Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy
The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopa...
Ausführliche Beschreibung
Gespeichert in:
| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2020 |
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| Übergeordnetes Werk: |
Enthalten in: The American journal of cardiology - Amsterdam [u.a.] : Elsevier, 1958, 125, Seite 1421-1428 |
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| Übergeordnetes Werk: |
volume:125 ; pages:1421-1428 |
| DOI / URN: |
10.1016/j.amjcard.2020.01.042 |
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| 520 | |a The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. | ||
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10.1016/j.amjcard.2020.01.042 doi (DE-627)ELV003915905 (ELSEVIER)S0002-9149(20)30107-7 DE-627 ger DE-627 rda eng 610 DE-600 44.85 bkl Galand, Vincent verfasserin aut Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. Flécher, Erwan verfasserin aut Chabanne, Céline verfasserin aut Lelong, Bernard verfasserin aut Goéminne, Céline verfasserin aut Vincentelli, André verfasserin (orcid)0000-0003-4081-786X aut Delmas, Clément verfasserin aut Dambrin, Camille verfasserin aut Picard, François verfasserin aut Sacher, Frédéric verfasserin aut Kindo, Michel verfasserin aut Minh, Tam Hoang verfasserin aut Gaudard, Philippe verfasserin aut Rouvière, Philippe verfasserin aut Sénage, Thomas verfasserin aut Michel, Magali verfasserin aut Boignard, Aude verfasserin aut Chavanon, Olivier verfasserin aut Verdonk, Constance verfasserin aut Ghodhbane, Walid verfasserin aut Pelcé, Edeline verfasserin aut Gariboldi, Vlad verfasserin aut Pozzi, Matteo verfasserin (orcid)0000-0001-6550-0872 aut Obadia, Jean-François verfasserin aut Litzler, Pierre-Yvesl verfasserin aut Anselme, Frédéric verfasserin aut Babatasi, Gerard verfasserin aut Blanchart, Katrien verfasserin aut Garnier, Fabien verfasserin aut Bielefeld, Marie verfasserin aut Hamon, David verfasserin aut Lellouche, Nicolas verfasserin aut Bourguignon, Thierry verfasserin aut Genet, Thibaud verfasserin (orcid)0000-0002-2829-7198 aut Eschalier, Romain verfasserin aut D'Ostrevy, Nicolas verfasserin aut Varlet, Emilie verfasserin aut Jouan, Jérôme verfasserin aut Vanhuyse, Fabrice verfasserin aut Blangy, Hugues verfasserin aut Leclercq, Christophe verfasserin aut Martins, Raphaël P. verfasserin aut Enthalten in The American journal of cardiology Amsterdam [u.a.] : Elsevier, 1958 125, Seite 1421-1428 Online-Ressource (DE-627)320596249 (DE-600)2019595-3 (DE-576)10963392X 1879-1913 nnns volume:125 pages:1421-1428 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.85 Kardiologie Angiologie AR 125 1421-1428 |
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10.1016/j.amjcard.2020.01.042 doi (DE-627)ELV003915905 (ELSEVIER)S0002-9149(20)30107-7 DE-627 ger DE-627 rda eng 610 DE-600 44.85 bkl Galand, Vincent verfasserin aut Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. Flécher, Erwan verfasserin aut Chabanne, Céline verfasserin aut Lelong, Bernard verfasserin aut Goéminne, Céline verfasserin aut Vincentelli, André verfasserin (orcid)0000-0003-4081-786X aut Delmas, Clément verfasserin aut Dambrin, Camille verfasserin aut Picard, François verfasserin aut Sacher, Frédéric verfasserin aut Kindo, Michel verfasserin aut Minh, Tam Hoang verfasserin aut Gaudard, Philippe verfasserin aut Rouvière, Philippe verfasserin aut Sénage, Thomas verfasserin aut Michel, Magali verfasserin aut Boignard, Aude verfasserin aut Chavanon, Olivier verfasserin aut Verdonk, Constance verfasserin aut Ghodhbane, Walid verfasserin aut Pelcé, Edeline verfasserin aut Gariboldi, Vlad verfasserin aut Pozzi, Matteo verfasserin (orcid)0000-0001-6550-0872 aut Obadia, Jean-François verfasserin aut Litzler, Pierre-Yvesl verfasserin aut Anselme, Frédéric verfasserin aut Babatasi, Gerard verfasserin aut Blanchart, Katrien verfasserin aut Garnier, Fabien verfasserin aut Bielefeld, Marie verfasserin aut Hamon, David verfasserin aut Lellouche, Nicolas