The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players

Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprin...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Williams, N. [verfasserIn] Russell, M. [verfasserIn] Cook, C.J. [verfasserIn] Kilduff, L.P. [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2018

Schlagwörter:	Occlusion Sprint Hormonal reactivity

Übergeordnetes Werk:	Enthalten in: Journal of science and medicine in sport - Amsterdam [u.a.] : Elsevier, 1998, 21, Seite 1095-1099
Übergeordnetes Werk:	volume:21 ; pages:1095-1099

DOI / URN:	10.1016/j.jsams.2018.02.012

Katalog-ID:	ELV000318264

Internformat


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100	1		\|a Williams, N. \|e verfasserin \|4 aut
245	1	0	\|a The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
264		1	\|c 2018
336			\|a nicht spezifiziert \|b zzz \|2 rdacontent
337			\|a Computermedien \|b c \|2 rdamedia
338			\|a Online-Ressource \|b cr \|2 rdacarrier
520			\|a Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.
650		4	\|a Occlusion
650		4	\|a Sprint
650		4	\|a Hormonal reactivity
700	1		\|a Russell, M. \|e verfasserin \|4 aut
700	1		\|a Cook, C.J. \|e verfasserin \|4 aut
700	1		\|a Kilduff, L.P. \|e verfasserin \|4 aut
773	0	8	\|i Enthalten in \|t Journal of science and medicine in sport \|d Amsterdam [u.a.] : Elsevier, 1998 \|g 21, Seite 1095-1099 \|h Online-Ressource \|w (DE-627)510462278 \|w (DE-600)2230157-4 \|w (DE-576)27158632X \|x 1878-1861 \|7 nnns
773	1	8	\|g volume:21 \|g pages:1095-1099
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912			\|a GBV_ILN_23
912			\|a GBV_ILN_24
912			\|a GBV_ILN_31
912			\|a GBV_ILN_32
912			\|a GBV_ILN_40
912			\|a GBV_ILN_60
912			\|a GBV_ILN_62
912			\|a GBV_ILN_63
912			\|a GBV_ILN_65
912			\|a GBV_ILN_69
912			\|a GBV_ILN_70
912			\|a GBV_ILN_73
912			\|a GBV_ILN_74
912			\|a GBV_ILN_90
912			\|a GBV_ILN_95
912			\|a GBV_ILN_100
912			\|a GBV_ILN_101
912			\|a GBV_ILN_105
912			\|a GBV_ILN_110
912			\|a GBV_ILN_151
912			\|a GBV_ILN_224
912			\|a GBV_ILN_370
912			\|a GBV_ILN_602
912			\|a GBV_ILN_702
912			\|a GBV_ILN_2003
912			\|a GBV_ILN_2004
912			\|a GBV_ILN_2005
912			\|a GBV_ILN_2006
912			\|a GBV_ILN_2008
912			\|a GBV_ILN_2011
912			\|a GBV_ILN_2014
912			\|a GBV_ILN_2015
912			\|a GBV_ILN_2020
912			\|a GBV_ILN_2021
912			\|a GBV_ILN_2025
912			\|a GBV_ILN_2027
912			\|a GBV_ILN_2034
912			\|a GBV_ILN_2038
912			\|a GBV_ILN_2044
912			\|a GBV_ILN_2048
912			\|a GBV_ILN_2049
912			\|a GBV_ILN_2050
912			\|a GBV_ILN_2056
912			\|a GBV_ILN_2059
912			\|a GBV_ILN_2061
912			\|a GBV_ILN_2064
912			\|a GBV_ILN_2065
912			\|a GBV_ILN_2068
912			\|a GBV_ILN_2088
912			\|a GBV_ILN_2111
912			\|a GBV_ILN_2112
912			\|a GBV_ILN_2113
912			\|a GBV_ILN_2118
912			\|a GBV_ILN_2122
912			\|a GBV_ILN_2129
912			\|a GBV_ILN_2143
912			\|a GBV_ILN_2147
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912			\|a GBV_ILN_2152
912			\|a GBV_ILN_2153
912			\|a GBV_ILN_2190
912			\|a GBV_ILN_2336
912			\|a GBV_ILN_2470
912			\|a GBV_ILN_2507
912			\|a GBV_ILN_2522
912			\|a GBV_ILN_4035
912			\|a GBV_ILN_4037
912			\|a GBV_ILN_4046
912			\|a GBV_ILN_4112
912			\|a GBV_ILN_4125
912			\|a GBV_ILN_4126
912			\|a GBV_ILN_4242
912			\|a GBV_ILN_4251
912			\|a GBV_ILN_4305
912			\|a GBV_ILN_4313
912			\|a GBV_ILN_4322
912			\|a GBV_ILN_4323
912			\|a GBV_ILN_4324
912			\|a GBV_ILN_4325
912			\|a GBV_ILN_4326
912			\|a GBV_ILN_4333
912			\|a GBV_ILN_4334
912			\|a GBV_ILN_4335
912			\|a GBV_ILN_4338
912			\|a GBV_ILN_4393
936	b	k	\|a 44.70 \|j Sportmedizin
951			\|a AR
952			\|d 21 \|h 1095-1099

