Ferroptosis, pyroptosis and necroptosis in acute respiratory distress syndrome

Abstract Acute respiratory distress syndrome (ARDS) is an acute and uncontrolled pulmonary inflammation caused by various insults. Cell death is a critical mechanism in the pathogenesis of ARDS. Ferroptosis, a novel form of cell death defined as iron-mediated lipid peroxidation, has been shown to pl...
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Autor*in:	Yongxin Zheng [verfasserIn] Yongbo Huang [verfasserIn] Yonghao Xu [verfasserIn] Ling Sang [verfasserIn] Xiaoqing Liu [verfasserIn] Yimin Li [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2023

Übergeordnetes Werk:	In: Cell Death Discovery - Nature Publishing Group, 2018, 9(2023), 1, Seite 12
Übergeordnetes Werk:	volume:9 ; year:2023 ; number:1 ; pages:12

Links:	Link aufrufen Link aufrufen Link aufrufen Journal toc

DOI / URN:	10.1038/s41420-023-01369-2

Katalog-ID:	DOAJ087774305

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520			\|a Abstract Acute respiratory distress syndrome (ARDS) is an acute and uncontrolled pulmonary inflammation caused by various insults. Cell death is a critical mechanism in the pathogenesis of ARDS. Ferroptosis, a novel form of cell death defined as iron-mediated lipid peroxidation, has been shown to play a role in the pathogenesis of ARDS. Additionally, pyroptosis and necroptosis are also involved in the pathophysiological process of ARDS. The crosstalk among ferroptosis, pyroptosis, and necroptosis is getting increasing attention. Therefore, this review will mainly summarize the molecular mechanisms and central pathophysiological role of ferroptosis in ARDS. We will also discuss our understanding of pyroptosis and necroptosis as they pertain to the pathogenesis of ARDS. Furthermore, we also describe the pathological processes that engage crosstalk among ferroptosis, pyroptosis, and necroptosis. We consider that individual pathways of ferroptosis, pyroptosis, and necroptosis are highly interconnected and can compensate for one another to promote cell death.
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allfieldsSound	10.1038/s41420-023-01369-2 doi (DE-627)DOAJ087774305 (DE-599)DOAJfcce78bd8fc742fba4a13d86ed767a56 DE-627 ger DE-627 rakwb eng RC254-282 QH573-671 Yongxin Zheng verfasserin aut Ferroptosis, pyroptosis and necroptosis in acute respiratory distress syndrome 2023 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Acute respiratory distress syndrome (ARDS) is an acute and uncontrolled pulmonary inflammation caused by various insults. Cell death is a critical mechanism in the pathogenesis of ARDS. Ferroptosis, a novel form of cell death defined as iron-mediated lipid peroxidation, has been shown to play a role in the pathogenesis of ARDS. Additionally, pyroptosis and necroptosis are also involved in the pathophysiological process of ARDS. The crosstalk among ferroptosis, pyroptosis, and necroptosis is getting increasing attention. Therefore, this review will mainly summarize the molecular mechanisms and central pathophysiological role of ferroptosis in ARDS. We will also discuss our understanding of pyroptosis and necroptosis as they pertain to the pathogenesis of ARDS. Furthermore, we also describe the pathological processes that engage crosstalk among ferroptosis, pyroptosis, and necroptosis. We consider that individual pathways of ferroptosis, pyroptosis, and necroptosis are highly interconnected and can compensate for one another to promote cell death. Neoplasms. Tumors. Oncology. Including cancer and carcinogens Cytology Yongbo Huang verfasserin aut Yonghao Xu verfasserin aut Ling Sang verfasserin aut Xiaoqing Liu verfasserin aut Yimin Li verfasserin aut In Cell Death Discovery Nature Publishing Group, 2018 9(2023), 1, Seite 12 (DE-627)843856351 (DE-600)2842546-7 20587716 nnns volume:9 year:2023 number:1 pages:12 https://doi.org/10.1038/s41420-023-01369-2 kostenfrei https://doaj.org/article/fcce78bd8fc742fba4a13d86ed767a56 kostenfrei https://doi.org/10.1038/s41420-023-01369-2 kostenfrei https://doaj.org/toc/2058-7716 Journal toc kostenfrei GBV_USEFLAG_A SYSFLAG_A GBV_DOAJ GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 9 2023 1 12
language	English
source	In Cell Death Discovery 9(2023), 1, Seite 12 volume:9 year:2023 number:1 pages:12
