Imaging of cellular aging in human retinal blood vessels
To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessel...
Ausführliche Beschreibung
Autor*in: |
Catita, J. [verfasserIn] |
---|
Format: |
E-Artikel |
---|---|
Sprache: |
Englisch |
Erschienen: |
2015transfer abstract |
---|
Schlagwörter: |
---|
Umfang: |
12 |
---|
Übergeordnetes Werk: |
Enthalten in: Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary - Billo, Georis ELSEVIER, 2022, official journal of the ISER, Amsterdam [u.a.] |
---|---|
Übergeordnetes Werk: |
volume:135 ; year:2015 ; pages:14-25 ; extent:12 |
Links: |
---|
DOI / URN: |
10.1016/j.exer.2015.03.019 |
---|
Katalog-ID: |
ELV018910971 |
---|
LEADER | 01000caa a22002652 4500 | ||
---|---|---|---|
001 | ELV018910971 | ||
003 | DE-627 | ||
005 | 20230625124919.0 | ||
007 | cr uuu---uuuuu | ||
008 | 180603s2015 xx |||||o 00| ||eng c | ||
024 | 7 | |a 10.1016/j.exer.2015.03.019 |2 doi | |
028 | 5 | 2 | |a GBVA2015021000010.pica |
035 | |a (DE-627)ELV018910971 | ||
035 | |a (ELSEVIER)S0014-4835(15)00109-8 | ||
040 | |a DE-627 |b ger |c DE-627 |e rakwb | ||
041 | |a eng | ||
082 | 0 | |a 610 | |
082 | 0 | 4 | |a 610 |q DE-600 |
082 | 0 | 4 | |a 510 |a 004 |q VZ |
084 | |a 31.80 |2 bkl | ||
084 | |a 54.80 |2 bkl | ||
100 | 1 | |a Catita, J. |e verfasserin |4 aut | |
245 | 1 | 0 | |a Imaging of cellular aging in human retinal blood vessels |
264 | 1 | |c 2015transfer abstract | |
300 | |a 12 | ||
336 | |a nicht spezifiziert |b zzz |2 rdacontent | ||
337 | |a nicht spezifiziert |b z |2 rdamedia | ||
338 | |a nicht spezifiziert |b zu |2 rdacarrier | ||
520 | |a To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. | ||
520 | |a To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. | ||
650 | 7 | |a Microaneurysms |2 Elsevier | |
650 | 7 | |a Cellular senescence |2 Elsevier | |
650 | 7 | |a Aging |2 Elsevier | |
650 | 7 | |a Lipofuscin accumulation |2 Elsevier | |
650 | 7 | |a Acellular capillaries |2 Elsevier | |
700 | 1 | |a López-Luppo, M. |4 oth | |
700 | 1 | |a Ramos, D. |4 oth | |
700 | 1 | |a Nacher, V. |4 oth | |
700 | 1 | |a Navarro, M. |4 oth | |
700 | 1 | |a Carretero, A. |4 oth | |
700 | 1 | |a Sánchez-Chardi, A. |4 oth | |
700 | 1 | |a Mendes-Jorge, L. |4 oth | |
700 | 1 | |a Rodriguez-Baeza, A. |4 oth | |
700 | 1 | |a Ruberte, J. |4 oth | |
773 | 0 | 8 | |i Enthalten in |n Elsevier |a Billo, Georis ELSEVIER |t Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |d 2022 |d official journal of the ISER |g Amsterdam [u.a.] |w (DE-627)ELV008415374 |
773 | 1 | 8 | |g volume:135 |g year:2015 |g pages:14-25 |g extent:12 |
856 | 4 | 0 | |u https://doi.org/10.1016/j.exer.2015.03.019 |3 Volltext |
912 | |a GBV_USEFLAG_U | ||
912 | |a GBV_ELV | ||
912 | |a SYSFLAG_U | ||
912 | |a SSG-OPC-MAT | ||
936 | b | k | |a 31.80 |j Angewandte Mathematik |q VZ |
936 | b | k | |a 54.80 |j Angewandte Informatik |q VZ |
951 | |a AR | ||
952 | |d 135 |j 2015 |h 14-25 |g 12 | ||
953 | |2 045F |a 610 |
author_variant |
j c jc |
---|---|
matchkey_str |
catitajlpezluppomramosdnachervnavarromca:2015----:mgnocluaaignuartnl |
hierarchy_sort_str |
2015transfer abstract |
bklnumber |
31.80 54.80 |
publishDate |
2015 |
allfields |
10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
spelling |
10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
allfields_unstemmed |
10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
allfieldsGer |
10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
allfieldsSound |
10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
language |
English |
source |
Enthalten in Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary Amsterdam [u.a.] volume:135 year:2015 pages:14-25 extent:12 |
sourceStr |
Enthalten in Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary Amsterdam [u.a.] volume:135 year:2015 pages:14-25 extent:12 |
format_phy_str_mv |
Article |
bklname |
Angewandte Mathematik Angewandte Informatik |
institution |
findex.gbv.de |
topic_facet |
Microaneurysms Cellular senescence Aging Lipofuscin accumulation Acellular capillaries |
dewey-raw |
610 |
isfreeaccess_bool |
false |
container_title |
Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |
authorswithroles_txt_mv |
Catita, J. @@aut@@ López-Luppo, M. @@oth@@ Ramos, D. @@oth@@ Nacher, V. @@oth@@ Navarro, M. @@oth@@ Carretero, A. @@oth@@ Sánchez-Chardi, A. @@oth@@ Mendes-Jorge, L. @@oth@@ Rodriguez-Baeza, A. @@oth@@ Ruberte, J. @@oth@@ |
publishDateDaySort_date |
2015-01-01T00:00:00Z |
hierarchy_top_id |
ELV008415374 |
dewey-sort |
3610 |
id |
ELV018910971 |
language_de |
englisch |
fullrecord |
<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">ELV018910971</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230625124919.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">180603s2015 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1016/j.exer.2015.03.019</subfield><subfield code="2">doi</subfield></datafield><datafield tag="028" ind1="5" ind2="2"><subfield code="a">GBVA2015021000010.pica</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)ELV018910971</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ELSEVIER)S0014-4835(15)00109-8</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2=" "><subfield code="a">610</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="q">DE-600</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">510</subfield><subfield code="a">004</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">31.80</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">54.80</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Catita, J.