Dengue virus infection and Nrf2 regulation of oxidative stress
Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune r...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Zevini, Alessandra [verfasserIn] Ferrari, Matteo [verfasserIn] Olagnier, David [verfasserIn] Hiscott, John [verfasserIn] |
|---|
| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2020 |
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| Übergeordnetes Werk: |
Enthalten in: No title available - 43, Seite 35-40 |
|---|---|
| Übergeordnetes Werk: |
volume:43 ; pages:35-40 |
| DOI / URN: |
10.1016/j.coviro.2020.07.015 |
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| 520 | |a Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. | ||
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10.1016/j.coviro.2020.07.015 doi (DE-627)ELV004940067 (ELSEVIER)S1879-6257(20)30058-4 DE-627 ger DE-627 rda eng Zevini, Alessandra verfasserin aut Dengue virus infection and Nrf2 regulation of oxidative stress 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. Ferrari, Matteo verfasserin aut Olagnier, David verfasserin aut Hiscott, John verfasserin aut Enthalten in No title available 43, Seite 35-40 (DE-627)ELV000891088 1879-6257 nnns volume:43 pages:35-40 GBV_USEFLAG_U SYSFLAG_U GBV_ELV AR 43 35-40 |
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10.1016/j.coviro.2020.07.015 doi (DE-627)ELV004940067 (ELSEVIER)S1879-6257(20)30058-4 DE-627 ger DE-627 rda eng Zevini, Alessandra verfasserin aut Dengue virus infection and Nrf2 regulation of oxidative stress 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. Ferrari, Matteo verfasserin aut Olagnier, David verfasserin aut Hiscott, John verfasserin aut Enthalten in No title available 43, Seite 35-40 (DE-627)ELV000891088 1879-6257 nnns volume:43 pages:35-40 GBV_USEFLAG_U SYSFLAG_U GBV_ELV AR 43 35-40 |
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10.1016/j.coviro.2020.07.015 doi (DE-627)ELV004940067 (ELSEVIER)S1879-6257(20)30058-4 DE-627 ger DE-627 rda eng Zevini, Alessandra verfasserin aut Dengue virus infection and Nrf2 regulation of oxidative stress 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. Ferrari, Matteo verfasserin aut Olagnier, David verfasserin aut Hiscott, John verfasserin aut Enthalten in No title available 43, Seite 35-40 (DE-627)ELV000891088 1879-6257 nnns volume:43 pages:35-40 GBV_USEFLAG_U SYSFLAG_U GBV_ELV AR 43 35-40 |
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10.1016/j.coviro.2020.07.015 doi (DE-627)ELV004940067 (ELSEVIER)S1879-6257(20)30058-4 DE-627 ger DE-627 rda eng Zevini, Alessandra verfasserin aut Dengue virus infection and Nrf2 regulation of oxidative stress 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. Ferrari, Matteo verfasserin aut Olagnier, David verfasserin aut Hiscott, John verfasserin aut Enthalten in No title available 43, Seite 35-40 (DE-627)ELV000891088 1879-6257 nnns volume:43 pages:35-40 GBV_USEFLAG_U SYSFLAG_U GBV_ELV AR 43 35-40 |
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10.1016/j.coviro.2020.07.015 doi (DE-627)ELV004940067 (ELSEVIER)S1879-6257(20)30058-4 DE-627 ger DE-627 rda eng Zevini, Alessandra verfasserin aut Dengue virus infection and Nrf2 regulation of oxidative stress 2020 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. Ferrari, Matteo verfasserin aut Olagnier, David verfasserin aut Hiscott, John verfasserin aut Enthalten in No title available 43, Seite 35-40 (DE-627)ELV000891088 1879-6257 nnns volume:43 pages:35-40 GBV_USEFLAG_U SYSFLAG_U GBV_ELV AR 43 35-40 |
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Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. |
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Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. |
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Dengue virus is a positive sense, single-stranded RNA virus of the Flaviviridae family that causes mild to severe dengue fever in hundreds of millions of people in tropical/subtropical regions of the world each year. Like many other viruses, dengue has evolved strategies to evade the innate immune response of the host to establish infection. Here we provide an overview of the major alterations provoked by dengue in infected cells, that is, oxidative stress, metabolic reprogramming, and antiviral/inflammatory responses. These biological processes are interconnected and coordinated through the anti-oxidant transcription factor Nrf2, which functions at the interface of metabolism and antiviral immunity. |
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