A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain
This study was performed to investigate the improving effect of a mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< (AASC) on cognitive dysfunction in mice with long-term exposure to fine particles (particulate matter smaller than 2.5 µm: PM<sub<2.5</su...
Ausführliche Beschreibung
Autor*in: |
Jin-Yong Kang [verfasserIn] Jong-Min Kim [verfasserIn] Seon-Kyeong Park [verfasserIn] Hyo-Lim Lee [verfasserIn] Ho-Jin Heo [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2023 |
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Schlagwörter: |
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Übergeordnetes Werk: |
In: Plants - MDPI AG, 2013, 12(2023), 6, p 1230 |
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Übergeordnetes Werk: |
volume:12 ; year:2023 ; number:6, p 1230 |
Links: |
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DOI / URN: |
10.3390/plants12061230 |
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Katalog-ID: |
DOAJ087262185 |
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10.3390/plants12061230 doi (DE-627)DOAJ087262185 (DE-599)DOAJ88e02c1b8acb4090b4806e8ad9fe142e DE-627 ger DE-627 rakwb eng QK1-989 Jin-Yong Kang verfasserin aut A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain 2023 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier This study was performed to investigate the improving effect of a mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< (AASC) on cognitive dysfunction in mice with long-term exposure to fine particles (particulate matter smaller than 2.5 µm: PM<sub<2.5</sub<). The main compounds of AASC were identified as dicaffeoylquinic acid isomers of <i<A. argyi</i< and a quercetin-3-glucoside of <i<S. chinesis</i<. As a result of behavioral tests for the evaluation of cognitive function, it was confirmed that cognitive dysfunction was induced in the PM<sub<2.5</sub< exposure group, and a tendency to improve in the AASC group was confirmed. Increased oxidative stress and inflammatory response and mitochondrial dysfunction were observed in the brain and lung tissues of the PM group. Damage to the brain and lung affected the accumulation of amyloid beta (Aβ) in the brain. It increased Aβ and induced the cholinergic dysfunction, hyperphosphorylation of the tau protein, and activation of apoptosis, leading to cognitive impairment. However, AASC suppressed brain and lung oxidative stress and inflammation, thereby suppressing brain Aβ expression. Consequently, this study shows the potential that a steady intake of plant resources with antioxidant and anti-inflammatory activity could prevent cognitive impairment caused by PM<sub<2.5</sub<. <i<Artemisia argyi</i< <i<Saururus chinensis</i< fine particles cognitive dysfunction inflammation oxidative stress Botany Jong-Min Kim verfasserin aut Seon-Kyeong Park verfasserin aut Hyo-Lim Lee verfasserin aut Ho-Jin Heo verfasserin aut In Plants MDPI AG, 2013 12(2023), 6, p 1230 (DE-627)737288345 (DE-600)2704341-1 22237747 nnns volume:12 year:2023 number:6, p 1230 https://doi.org/10.3390/plants12061230 kostenfrei https://doaj.org/article/88e02c1b8acb4090b4806e8ad9fe142e kostenfrei https://www.mdpi.com/2223-7747/12/6/1230 kostenfrei https://doaj.org/toc/2223-7747 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 12 2023 6, p 1230 |
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QK1-989 A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain <i<Artemisia argyi</i< <i<Saururus chinensis</i< fine particles cognitive dysfunction inflammation oxidative stress |
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A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain |
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A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain |
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mixture of <i<artemisia argyi</i< and <i<saururus chinensis</i< improves pm<sub<2.5</sub<-induced cognitive dysfunction by regulating oxidative stress and inflammatory response in the lung and brain |
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A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain |
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This study was performed to investigate the improving effect of a mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< (AASC) on cognitive dysfunction in mice with long-term exposure to fine particles (particulate matter smaller than 2.5 µm: PM<sub<2.5</sub<). The main compounds of AASC were identified as dicaffeoylquinic acid isomers of <i<A. argyi</i< and a quercetin-3-glucoside of <i<S. chinesis</i<. As a result of behavioral tests for the evaluation of cognitive function, it was confirmed that cognitive dysfunction was induced in the PM<sub<2.5</sub< exposure group, and a tendency to improve in the AASC group was confirmed. Increased oxidative stress and inflammatory response and mitochondrial dysfunction were observed in the brain and lung tissues of the PM group. Damage to the brain and lung affected the accumulation of amyloid beta (Aβ) in the brain. It increased Aβ and induced the cholinergic dysfunction, hyperphosphorylation of the tau protein, and activation of apoptosis, leading to cognitive impairment. However, AASC suppressed brain and lung oxidative stress and inflammation, thereby suppressing brain Aβ expression. Consequently, this study shows the potential that a steady intake of plant resources with antioxidant and anti-inflammatory activity could prevent cognitive impairment caused by PM<sub<2.5</sub<. |
abstractGer |
This study was performed to investigate the improving effect of a mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< (AASC) on cognitive dysfunction in mice with long-term exposure to fine particles (particulate matter smaller than 2.5 µm: PM<sub<2.5</sub<). The main compounds of AASC were identified as dicaffeoylquinic acid isomers of <i<A. argyi</i< and a quercetin-3-glucoside of <i<S. chinesis</i<. As a result of behavioral tests for the evaluation of cognitive function, it was confirmed that cognitive dysfunction was induced in the PM<sub<2.5</sub< exposure group, and a tendency to improve in the AASC group was confirmed. Increased oxidative stress and inflammatory response and mitochondrial dysfunction were observed in the brain and lung tissues of the PM group. Damage to the brain and lung affected the accumulation of amyloid beta (Aβ) in the brain. It increased Aβ and induced the cholinergic dysfunction, hyperphosphorylation of the tau protein, and activation of apoptosis, leading to cognitive impairment. However, AASC suppressed brain and lung oxidative stress and inflammation, thereby suppressing brain Aβ expression. Consequently, this study shows the potential that a steady intake of plant resources with antioxidant and anti-inflammatory activity could prevent cognitive impairment caused by PM<sub<2.5</sub<. |
abstract_unstemmed |
This study was performed to investigate the improving effect of a mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< (AASC) on cognitive dysfunction in mice with long-term exposure to fine particles (particulate matter smaller than 2.5 µm: PM<sub<2.5</sub<). The main compounds of AASC were identified as dicaffeoylquinic acid isomers of <i<A. argyi</i< and a quercetin-3-glucoside of <i<S. chinesis</i<. As a result of behavioral tests for the evaluation of cognitive function, it was confirmed that cognitive dysfunction was induced in the PM<sub<2.5</sub< exposure group, and a tendency to improve in the AASC group was confirmed. Increased oxidative stress and inflammatory response and mitochondrial dysfunction were observed in the brain and lung tissues of the PM group. Damage to the brain and lung affected the accumulation of amyloid beta (Aβ) in the brain. It increased Aβ and induced the cholinergic dysfunction, hyperphosphorylation of the tau protein, and activation of apoptosis, leading to cognitive impairment. However, AASC suppressed brain and lung oxidative stress and inflammation, thereby suppressing brain Aβ expression. Consequently, this study shows the potential that a steady intake of plant resources with antioxidant and anti-inflammatory activity could prevent cognitive impairment caused by PM<sub<2.5</sub<. |
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A Mixture of <i<Artemisia argyi</i< and <i<Saururus chinensis</i< Improves PM<sub<2.5</sub<-Induced Cognitive Dysfunction by Regulating Oxidative Stress and Inflammatory Response in the Lung and Brain |
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7.3997936 |