Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study
Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Soler, J. [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2016transfer abstract |
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| Schlagwörter: |
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| Umfang: |
6 |
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| Übergeordnetes Werk: |
Enthalten in: Modulation of the fluorescent properties of rhodamine 6G by Zn - Alhasani, Mona ELSEVIER, 2017, the official journal of the Association of European Psychiatrists (AEP), Amsterdam [u.a.] |
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| Übergeordnetes Werk: |
volume:32 ; year:2016 ; pages:42-47 ; extent:6 |
| Links: |
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| DOI / URN: |
10.1016/j.eurpsy.2015.11.002 |
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| 520 | |a Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. | ||
| 520 | |a Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. | ||
| 650 | 7 | |a Executive function |2 Elsevier | |
| 650 | 7 | |a DAOA |2 Elsevier | |
| 650 | 7 | |a Schizophrenia |2 Elsevier | |
| 650 | 7 | |a Bipolar disorder |2 Elsevier | |
| 650 | 7 | |a RGS4 |2 Elsevier | |
| 650 | 7 | |a Schizophrenia-spectrum disorders |2 Elsevier | |
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| 700 | 1 | |a Lázaro, L. |4 oth | |
| 700 | 1 | |a Parellada, M. |4 oth | |
| 700 | 1 | |a Martín, M. |4 oth | |
| 700 | 1 | |a Lera-Miguel, S. |4 oth | |
| 700 | 1 | |a Rosa, A. |4 oth | |
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10.1016/j.eurpsy.2015.11.002 doi GBVA2016014000026.pica (DE-627)ELV014226480 (ELSEVIER)S0924-9338(15)00679-3 DE-627 ger DE-627 rakwb eng 610 610 DE-600 530 VZ 33.00 bkl Soler, J. verfasserin aut Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study 2016transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Executive function Elsevier DAOA Elsevier Schizophrenia Elsevier Bipolar disorder Elsevier RGS4 Elsevier Schizophrenia-spectrum disorders Elsevier Miret, S. oth Lázaro, L. oth Parellada, M. oth Martín, M. oth Lera-Miguel, S. oth Rosa, A. oth de Castro-Catala, M. oth Cuesta, M.J. oth Fañanás, L. oth Krebs, M.O. oth Fatjó-Vilas, M. oth Enthalten in Elsevier Science Alhasani, Mona ELSEVIER Modulation of the fluorescent properties of rhodamine 6G by Zn 2017 the official journal of the Association of European Psychiatrists (AEP) Amsterdam [u.a.] (DE-627)ELV000186759 volume:32 year:2016 pages:42-47 extent:6 https://doi.org/10.1016/j.eurpsy.2015.11.002 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 33.00 Physik: Allgemeines VZ AR 32 2016 42-47 6 045F 610 |
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10.1016/j.eurpsy.2015.11.002 doi GBVA2016014000026.pica (DE-627)ELV014226480 (ELSEVIER)S0924-9338(15)00679-3 DE-627 ger DE-627 rakwb eng 610 610 DE-600 530 VZ 33.00 bkl Soler, J. verfasserin aut Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study 2016transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Executive function Elsevier DAOA Elsevier Schizophrenia Elsevier Bipolar disorder Elsevier RGS4 Elsevier Schizophrenia-spectrum disorders Elsevier Miret, S. oth Lázaro, L. oth Parellada, M. oth Martín, M. oth Lera-Miguel, S. oth Rosa, A. oth de Castro-Catala, M. oth Cuesta, M.J. oth Fañanás, L. oth Krebs, M.O. oth Fatjó-Vilas, M. oth Enthalten in Elsevier Science Alhasani, Mona ELSEVIER Modulation of the fluorescent properties of rhodamine 6G by Zn 2017 the official journal of the Association of European Psychiatrists (AEP) Amsterdam [u.a.] (DE-627)ELV000186759 volume:32 year:2016 pages:42-47 extent:6 https://doi.org/10.1016/j.eurpsy.2015.11.002 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 33.00 Physik: Allgemeines VZ AR 32 2016 42-47 6 045F 610 |