verfasserin aut Bourguignon, Thierry verfasserin aut Genet, Thibaud verfasserin (orcid)0000-0002-2829-7198 aut Eschalier, Romain verfasserin aut D'Ostrevy, Nicolas verfasserin aut Varlet, Emilie verfasserin aut Jouan, Jérôme verfasserin aut Vanhuyse, Fabrice verfasserin aut Blangy, Hugues verfasserin aut Leclercq, Christophe verfasserin aut Martins, Raphaël P. verfasserin aut Enthalten in The American journal of cardiology Amsterdam [u.a.] : Elsevier, 1958 125, Seite 1421-1428 Online-Ressource (DE-627)320596249 (DE-600)2019595-3 (DE-576)10963392X 1879-1913 nnns volume:125 pages:1421-1428 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.85 Kardiologie Angiologie AR 125 1421-1428 |
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10.1016/j.amjcard.2020.01.042 doi (DE-627)ELV003915905 (ELSEVIER)S0002-9149(20)30107-7 DE-627 ger DE-627 rda eng 610 DE-600 44.85 bkl Galand, Vincent verfasserin aut Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. Flécher, Erwan verfasserin aut Chabanne, Céline verfasserin aut Lelong, Bernard verfasserin aut Goéminne, Céline verfasserin aut Vincentelli, André verfasserin (orcid)0000-0003-4081-786X aut Delmas, Clément verfasserin aut Dambrin, Camille verfasserin aut Picard, François verfasserin aut Sacher, Frédéric verfasserin aut Kindo, Michel verfasserin aut Minh, Tam Hoang verfasserin aut Gaudard, Philippe verfasserin aut Rouvière, Philippe verfasserin aut Sénage, Thomas verfasserin aut Michel, Magali verfasserin aut Boignard, Aude verfasserin aut Chavanon, Olivier verfasserin aut Verdonk, Constance verfasserin aut Ghodhbane, Walid verfasserin aut Pelcé, Edeline verfasserin aut Gariboldi, Vlad verfasserin aut Pozzi, Matteo verfasserin (orcid)0000-0001-6550-0872 aut Obadia, Jean-François verfasserin aut Litzler, Pierre-Yvesl verfasserin aut Anselme, Frédéric verfasserin aut Babatasi, Gerard verfasserin aut Blanchart, Katrien verfasserin aut Garnier, Fabien verfasserin aut Bielefeld, Marie verfasserin aut Hamon, David verfasserin aut Lellouche, Nicolas verfasserin aut Bourguignon, Thierry verfasserin aut Genet, Thibaud verfasserin (orcid)0000-0002-2829-7198 aut Eschalier, Romain verfasserin aut D'Ostrevy, Nicolas verfasserin aut Varlet, Emilie verfasserin aut Jouan, Jérôme verfasserin aut Vanhuyse, Fabrice verfasserin aut Blangy, Hugues verfasserin aut Leclercq, Christophe verfasserin aut Martins, Raphaël P. verfasserin aut Enthalten in The American journal of cardiology Amsterdam [u.a.] : Elsevier, 1958 125, Seite 1421-1428 Online-Ressource (DE-627)320596249 (DE-600)2019595-3 (DE-576)10963392X 1879-1913 nnns volume:125 pages:1421-1428 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.85 Kardiologie Angiologie AR 125 1421-1428 |
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10.1016/j.amjcard.2020.01.042 doi (DE-627)ELV003915905 (ELSEVIER)S0002-9149(20)30107-7 DE-627 ger DE-627 rda eng 610 DE-600 44.85 bkl Galand, Vincent verfasserin aut Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. Flécher, Erwan verfasserin aut Chabanne, Céline verfasserin aut Lelong, Bernard verfasserin aut Goéminne, Céline verfasserin aut Vincentelli, André verfasserin (orcid)0000-0003-4081-786X aut Delmas, Clément verfasserin aut Dambrin, Camille verfasserin aut Picard, François verfasserin aut Sacher, Frédéric verfasserin aut Kindo, Michel verfasserin aut Minh, Tam Hoang verfasserin aut Gaudard, Philippe verfasserin aut Rouvière, Philippe verfasserin aut Sénage, Thomas verfasserin aut Michel, Magali verfasserin aut Boignard, Aude verfasserin aut Chavanon, Olivier verfasserin aut Verdonk, Constance verfasserin aut Ghodhbane, Walid verfasserin aut Pelcé, Edeline verfasserin aut Gariboldi, Vlad verfasserin aut Pozzi, Matteo verfasserin (orcid)0000-0001-6550-0872 aut Obadia, Jean-François verfasserin aut Litzler, Pierre-Yvesl verfasserin aut Anselme, Frédéric verfasserin aut Babatasi, Gerard verfasserin aut Blanchart, Katrien verfasserin aut Garnier, Fabien verfasserin aut Bielefeld, Marie verfasserin aut Hamon, David verfasserin aut Lellouche, Nicolas