The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players
Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprin...
Ausführliche Beschreibung
Autor*in: |
Williams, N. [verfasserIn] Russell, M. [verfasserIn] Cook, C.J. [verfasserIn] Kilduff, L.P. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2018 |
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Schlagwörter: |
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Übergeordnetes Werk: |
Enthalten in: Journal of science and medicine in sport - Amsterdam [u.a.] : Elsevier, 1998, 21, Seite 1095-1099 |
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Übergeordnetes Werk: |
volume:21 ; pages:1095-1099 |
DOI / URN: |
10.1016/j.jsams.2018.02.012 |
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Katalog-ID: |
ELV000318264 |
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245 | 1 | 0 | |a The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players |
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520 | |a Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. | ||
650 | 4 | |a Occlusion | |
650 | 4 | |a Sprint | |
650 | 4 | |a Hormonal reactivity | |
700 | 1 | |a Russell, M. |e verfasserin |4 aut | |
700 | 1 | |a Cook, C.J. |e verfasserin |4 aut | |
700 | 1 | |a Kilduff, L.P. |e verfasserin |4 aut | |
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2018 |
allfields |
10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099 |
spelling |
10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099 |
allfields_unstemmed |
10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099 |
allfieldsGer |
10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099 |
allfieldsSound |
10.1016/j.jsams.2018.02.012 doi (DE-627)ELV000318264 (ELSEVIER)S1440-2440(18)30064-1 DE-627 ger DE-627 rda eng 610 796 DE-600 44.70 bkl Williams, N. verfasserin aut The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players 2018 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. Occlusion Sprint Hormonal reactivity Russell, M. verfasserin aut Cook, C.J. verfasserin aut Kilduff, L.P. verfasserin aut Enthalten in Journal of science and medicine in sport Amsterdam [u.a.] : Elsevier, 1998 21, Seite 1095-1099 Online-Ressource (DE-627)510462278 (DE-600)2230157-4 (DE-576)27158632X 1878-1861 nnns volume:21 pages:1095-1099 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.70 Sportmedizin AR 21 1095-1099 |
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Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. 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Williams, N. |
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Williams, N. ddc 610 bkl 44.70 misc Occlusion misc Sprint misc Hormonal reactivity The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players |
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610 796 DE-600 44.70 bkl The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players Occlusion Sprint Hormonal reactivity |
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The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players |
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the effect of lower limb occlusion on recovery following sprint exercise in academy rugby players |
title_auth |
The effect of lower limb occlusion on recovery following sprint exercise in academy rugby players |
abstract |
Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. |
abstractGer |
Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. |
abstract_unstemmed |
Objectives: The effects of vascular occlusion on recovery of physiological and neuromuscular markers over 24h, and hormonal reactivity to subsequent exercise were investigated.Design: Counterbalanced, randomised, crossover.Methods: Academy rugby players (n =24) completed six 50-m sprints (5-min inter-set recovery) before occlusion cuff application (thighs) and intermittent inflation to 171–266mmHg (Recovery) or 15mmHg (Con) for 12-min (two sets, 3-min repetitions, 3-min non-occluded reperfusion). Countermovement jumps, blood (lactate, creatine kinase), saliva (testosterone, cortisol), and perceptual (soreness, recovery) responses were measured before (baseline) and after (post, +2h, +24h) sprinting. Saliva was sampled after a 30-min resistance exercise session performed 24h after sprinting.Results: Although sprinting (total: 40.0±2.8s, p=0.238; average: 6.7±0.5s, p=0.674) influenced creatine kinase (p<0.001, +457.1±327.3μL−1, at 24h), lactate (p<0.001, 6.8±2.3mmolL−1, post), testosterone (p<0.001, −55.9±63.2pgmL−1, at 2h) and cortisol (p<0.001, −0.3±0.3μgdL−1, at 2h) concentrations, countermovement jump power output (p<0.001, −409.6±310.1W; −5.4±3.4cm, post), perceived recovery (p<0.001, −3.0±2.3, post), and muscle soreness (p<0.001; 1.5±1.1, at 24h), vascular occlusion had no effect (all p>0.05) on recovery. In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test. |
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In response to subsequent exercise performed 24h after vascular occlusion, testosterone increased pre-to-post-exercise (Recovery: p=0.031, 21.6±44.9pgmL−1; Con: p=0.178, 10.6±36.6pgmL−1) however Δtestosterone was not significantly different (p=0.109) between conditions.Conclusions: Vascular occlusion had no effect on physiological or neuromuscular markers 2h or 24h after sprinting or in response to a physical stress test.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Occlusion</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Sprint</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Hormonal reactivity</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Russell, M.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Cook, C.J.</subfield><subfield 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