Indexfelder

author_variant	n w nw m r mr c c cc l k lk
matchkey_str	article:18781861:2018----::hefcolwribclsoorcvrfloigpitxri
hierarchy_sort_str	2018
bklnumber	44.70
publishDate	2018
allfields	10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099
spelling	10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099
allfields_unstemmed	10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099
allfieldsGer	10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099
allfieldsSound	10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099
language	English
source	Enthalten in Journal of science and medicine in sport 21, Seite 1095-1099 volume:21 pages:1095-1099
sourceStr	Enthalten in Journal of science and medicine in sport 21, Seite 1095-1099 volume:21 pages:1095-1099
format_phy_str_mv	Article
bklname	Sportmedizin
institution	findex.gbv.de
topic_facet	Occlusion Sprint Hormonal reactivity
dewey-raw	610
isfreeaccess_bool	false
container_title	Journal of science and medicine in sport
authorswithroles_txt_mv	Williams, N. @@aut@@ Russell, M. @@aut@@ Cook, C.J. @@aut@@ Kilduff, L.P. @@aut@@
publishDateDaySort_date	2018-01-01T00:00:00Z
hierarchy_top_id	510462278
dewey-sort	3610
id	ELV000318264
language_de	englisch
fullrecord	<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">ELV000318264</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230524150054.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">230427s2018 xx \|\|\|\|\|o 00\| \|\|eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1016/j.jsams.2018.02.012</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)ELV000318264</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ELSEVIER)S1440-2440(18)30064-1</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rda</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="a">796</subfield><subfield code="q">DE-600</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">44.70</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Williams, N.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2018</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Occlusion</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Sprint</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Hormonal reactivity</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Russell, M.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Cook, C.J.</subfield><subfield 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author	Williams, N.
spellingShingle	Williams, N. ddc 610 bkl 44.70 misc Occlusion misc Sprint misc Hormonal reactivity The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
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topic_title	610 796 DE-600 44.70 bkl The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players Occlusion Sprint Hormonal reactivity
topic	ddc 610 bkl 44.70 misc Occlusion misc Sprint misc Hormonal reactivity
topic_unstemmed	ddc 610 bkl 44.70 misc Occlusion misc Sprint misc Hormonal reactivity
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title	The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
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title_full	The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
author_sort	Williams, N.
journal	Journal of science and medicine in sport
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author_browse	Williams, N. Russell, M. Cook, C.J. Kilduff, L.P.
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title_sort	the effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
title_auth	The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
abstract	Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.
abstractGer	Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.
abstract_unstemmed	Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.
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title_short	The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
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up_date	2024-07-06T17:33:39.996Z
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fullrecord_marcxml	<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">ELV000318264</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230524150054.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">230427s2018 xx \|\|\|\|\|o 00\| \|\|eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1016/j.jsams.2018.02.012</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)ELV000318264</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ELSEVIER)S1440-2440(18)30064-1</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rda</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="a">796</subfield><subfield code="q">DE-600</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">44.70</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Williams, N.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2018</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Occlusion</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Sprint</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Hormonal reactivity</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Russell, M.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Cook, C.J.</subfield><subfield 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The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players

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