New perspectives on target intraocular pressure
The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure...
Ausführliche Beschreibung
Autor*in: |
Clement, Colin I. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2014transfer abstract |
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Schlagwörter: |
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Umfang: |
12 |
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Übergeordnetes Werk: |
Enthalten in: Supraglottic subepithelial benign mass lesions: Focus on clinical features of sialolipoma-like lesion - Tachibana, Tomoyasu ELSEVIER, 2020, Amsterdam [u.a.] |
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Übergeordnetes Werk: |
volume:59 ; year:2014 ; number:6 ; pages:615-626 ; extent:12 |
Links: |
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DOI / URN: |
10.1016/j.survophthal.2014.04.001 |
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ELV017187257 |
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520 | |a The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. | ||
520 | |a The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. | ||
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10.1016/j.survophthal.2014.04.001 doi GBVA2014001000013.pica (DE-627)ELV017187257 (ELSEVIER)S0039-6257(14)00076-9 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.94 bkl Clement, Colin I. verfasserin aut New perspectives on target intraocular pressure 2014transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. intraocular pressure Elsevier glaucoma Elsevier Bhartiya, Shibal oth Shaarawy, Tarek oth Enthalten in Elsevier Science Tachibana, Tomoyasu ELSEVIER Supraglottic subepithelial benign mass lesions: Focus on clinical features of sialolipoma-like lesion 2020 Amsterdam [u.a.] (DE-627)ELV005493498 volume:59 year:2014 number:6 pages:615-626 extent:12 https://doi.org/10.1016/j.survophthal.2014.04.001 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 59 2014 6 615-626 12 045F 610 |
spelling |
10.1016/j.survophthal.2014.04.001 doi GBVA2014001000013.pica (DE-627)ELV017187257 (ELSEVIER)S0039-6257(14)00076-9 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.94 bkl Clement, Colin I. verfasserin aut New perspectives on target intraocular pressure 2014transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. intraocular pressure Elsevier glaucoma Elsevier Bhartiya, Shibal oth Shaarawy, Tarek oth Enthalten in Elsevier Science Tachibana, Tomoyasu ELSEVIER Supraglottic subepithelial benign mass lesions: Focus on clinical features of sialolipoma-like lesion 2020 Amsterdam [u.a.] (DE-627)ELV005493498 volume:59 year:2014 number:6 pages:615-626 extent:12 https://doi.org/10.1016/j.survophthal.2014.04.001 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 59 2014 6 615-626 12 045F 610 |
allfields_unstemmed |
10.1016/j.survophthal.2014.04.001 doi GBVA2014001000013.pica (DE-627)ELV017187257 (ELSEVIER)S0039-6257(14)00076-9 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.94 bkl Clement, Colin I. verfasserin aut New perspectives on target intraocular pressure 2014transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. intraocular pressure Elsevier glaucoma Elsevier Bhartiya, Shibal oth Shaarawy, Tarek oth Enthalten in Elsevier Science Tachibana, Tomoyasu ELSEVIER Supraglottic subepithelial benign mass lesions: Focus on clinical features of sialolipoma-like lesion 2020 Amsterdam [u.a.] (DE-627)ELV005493498 volume:59 year:2014 number:6 pages:615-626 extent:12 https://doi.org/10.1016/j.survophthal.2014.04.001 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 59 2014 6 615-626 12 045F 610 |
allfieldsGer |
10.1016/j.survophthal.2014.04.001 doi GBVA2014001000013.pica (DE-627)ELV017187257 (ELSEVIER)S0039-6257(14)00076-9 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.94 bkl Clement, Colin I. verfasserin aut New perspectives on target intraocular pressure 2014transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. intraocular pressure Elsevier glaucoma Elsevier Bhartiya, Shibal oth Shaarawy, Tarek oth Enthalten in Elsevier Science Tachibana, Tomoyasu ELSEVIER Supraglottic subepithelial benign mass lesions: Focus on clinical features of sialolipoma-like lesion 2020 Amsterdam [u.a.] (DE-627)ELV005493498 volume:59 year:2014 number:6 pages:615-626 extent:12 https://doi.org/10.1016/j.survophthal.2014.04.001 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 59 2014 6 615-626 12 045F 610 |
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10.1016/j.survophthal.2014.04.001 doi GBVA2014001000013.pica (DE-627)ELV017187257 (ELSEVIER)S0039-6257(14)00076-9 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.94 bkl Clement, Colin I. verfasserin aut New perspectives on target intraocular pressure 2014transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. intraocular pressure Elsevier glaucoma Elsevier Bhartiya, Shibal oth Shaarawy, Tarek oth Enthalten in Elsevier Science Tachibana, Tomoyasu ELSEVIER Supraglottic subepithelial benign mass lesions: Focus on clinical features of sialolipoma-like lesion 2020 Amsterdam [u.a.] (DE-627)ELV005493498 volume:59 year:2014 number:6 pages:615-626 extent:12 https://doi.org/10.1016/j.survophthal.2014.04.001 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 59 2014 6 615-626 12 045F 610 |
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New perspectives on target intraocular pressure |
abstract |
The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. |
abstractGer |
The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. |
abstract_unstemmed |
The evidence in support of intraocular pressure (IOP) lowering to reduce risk of glaucoma onset or progression is strong, although the amount and quality of IOP reduction is less well defined. The concept of a target IOP includes a percentage reduction, calculated IOP, or a predetermined IOP figure or range. Yet none of these strategies have been validated. In addition, our understanding of the way IOP influences glaucoma risk is continuously evolving. Examples of this include the data on IOP fluctuation and lamina cribrosa and cerebrospinal fluid pressure differentials. That these variables are not included in target IOP calculation potentially undermines its accuracy and usefulness. We summarize the evidence for target IOP, new developments in our understanding of IOP and glaucoma pathogenesis, as well as emerging strategies for setting targets and assessing response to treatment. |
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title_short |
New perspectives on target intraocular pressure |
url |
https://doi.org/10.1016/j.survophthal.2014.04.001 |
remote_bool |
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author2 |
Bhartiya, Shibal Shaarawy, Tarek |
author2Str |
Bhartiya, Shibal Shaarawy, Tarek |
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doi_str |
10.1016/j.survophthal.2014.04.001 |
up_date |
2024-07-06T21:20:33.932Z |
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