ID: 91
Introduction: Mechanical stress-induced chronic hypertension can initiate the development of cardiac hypertrophy and, ultimately, congestive heart failure. It is widely accepted that various chemokines are expressed in the heart in response to pressure overload. These chemokines might be involved in...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Kimura, Akihiko [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2015 |
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| Übergeordnetes Werk: |
Enthalten in: The minimal clinically important differences of the Simple Shoulder Test are different for different arthroplasty types - McLaughlin, Richard J. ELSEVIER, 2022, the official journal of the International Cytokine Society, Oxford [u.a.] |
|---|---|
| Übergeordnetes Werk: |
volume:76 ; year:2015 ; number:1 ; pages:82 |
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| DOI / URN: |
10.1016/j.cyto.2015.08.118 |
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| 520 | |a Introduction: Mechanical stress-induced chronic hypertension can initiate the development of cardiac hypertrophy and, ultimately, congestive heart failure. It is widely accepted that various chemokines are expressed in the heart in response to pressure overload. These chemokines might be involved in the cardiac remodeling and pathogenesis of heart failure. In this study, we investigated the roles of CCR5 in pressure overload-induced cardiac hypertrophy and heart failure in mice. | ||
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