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10.1016/j.eurpsy.2015.11.002 doi GBVA2016014000026.pica (DE-627)ELV014226480 (ELSEVIER)S0924-9338(15)00679-3 DE-627 ger DE-627 rakwb eng 610 610 DE-600 530 VZ 33.00 bkl Soler, J. verfasserin aut Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study 2016transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Executive function Elsevier DAOA Elsevier Schizophrenia Elsevier Bipolar disorder Elsevier RGS4 Elsevier Schizophrenia-spectrum disorders Elsevier Miret, S. oth Lázaro, L. oth Parellada, M. oth Martín, M. oth Lera-Miguel, S. oth Rosa, A. oth de Castro-Catala, M. oth Cuesta, M.J. oth Fañanás, L. oth Krebs, M.O. oth Fatjó-Vilas, M. oth Enthalten in Elsevier Science Alhasani, Mona ELSEVIER Modulation of the fluorescent properties of rhodamine 6G by Zn 2017 the official journal of the Association of European Psychiatrists (AEP) Amsterdam [u.a.] (DE-627)ELV000186759 volume:32 year:2016 pages:42-47 extent:6 https://doi.org/10.1016/j.eurpsy.2015.11.002 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 33.00 Physik: Allgemeines VZ AR 32 2016 42-47 6 045F 610 |
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10.1016/j.eurpsy.2015.11.002 doi GBVA2016014000026.pica (DE-627)ELV014226480 (ELSEVIER)S0924-9338(15)00679-3 DE-627 ger DE-627 rakwb eng 610 610 DE-600 530 VZ 33.00 bkl Soler, J. verfasserin aut Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study 2016transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Executive function Elsevier DAOA Elsevier Schizophrenia Elsevier Bipolar disorder Elsevier RGS4 Elsevier Schizophrenia-spectrum disorders Elsevier Miret, S. oth Lázaro, L. oth Parellada, M. oth Martín, M. oth Lera-Miguel, S. oth Rosa, A. oth de Castro-Catala, M. oth Cuesta, M.J. oth Fañanás, L. oth Krebs, M.O. oth Fatjó-Vilas, M. oth Enthalten in Elsevier Science Alhasani, Mona ELSEVIER Modulation of the fluorescent properties of rhodamine 6G by Zn 2017 the official journal of the Association of European Psychiatrists (AEP) Amsterdam [u.a.] (DE-627)ELV000186759 volume:32 year:2016 pages:42-47 extent:6 https://doi.org/10.1016/j.eurpsy.2015.11.002 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 33.00 Physik: Allgemeines VZ AR 32 2016 42-47 6 045F 610 |
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10.1016/j.eurpsy.2015.11.002 doi GBVA2016014000026.pica (DE-627)ELV014226480 (ELSEVIER)S0924-9338(15)00679-3 DE-627 ger DE-627 rakwb eng 610 610 DE-600 530 VZ 33.00 bkl Soler, J. verfasserin aut Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study 2016transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. Executive function Elsevier DAOA Elsevier Schizophrenia Elsevier Bipolar disorder Elsevier RGS4 Elsevier Schizophrenia-spectrum disorders Elsevier Miret, S. oth Lázaro, L. oth Parellada, M. oth Martín, M. oth Lera-Miguel, S. oth Rosa, A. oth de Castro-Catala, M. oth Cuesta, M.J. oth Fañanás, L. oth Krebs, M.O. oth Fatjó-Vilas, M. oth Enthalten in Elsevier Science Alhasani, Mona ELSEVIER Modulation of the fluorescent properties of rhodamine 6G by Zn 2017 the official journal of the Association of European Psychiatrists (AEP) Amsterdam [u.a.] (DE-627)ELV000186759 volume:32 year:2016 pages:42-47 extent:6 https://doi.org/10.1016/j.eurpsy.2015.11.002 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 33.00 Physik: Allgemeines VZ AR 32 2016 42-47 6 045F 610 |
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Influence of DAOA and RGS4 genes on the risk for psychotic disorders and their associated executive dysfunctions: A family-based study |
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Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. |
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Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. |
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Glutamatergic neurotransmission dysfunction has classically been related to the aetiology of psychotic disorders. A substantial polygenic component shared across these disorders has been reported and molecular genetics studies have associated glutamatergic-related genes, such as d-amino acid oxidase activator (DAOA) and regulator of G-protein signalling 4 (RGS4) with the risk for psychotic disorders. Our aims were to examine: (i) the relationship between DAOA and RGS4 and the risk for psychotic disorders using a family-based association approach, and (ii) whether variations in these genes are associated with differences in patients’ cognitive performance. |
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