Anthrax toxin receptor 2 promotes human uterine smooth muscle cell viability, migration and contractility
Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2 −/−) mice are fertile but fail to deliver their pups at term. This parturition defect is associated with overaccumulation of extracellular matrix proteins and decreased myometrial cell content in the uterus. Myometrial cell loss in...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Vink, Joy Yumiko [verfasserIn] |
|---|
| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2014 |
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| Schlagwörter: |
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| Umfang: |
8 |
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| Übergeordnetes Werk: |
Enthalten in: IS - Domingues, Sara ELSEVIER, 2018, AJOG, Orlando, Fla |
|---|---|
| Übergeordnetes Werk: |
volume:210 ; year:2014 ; number:2 ; pages:1541-1548 ; extent:8 |
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| DOI / URN: |
10.1016/j.ajog.2013.09.030 |
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| 520 | |a Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2 −/−) mice are fertile but fail to deliver their pups at term. This parturition defect is associated with overaccumulation of extracellular matrix proteins and decreased myometrial cell content in the uterus. Myometrial cell loss in Antxr2 −/− uterine tissue prompted us to evaluate if ANTXR2 is essential for human uterine smooth muscle cell viability and function. | ||
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10.1016/j.ajog.2013.09.030 doi GBVA2014019000017.pica (DE-627)ELV017919118 (ELSEVIER)S0002-9378(13)00984-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 570 VZ BIODIV DE-30 fid Vink, Joy Yumiko verfasserin aut Anthrax toxin receptor 2 promotes human uterine smooth muscle cell viability, migration and contractility 2014 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2 −/−) mice are fertile but fail to deliver their pups at term. This parturition defect is associated with overaccumulation of extracellular matrix proteins and decreased myometrial cell content in the uterus. Myometrial cell loss in Antxr2 −/− uterine tissue prompted us to evaluate if ANTXR2 is essential for human uterine smooth muscle cell viability and function. anthrax toxin receptor 2 Elsevier uterine smooth muscle cells Elsevier contractility Elsevier migration Elsevier apoptosis Elsevier extracellular matrix Elsevier Charles-Horvath, Pelisa Cheryll oth Kitajewski, Jan Krzysztof oth Reeves, Claire Vech oth Enthalten in Elsevier Domingues, Sara ELSEVIER IS 2018 AJOG Orlando, Fla (DE-627)ELV000288284 volume:210 year:2014 number:2 pages:1541-1548 extent:8 https://doi.org/10.1016/j.ajog.2013.09.030 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U FID-BIODIV SSG-OLC-PHA AR 210 2014 2 1541-1548 8 210.2014, 2, 154.e1-, (8 S.) 045F 610 |
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Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2 −/−) mice are fertile but fail to deliver their pups at term. This parturition defect is associated with overaccumulation of extracellular matrix proteins and decreased myometrial cell content in the uterus. Myometrial cell loss in Antxr2 −/− uterine tissue prompted us to evaluate if ANTXR2 is essential for human uterine smooth muscle cell viability and function. |
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Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2 −/−) mice are fertile but fail to deliver their pups at term. This parturition defect is associated with overaccumulation of extracellular matrix proteins and decreased myometrial cell content in the uterus. Myometrial cell loss in Antxr2 −/− uterine tissue prompted us to evaluate if ANTXR2 is essential for human uterine smooth muscle cell viability and function. |
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Previously we demonstrated anthrax toxin receptor 2 knockout (Antxr2 −/−) mice are fertile but fail to deliver their pups at term. This parturition defect is associated with overaccumulation of extracellular matrix proteins and decreased myometrial cell content in the uterus. Myometrial cell loss in Antxr2 −/− uterine tissue prompted us to evaluate if ANTXR2 is essential for human uterine smooth muscle cell viability and function. |
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