verfasserin aut Bourguignon, Thierry verfasserin aut Genet, Thibaud verfasserin (orcid)0000-0002-2829-7198 aut Eschalier, Romain verfasserin aut D'Ostrevy, Nicolas verfasserin aut Varlet, Emilie verfasserin aut Jouan, Jérôme verfasserin aut Vanhuyse, Fabrice verfasserin aut Blangy, Hugues verfasserin aut Leclercq, Christophe verfasserin aut Martins, Raphaël P. verfasserin aut Enthalten in The American journal of cardiology Amsterdam [u.a.] : Elsevier, 1958 125, Seite 1421-1428 Online-Ressource (DE-627)320596249 (DE-600)2019595-3 (DE-576)10963392X 1879-1913 nnns volume:125 pages:1421-1428 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.85 Kardiologie Angiologie AR 125 1421-1428 |
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10.1016/j.amjcard.2020.01.042 doi (DE-627)ELV003915905 (ELSEVIER)S0002-9149(20)30107-7 DE-627 ger DE-627 rda eng 610 DE-600 44.85 bkl Galand, Vincent verfasserin aut Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. Flécher, Erwan verfasserin aut Chabanne, Céline verfasserin aut Lelong, Bernard verfasserin aut Goéminne, Céline verfasserin aut Vincentelli, André verfasserin (orcid)0000-0003-4081-786X aut Delmas, Clément verfasserin aut Dambrin, Camille verfasserin aut Picard, François verfasserin aut Sacher, Frédéric verfasserin aut Kindo, Michel verfasserin aut Minh, Tam Hoang verfasserin aut Gaudard, Philippe verfasserin aut Rouvière, Philippe verfasserin aut Sénage, Thomas verfasserin aut Michel, Magali verfasserin aut Boignard, Aude verfasserin aut Chavanon, Olivier verfasserin aut Verdonk, Constance verfasserin aut Ghodhbane, Walid verfasserin aut Pelcé, Edeline verfasserin aut Gariboldi, Vlad verfasserin aut Pozzi, Matteo verfasserin (orcid)0000-0001-6550-0872 aut Obadia, Jean-François verfasserin aut Litzler, Pierre-Yvesl verfasserin aut Anselme, Frédéric verfasserin aut Babatasi, Gerard verfasserin aut Blanchart, Katrien verfasserin aut Garnier, Fabien verfasserin aut Bielefeld, Marie verfasserin aut Hamon, David verfasserin aut Lellouche, Nicolas verfasserin aut Bourguignon, Thierry verfasserin aut Genet, Thibaud verfasserin (orcid)0000-0002-2829-7198 aut Eschalier, Romain verfasserin aut D'Ostrevy, Nicolas verfasserin aut Varlet, Emilie verfasserin aut Jouan, Jérôme verfasserin aut Vanhuyse, Fabrice verfasserin aut Blangy, Hugues verfasserin aut Leclercq, Christophe verfasserin aut Martins, Raphaël P. verfasserin aut Enthalten in The American journal of cardiology Amsterdam [u.a.] : Elsevier, 1958 125, Seite 1421-1428 Online-Ressource (DE-627)320596249 (DE-600)2019595-3 (DE-576)10963392X 1879-1913 nnns volume:125 pages:1421-1428 GBV_USEFLAG_U SYSFLAG_U GBV_ELV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2336 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.85 Kardiologie Angiologie AR 125 1421-1428 |
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Galand, Vincent @@aut@@ Flécher, Erwan @@aut@@ Chabanne, Céline @@aut@@ Lelong, Bernard @@aut@@ Goéminne, Céline @@aut@@ Vincentelli, André @@aut@@ Delmas, Clément @@aut@@ Dambrin, Camille @@aut@@ Picard, François @@aut@@ Sacher, Frédéric @@aut@@ Kindo, Michel @@aut@@ Minh, Tam Hoang @@aut@@ Gaudard, Philippe @@aut@@ Rouvière, Philippe @@aut@@ Sénage, Thomas @@aut@@ Michel, Magali @@aut@@ Boignard, Aude @@aut@@ Chavanon, Olivier @@aut@@ Verdonk, Constance @@aut@@ Ghodhbane, Walid @@aut@@ Pelcé, Edeline @@aut@@ Gariboldi, Vlad @@aut@@ Pozzi, Matteo @@aut@@ Obadia, Jean-François @@aut@@ Litzler, Pierre-Yvesl @@aut@@ Anselme, Frédéric @@aut@@ Babatasi, Gerard @@aut@@ Blanchart, Katrien @@aut@@ Garnier, Fabien @@aut@@ Bielefeld, Marie @@aut@@ Hamon, David @@aut@@ Lellouche, Nicolas @@aut@@ Bourguignon, Thierry @@aut@@ Genet, Thibaud @@aut@@ Eschalier, Romain @@aut@@ D'Ostrevy, Nicolas @@aut@@ Varlet, Emilie @@aut@@ Jouan, Jérôme @@aut@@ Vanhuyse, Fabrice @@aut@@ Blangy, Hugues @@aut@@ Leclercq, Christophe @@aut@@ Martins, Raphaël P. @@aut@@ |
