJ084415835 (DE-599)DOAJa8826dc72f254ae9b1994e37b6ab678b DE-627 ger DE-627 rakwb eng RC321-571 Zitong Zhao verfasserin aut The pathogenesis of idiopathic normal pressure hydrocephalus based on the understanding of AQP1 and AQP4 2022 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Idiopathic normal pressure hydrocephalus (iNPH) is a neurological disorder without a recognized cause. Aquaporins (AQPs) are transmembrane channels that carry water through cell membranes and are critical for cerebrospinal fluid circulation and cerebral water balance. The function of AQPs in developing and maintaining hydrocephalus should be studied in greater detail as a possible diagnostic and therapeutic tool. Recent research indicates that patients with iNPH exhibited high levels of aquaporin 1 and low levels of aquaporin 4 expression, suggesting that these AQPs are essential in iNPH pathogenesis. To determine the source of iNPH and diagnose and treat it, it is necessary to examine and appreciate their function in the genesis and maintenance of hydrocephalus. The expression, function, and regulation of AQPs in iNPH are reviewed in this article, in order to provide fresh targets and suggestions for future research. iNPH AQP1 AQP4 hydrocephalus pathogenesis Neurosciences. Biological psychiatry. Neuropsychiatry Zitong Zhao verfasserin aut Zitong Zhao verfasserin aut Jian He verfasserin aut Yibing Chen verfasserin aut Yibing Chen verfasserin aut Yibing Chen verfasserin aut Yuchang Wang verfasserin aut Yuchang Wang verfasserin aut Yuchang Wang verfasserin aut Chuansen Wang verfasserin aut Chuansen Wang verfasserin aut Chuansen Wang verfasserin aut Changwu Tan verfasserin aut Changwu Tan verfasserin aut Changwu Tan verfasserin aut Junbo Liao verfasserin aut Junbo Liao verfasserin aut Junbo Liao verfasserin aut Gelei Xiao verfasserin aut Gelei Xiao verfasserin aut Gelei Xiao verfasserin aut In Frontiers in Molecular Neuroscience Frontiers Media S.A., 2008 15(2022) (DE-627)579826449 (DE-600)2452967-9 16625099 nnns volume:15 year:2022 https://doi.org/10.3389/fnmol.2022.952036 kostenfrei https://doaj.org/article/a8826dc72f254ae9b1994e37b6ab678b kostenfrei https://www.frontiersin.org/articles/10.3389/fnmol.2022.952036/full kostenfrei https://doaj.org/toc/1662-5099 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 15 2022 |
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Zitong Zhao misc RC321-571 misc iNPH misc AQP1 misc AQP4 misc hydrocephalus misc pathogenesis misc Neurosciences. Biological psychiatry. Neuropsychiatry The pathogenesis of idiopathic normal pressure hydrocephalus based on the understanding of AQP1 and AQP4 |
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The pathogenesis of idiopathic normal pressure hydrocephalus based on the understanding of AQP1 and AQP4 |
abstract |
Idiopathic normal pressure hydrocephalus (iNPH) is a neurological disorder without a recognized cause. Aquaporins (AQPs) are transmembrane channels that carry water through cell membranes and are critical for cerebrospinal fluid circulation and cerebral water balance. The function of AQPs in developing and maintaining hydrocephalus should be studied in greater detail as a possible diagnostic and therapeutic tool. Recent research indicates that patients with iNPH exhibited high levels of aquaporin 1 and low levels of aquaporin 4 expression, suggesting that these AQPs are essential in iNPH pathogenesis. To determine the source of iNPH and diagnose and treat it, it is necessary to examine and appreciate their function in the genesis and maintenance of hydrocephalus. The expression, function, and regulation of AQPs in iNPH are reviewed in this article, in order to provide fresh targets and suggestions for future research. |
abstractGer |
Idiopathic normal pressure hydrocephalus (iNPH) is a neurological disorder without a recognized cause. Aquaporins (AQPs) are transmembrane channels that carry water through cell membranes and are critical for cerebrospinal fluid circulation and cerebral water balance. The function of AQPs in developing and maintaining hydrocephalus should be studied in greater detail as a possible diagnostic and therapeutic tool. Recent research indicates that patients with iNPH exhibited high levels of aquaporin 1 and low levels of aquaporin 4 expression, suggesting that these AQPs are essential in iNPH pathogenesis. To determine the source of iNPH and diagnose and treat it, it is necessary to examine and appreciate their function in the genesis and maintenance of hydrocephalus. The expression, function, and regulation of AQPs in iNPH are reviewed in this article, in order to provide fresh targets and suggestions for future research. |
abstract_unstemmed |
Idiopathic normal pressure hydrocephalus (iNPH) is a neurological disorder without a recognized cause. Aquaporins (AQPs) are transmembrane channels that carry water through cell membranes and are critical for cerebrospinal fluid circulation and cerebral water balance. The function of AQPs in developing and maintaining hydrocephalus should be studied in greater detail as a possible diagnostic and therapeutic tool. Recent research indicates that patients with iNPH exhibited high levels of aquaporin 1 and low levels of aquaporin 4 expression, suggesting that these AQPs are essential in iNPH pathogenesis. To determine the source of iNPH and diagnose and treat it, it is necessary to examine and appreciate their function in the genesis and maintenance of hydrocephalus. The expression, function, and regulation of AQPs in iNPH are reviewed in this article, in order to provide fresh targets and suggestions for future research. |
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Aquaporins (AQPs) are transmembrane channels that carry water through cell membranes and are critical for cerebrospinal fluid circulation and cerebral water balance. The function of AQPs in developing and maintaining hydrocephalus should be studied in greater detail as a possible diagnostic and therapeutic tool. Recent research indicates that patients with iNPH exhibited high levels of aquaporin 1 and low levels of aquaporin 4 expression, suggesting that these AQPs are essential in iNPH pathogenesis. To determine the source of iNPH and diagnose and treat it, it is necessary to examine and appreciate their function in the genesis and maintenance of hydrocephalus. 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Neuropsychiatry</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">Zitong Zhao</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">Zitong Zhao</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">Jian He</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">Yibing Chen</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">Yibing Chen</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="0" ind2=" "><subfield code="a">Yibing Chen</subfield><subfield code="e">verfasserin</subfield><subfield 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