Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters
In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be ex...
Ausführliche Beschreibung
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| Autor*in: |
Blewett, Tamzin A. [verfasserIn] |
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E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2017transfer abstract |
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| Umfang: |
12 |
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| Übergeordnetes Werk: |
Enthalten in: Structural failure performance of the encased functionally graded porous cylinder consolidated by graphene platelet under uniform radial loading - Li, Zhaochao ELSEVIER, 2019, Amsterdam [u.a.] |
|---|---|
| Übergeordnetes Werk: |
volume:223 ; year:2017 ; pages:311-322 ; extent:12 |
| Links: |
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| DOI / URN: |
10.1016/j.envpol.2017.01.028 |
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| 520 | |a In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. | ||
| 520 | |a In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. | ||
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10.1016/j.envpol.2017.01.028 doi GBV00000000000110A.pica (DE-627)ELV014969157 (ELSEVIER)S0269-7491(16)31776-6 DE-627 ger DE-627 rakwb eng 333.7 570 690 333.7 DE-600 570 DE-600 690 DE-600 690 VZ 50.31 bkl 56.11 bkl Blewett, Tamzin A. verfasserin aut Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters 2017transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. Leonard, Erin M. oth Enthalten in Elsevier Science Li, Zhaochao ELSEVIER Structural failure performance of the encased functionally graded porous cylinder consolidated by graphene platelet under uniform radial loading 2019 Amsterdam [u.a.] (DE-627)ELV00327988X volume:223 year:2017 pages:311-322 extent:12 https://doi.org/10.1016/j.envpol.2017.01.028 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 50.31 Technische Mechanik VZ 56.11 Baukonstruktion VZ AR 223 2017 311-322 12 045F 333.7 |
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10.1016/j.envpol.2017.01.028 doi GBV00000000000110A.pica (DE-627)ELV014969157 (ELSEVIER)S0269-7491(16)31776-6 DE-627 ger DE-627 rakwb eng 333.7 570 690 333.7 DE-600 570 DE-600 690 DE-600 690 VZ 50.31 bkl 56.11 bkl Blewett, Tamzin A. verfasserin aut Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters 2017transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. Leonard, Erin M. oth Enthalten in Elsevier Science Li, Zhaochao ELSEVIER Structural failure performance of the encased functionally graded porous cylinder consolidated by graphene platelet under uniform radial loading 2019 Amsterdam [u.a.] (DE-627)ELV00327988X volume:223 year:2017 pages:311-322 extent:12 https://doi.org/10.1016/j.envpol.2017.01.028 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 50.31 Technische Mechanik VZ 56.11 Baukonstruktion VZ AR 223 2017 311-322 12 045F 333.7 |
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10.1016/j.envpol.2017.01.028 doi GBV00000000000110A.pica (DE-627)ELV014969157 (ELSEVIER)S0269-7491(16)31776-6 DE-627 ger DE-627 rakwb eng 333.7 570 690 333.7 DE-600 570 DE-600 690 DE-600 690 VZ 50.31 bkl 56.11 bkl Blewett, Tamzin A. verfasserin aut Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters 2017transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. Leonard, Erin M. oth Enthalten in Elsevier Science Li, Zhaochao ELSEVIER Structural failure performance of the encased functionally graded porous cylinder consolidated by graphene platelet under uniform radial loading 2019 Amsterdam [u.a.] (DE-627)ELV00327988X volume:223 year:2017 pages:311-322 extent:12 https://doi.org/10.1016/j.envpol.2017.01.028 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 50.31 Technische Mechanik VZ 56.11 Baukonstruktion VZ AR 223 2017 311-322 12 045F 333.7 |
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Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters |
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Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters |
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mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters |
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Mechanisms of nickel toxicity to fish and invertebrates in marine and estuarine waters |
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In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. |
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In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. |
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In freshwater settings the toxicity of the trace metal nickel (Ni) is relatively well understood. However, until recently, there was little knowledge regarding Ni toxicity in waters of higher salinity, where factors such as water chemistry and the physiology of estuarine and marine biota would be expected to alter toxicological impact. This review summarizes recent literature investigating Ni toxicity in marine and estuarine invertebrates and fish. As in freshwater, three main mechanisms of Ni toxicity exist: ionoregulatory impairment, inhibition of respiration, and promotion of oxidative stress. However, unlike in freshwater biota, where mechanisms of toxicity are largely Class-specific, the delineation of toxic mechanisms between different species is less defined. In general, despite changes in Ni speciation in marine waters, organism physiology appears to be the main driver of toxic impact, a fact that will need to be accounted for when adapting regulatory tools (such as bioavailability normalization) from freshwater to estuarine and marine environments. |
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