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This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. 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Galand, Vincent Flécher, Erwan Chabanne, Céline Lelong, Bernard Goéminne, Céline Vincentelli, André Delmas, Clément Dambrin, Camille Picard, François Sacher, Frédéric Kindo, Michel Minh, Tam Hoang Gaudard, Philippe Rouvière, Philippe Sénage, Thomas Michel, Magali Boignard, Aude Chavanon, Olivier Verdonk, Constance Ghodhbane, Walid Pelcé, Edeline Gariboldi, Vlad Pozzi, Matteo Obadia, Jean-François Litzler, Pierre-Yvesl Anselme, Frédéric Babatasi, Gerard Blanchart, Katrien Garnier, Fabien Bielefeld, Marie Hamon, David Lellouche, Nicolas Bourguignon, Thierry Genet, Thibaud Eschalier, Romain D'Ostrevy, Nicolas Varlet, Emilie Jouan, Jérôme Vanhuyse, Fabrice Blangy, Hugues Leclercq, Christophe Martins, Raphaël P. |
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outcomes of left ventricular assist device implantation in patients with uncommon etiology cardiomyopathy |
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Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy |
| abstract |
The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. |
| abstractGer |
The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. |
| abstract_unstemmed |
The impact of uncommon etiology cardiomyopathies on Left-ventricular assist device (LVAD)-recipient outcomes is not very well known. This study aimed to characterize patients with uncommon cardiomyopathy etiologies and examine the outcomes between uncommon and ischemic/idiopathic dilated cardiomyopathy. This observational study was conducted in 19 centers between 2006 and 2016. Baseline characteristics and outcomes of patients with uncommon etiology were compared to patients with idiopathic dilated/ischemic cardiomyopathies. Among 652 LVAD-recipients included, a total of 590 (90.5%) patients were classified as ischemic/idiopathic and 62 (9.5%) patients were classified in the “uncommon etiologies” group. Main uncommon etiologies were: hypertrophic (n = 12(19%)); cancer therapeutics–related cardiac dysfunction (CTRCD) (n = 12(19%)); myocarditis (n = 11(18%)); valvulopathy (n = 9(15%)) and others (n = 18(29%)). Patients with uncommon etiologies were significantly younger with more female and presented less co-morbidities. Additionally, patients with uncommon cardiomyopathies were less implanted as destination therapy compared with ischemic/idiopathic group (29% vs 38.8%). During a follow-up period of 9.1 months, both groups experienced similar survival. However, subgroup of hypertrophic/valvular cardiomyopathies and CTRCD had significantly higher mortality compared to the ischemic/idiopathic or myocarditis/others cardiomyopathies. Conversely, patients with myocarditis/others etiologies experienced a better survival. Indeed, the 12-months survival in the myocarditis/others; ischemic/idiopathic and hypertrophic/CTRCD/valvulopathy group were 77%; 65%, and 46% respectively. In conclusion, LVAD-recipients with hypertrophic cardiomyopathy, valvular heart disease and CTRCD experienced the higher mortality rate. |
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| title_short |
Outcomes of Left Ventricular Assist Device Implantation in Patients With Uncommon Etiology Cardiomyopathy |
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Flécher, Erwan Chabanne, Céline Lelong, Bernard Goéminne, Céline Vincentelli, André Delmas, Clément Dambrin, Camille Picard, François Sacher, Frédéric Kindo, Michel Minh, Tam Hoang Gaudard, Philippe Rouvière, Philippe Sénage, Thomas Michel, Magali Boignard, Aude Chavanon, Olivier Verdonk, Constance Ghodhbane, Walid Pelcé, Edeline Gariboldi, Vlad Pozzi, Matteo Obadia, Jean-François Litzler, Pierre-Yvesl Anselme, Frédéric Babatasi, Gerard Blanchart, Katrien Garnier, Fabien Bielefeld, Marie Hamon, David Lellouche, Nicolas Bourguignon, Thierry Genet, Thibaud Eschalier, Romain D'Ostrevy, Nicolas Varlet, Emilie Jouan, Jérôme Vanhuyse, Fabrice Blangy, Hugues Leclercq, Christophe Martins, Raphaël P. |
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|
| score |
7.3992968 |