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authorswithroles_txt_mv	Yongxin Zheng @@aut@@ Yongbo Huang @@aut@@ Yonghao Xu @@aut@@ Ling Sang @@aut@@ Xiaoqing Liu @@aut@@ Yimin Li @@aut@@
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callnumber-first	R - Medicine
author	Yongxin Zheng
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title	Ferroptosis, pyroptosis and necroptosis in acute respiratory distress syndrome
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title_auth	Ferroptosis, pyroptosis and necroptosis in acute respiratory distress syndrome
abstract	Abstract Acute respiratory distress syndrome (ARDS) is an acute and uncontrolled pulmonary inflammation caused by various insults. Cell death is a critical mechanism in the pathogenesis of ARDS. Ferroptosis, a novel form of cell death defined as iron-mediated lipid peroxidation, has been shown to play a role in the pathogenesis of ARDS. Additionally, pyroptosis and necroptosis are also involved in the pathophysiological process of ARDS. The crosstalk among ferroptosis, pyroptosis, and necroptosis is getting increasing attention. Therefore, this review will mainly summarize the molecular mechanisms and central pathophysiological role of ferroptosis in ARDS. We will also discuss our understanding of pyroptosis and necroptosis as they pertain to the pathogenesis of ARDS. Furthermore, we also describe the pathological processes that engage crosstalk among ferroptosis, pyroptosis, and necroptosis. We consider that individual pathways of ferroptosis, pyroptosis, and necroptosis are highly interconnected and can compensate for one another to promote cell death.
abstractGer	Abstract Acute respiratory distress syndrome (ARDS) is an acute and uncontrolled pulmonary inflammation caused by various insults. Cell death is a critical mechanism in the pathogenesis of ARDS. Ferroptosis, a novel form of cell death defined as iron-mediated lipid peroxidation, has been shown to play a role in the pathogenesis of ARDS. Additionally, pyroptosis and necroptosis are also involved in the pathophysiological process of ARDS. The crosstalk among ferroptosis, pyroptosis, and necroptosis is getting increasing attention. Therefore, this review will mainly summarize the molecular mechanisms and central pathophysiological role of ferroptosis in ARDS. We will also discuss our understanding of pyroptosis and necroptosis as they pertain to the pathogenesis of ARDS. Furthermore, we also describe the pathological processes that engage crosstalk among ferroptosis, pyroptosis, and necroptosis. We consider that individual pathways of ferroptosis, pyroptosis, and necroptosis are highly interconnected and can compensate for one another to promote cell death.
abstract_unstemmed	Abstract Acute respiratory distress syndrome (ARDS) is an acute and uncontrolled pulmonary inflammation caused by various insults. Cell death is a critical mechanism in the pathogenesis of ARDS. Ferroptosis, a novel form of cell death defined as iron-mediated lipid peroxidation, has been shown to play a role in the pathogenesis of ARDS. Additionally, pyroptosis and necroptosis are also involved in the pathophysiological process of ARDS. The crosstalk among ferroptosis, pyroptosis, and necroptosis is getting increasing attention. Therefore, this review will mainly summarize the molecular mechanisms and central pathophysiological role of ferroptosis in ARDS. We will also discuss our understanding of pyroptosis and necroptosis as they pertain to the pathogenesis of ARDS. Furthermore, we also describe the pathological processes that engage crosstalk among ferroptosis, pyroptosis, and necroptosis. We consider that individual pathways of ferroptosis, pyroptosis, and necroptosis are highly interconnected and can compensate for one another to promote cell death.
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title_short	Ferroptosis, pyroptosis and necroptosis in acute respiratory distress syndrome
url	https://doi.org/10.1038/s41420-023-01369-2 https://doaj.org/article/fcce78bd8fc742fba4a13d86ed767a56 https://doaj.org/toc/2058-7716
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Ferroptosis, pyroptosis and necroptosis in acute respiratory distress syndrome

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