Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism

Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and...
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Autor*in:	Kim, Hyo Jeong [verfasserIn] Lee, Dong-Yun [verfasserIn] Yoon, Byung-Koo [verfasserIn] Choi, DooSeok [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2016

Schlagwörter:	Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome

Übergeordnetes Werk:	Enthalten in: Journal of pediatric and adolescent gynecology - Amsterdam [u.a.] : Elsevier Science, 1996, 29, Seite 344-347
Übergeordnetes Werk:	volume:29 ; pages:344-347

DOI / URN:	10.1016/j.jpag.2015.11.011

Katalog-ID:	ELV00644718X

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245	1	0	\|a Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism
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520			\|a Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism.
650		4	\|a Hypogonadism
650		4	\|a Estrogen replacement therapy
650		4	\|a Uterus
650		4	\|a Premature ovarian insufficiency
650		4	\|a Turner syndrome
700	1		\|a Lee, Dong-Yun \|e verfasserin \|4 aut
700	1		\|a Yoon, Byung-Koo \|e verfasserin \|4 aut
700	1		\|a Choi, DooSeok \|e verfasserin \|4 aut
773	0	8	\|i Enthalten in \|t Journal of pediatric and adolescent gynecology \|d Amsterdam [u.a.] : Elsevier Science, 1996 \|g 29, Seite 344-347 \|h Online-Ressource \|w (DE-627)324744897 \|w (DE-600)2030480-8 \|w (DE-576)271585323 \|x 1873-4332 \|7 nnns
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publishDate	2016
allfields	10.1016/j.jpag.2015.11.011 doi (DE-627)ELV00644718X (ELSEVIER)S1083-3188(15)00384-8 DE-627 ger DE-627 rda eng 610 DE-600 44.67 bkl Kim, Hyo Jeong verfasserin aut Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism 2016 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism. Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome Lee, Dong-Yun verfasserin aut Yoon, Byung-Koo verfasserin aut Choi, DooSeok verfasserin aut Enthalten in Journal of pediatric and adolescent gynecology Amsterdam [u.a.] : Elsevier Science, 1996 29, Seite 344-347 Online-Ressource (DE-627)324744897 (DE-600)2030480-8 (DE-576)271585323 1873-4332 nnns volume:29 pages:344-347 GBV_USEFLAG_U SYSFLAG_U GBV_ELV GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.67 Kinderheilkunde AR 29 344-347
spelling	10.1016/j.jpag.2015.11.011 doi (DE-627)ELV00644718X (ELSEVIER)S1083-3188(15)00384-8 DE-627 ger DE-627 rda eng 610 DE-600 44.67 bkl Kim, Hyo Jeong verfasserin aut Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism 2016 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism. Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome Lee, Dong-Yun verfasserin aut Yoon, Byung-Koo verfasserin aut Choi, DooSeok verfasserin aut Enthalten in Journal of pediatric and adolescent gynecology Amsterdam [u.a.] : Elsevier Science, 1996 29, Seite 344-347 Online-Ressource (DE-627)324744897 (DE-600)2030480-8 (DE-576)271585323 1873-4332 nnns volume:29 pages:344-347 GBV_USEFLAG_U SYSFLAG_U GBV_ELV GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.67 Kinderheilkunde AR 29 344-347
allfields_unstemmed	10.1016/j.jpag.2015.11.011 doi (DE-627)ELV00644718X (ELSEVIER)S1083-3188(15)00384-8 DE-627 ger DE-627 rda eng 610 DE-600 44.67 bkl Kim, Hyo Jeong verfasserin aut Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism 2016 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism. Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome Lee, Dong-Yun verfasserin aut Yoon, Byung-Koo verfasserin aut Choi, DooSeok verfasserin aut Enthalten in Journal of pediatric and adolescent gynecology Amsterdam [u.a.] : Elsevier Science, 1996 29, Seite 344-347 Online-Ressource (DE-627)324744897 (DE-600)2030480-8 (DE-576)271585323 1873-4332 nnns volume:29 pages:344-347 GBV_USEFLAG_U SYSFLAG_U GBV_ELV GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.67 Kinderheilkunde AR 29 344-347
allfieldsGer	10.1016/j.jpag.2015.11.011 doi (DE-627)ELV00644718X (ELSEVIER)S1083-3188(15)00384-8 DE-627 ger DE-627 rda eng 610 DE-600 44.67 bkl Kim, Hyo Jeong verfasserin aut Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism 2016 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism. Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome Lee, Dong-Yun verfasserin aut Yoon, Byung-Koo verfasserin aut Choi, DooSeok verfasserin aut Enthalten in Journal of pediatric and adolescent gynecology Amsterdam [u.a.] : Elsevier Science, 1996 29, Seite 344-347 Online-Ressource (DE-627)324744897 (DE-600)2030480-8 (DE-576)271585323 1873-4332 nnns volume:29 pages:344-347 GBV_USEFLAG_U SYSFLAG_U GBV_ELV GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.67 Kinderheilkunde AR 29 344-347
allfieldsSound	10.1016/j.jpag.2015.11.011 doi (DE-627)ELV00644718X (ELSEVIER)S1083-3188(15)00384-8 DE-627 ger DE-627 rda eng 610 DE-600 44.67 bkl Kim, Hyo Jeong verfasserin aut Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism 2016 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism. Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome Lee, Dong-Yun verfasserin aut Yoon, Byung-Koo verfasserin aut Choi, DooSeok verfasserin aut Enthalten in Journal of pediatric and adolescent gynecology Amsterdam [u.a.] : Elsevier Science, 1996 29, Seite 344-347 Online-Ressource (DE-627)324744897 (DE-600)2030480-8 (DE-576)271585323 1873-4332 nnns volume:29 pages:344-347 GBV_USEFLAG_U SYSFLAG_U GBV_ELV GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_224 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2008 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2038 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4338 GBV_ILN_4393 44.67 Kinderheilkunde AR 29 344-347
language	English
source	Enthalten in Journal of pediatric and adolescent gynecology 29, Seite 344-347 volume:29 pages:344-347
sourceStr	Enthalten in Journal of pediatric and adolescent gynecology 29, Seite 344-347 volume:29 pages:344-347
format_phy_str_mv	Article
bklname	Kinderheilkunde
institution	findex.gbv.de
topic_facet	Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome
dewey-raw	610
isfreeaccess_bool	false
container_title	Journal of pediatric and adolescent gynecology
authorswithroles_txt_mv	Kim, Hyo Jeong @@aut@@ Lee, Dong-Yun @@aut@@ Yoon, Byung-Koo @@aut@@ Choi, DooSeok @@aut@@
publishDateDaySort_date	2016-01-01T00:00:00Z
hierarchy_top_id	324744897
dewey-sort	3610
id	ELV00644718X
language_de	englisch
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Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. 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author	Kim, Hyo Jeong
spellingShingle	Kim, Hyo Jeong ddc 610 bkl 44.67 misc Hypogonadism misc Estrogen replacement therapy misc Uterus misc Premature ovarian insufficiency misc Turner syndrome Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism
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topic_title	610 DE-600 44.67 bkl Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism Hypogonadism Estrogen replacement therapy Uterus Premature ovarian insufficiency Turner syndrome
topic	ddc 610 bkl 44.67 misc Hypogonadism misc Estrogen replacement therapy misc Uterus misc Premature ovarian insufficiency misc Turner syndrome
topic_unstemmed	ddc 610 bkl 44.67 misc Hypogonadism misc Estrogen replacement therapy misc Uterus misc Premature ovarian insufficiency misc Turner syndrome
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title	Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism
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title_full	Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism
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author_browse	Kim, Hyo Jeong Lee, Dong-Yun Yoon, Byung-Koo Choi, DooSeok
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title_sort	uterine development after estrogen replacement therapy in women with different etiologies of primary hypogonadism
title_auth	Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism
abstract	Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism.
abstractGer	Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism.
abstract_unstemmed	Study Objective: To evaluate uterine development with estrogen replacement therapy in patients with primary amenorrhea due to hypogonadism.Design and Setting: Retrospective study.Participants: Thirty-five women.Interventions and Main Outcome Measures: Women who were younger than 20 years of age and who had primary amenorrhea and an immaturely shaped uterus were included. Changes in uterine cross-sectional area (UXA) and uterine maturity in pelvic ultrasound after 2 year of estrogen replacement therapy were assessed on the basis of the etiology of primary hypogonadism.Results: Patients were classified into three groups according to the etiology of primary hypogonadism: Turner syndrome (n = 19), hypogonadotropic hypogonadism after brain surgery (n = 10), and premature ovarian insufficiency after cancer treatment (n = 6). Overall, the mean UXA significantly increased (from 3.1 ± 1.8 to 11.6 ± 4.9 cm2) after estrogen replacement therapy (P < .001), but the final UXA was significantly smaller in patients with premature ovarian insufficiency compared with other etiologies. In logistic regression analysis, etiology and the cumulative dose of estrogen were associated with uterine maturation (P = .011 and .004, respectively).Conclusion: Estrogen replacement therapy induced growth of the uterus in patients with primary hypogonadism. However, the response to estrogen replacement therapy varied on the basis of the total cumulative dose of estrogen and etiology of primary hypogonadism.
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title_short	Uterine Development After Estrogen Replacement Therapy in Women with Different Etiologies of Primary Hypogonadism
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author2	Lee, Dong-Yun Yoon, Byung-Koo Choi, DooSeok
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up_date	2024-07-06T21:24:28.147Z
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