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Imaging of cellular aging in human retinal blood vessels</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2015transfer abstract</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">12</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels.</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels.</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Microaneurysms</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Cellular senescence</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Aging</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Lipofuscin accumulation</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Acellular capillaries</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">López-Luppo, M.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Ramos, D.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Nacher, V.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Navarro, M.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Carretero, A.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Sánchez-Chardi, A.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Mendes-Jorge, L.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Rodriguez-Baeza, A.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Ruberte, J.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="n">Elsevier</subfield><subfield code="a">Billo, Georis ELSEVIER</subfield><subfield code="t">Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary</subfield><subfield code="d">2022</subfield><subfield code="d">official journal of the ISER</subfield><subfield code="g">Amsterdam [u.a.]</subfield><subfield code="w">(DE-627)ELV008415374</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:135</subfield><subfield code="g">year:2015</subfield><subfield code="g">pages:14-25</subfield><subfield code="g">extent:12</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1016/j.exer.2015.03.019</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ELV</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OPC-MAT</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">31.80</subfield><subfield code="j">Angewandte Mathematik</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">54.80</subfield><subfield code="j">Angewandte Informatik</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">135</subfield><subfield code="j">2015</subfield><subfield code="h">14-25</subfield><subfield code="g">12</subfield></datafield><datafield tag="953" ind1=" " ind2=" "><subfield code="2">045F</subfield><subfield code="a">610</subfield></datafield></record></collection>
|
author |
Catita, J. |
spellingShingle |
Catita, J. ddc 610 ddc 510 bkl 31.80 bkl 54.80 Elsevier Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Imaging of cellular aging in human retinal blood vessels |
authorStr |
Catita, J. |
ppnlink_with_tag_str_mv |
@@773@@(DE-627)ELV008415374 |
format |
electronic Article |
dewey-ones |
610 - Medicine & health 510 - Mathematics 004 - Data processing & computer science |
delete_txt_mv |
keep |
author_role |
aut |
collection |
elsevier |
remote_str |
true |
illustrated |
Not Illustrated |
topic_title |
610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Imaging of cellular aging in human retinal blood vessels Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier |
topic |
ddc 610 ddc 510 bkl 31.80 bkl 54.80 Elsevier Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries |
topic_unstemmed |
ddc 610 ddc 510 bkl 31.80 bkl 54.80 Elsevier Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries |
topic_browse |
ddc 610 ddc 510 bkl 31.80 bkl 54.80 Elsevier Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries |
format_facet |
Elektronische Aufsätze Aufsätze Elektronische Ressource |
format_main_str_mv |
Text Zeitschrift/Artikel |
carriertype_str_mv |
zu |
author2_variant |
m l l mll d r dr v n vn m n mn a c ac a s c asc l m j lmj a r b arb j r jr |
hierarchy_parent_title |
Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |
hierarchy_parent_id |
ELV008415374 |
dewey-tens |
610 - Medicine & health 510 - Mathematics 000 - Computer science, knowledge & systems |
hierarchy_top_title |
Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |
isfreeaccess_txt |
false |
familylinks_str_mv |
(DE-627)ELV008415374 |
title |
Imaging of cellular aging in human retinal blood vessels |
ctrlnum |
(DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 |
title_full |
Imaging of cellular aging in human retinal blood vessels |
author_sort |
Catita, J. |
journal |
Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |
journalStr |
Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |
lang_code |
eng |
isOA_bool |
false |
dewey-hundreds |
600 - Technology 500 - Science 000 - Computer science, information & general works |
recordtype |
marc |
publishDateSort |
2015 |
contenttype_str_mv |
zzz |
container_start_page |
14 |
author_browse |
Catita, J. |
container_volume |
135 |
physical |
12 |
class |
610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl |
format_se |
Elektronische Aufsätze |
author-letter |
Catita, J. |
doi_str_mv |
10.1016/j.exer.2015.03.019 |
dewey-full |
610 510 004 |
title_sort |
imaging of cellular aging in human retinal blood vessels |
title_auth |
Imaging of cellular aging in human retinal blood vessels |
abstract |
To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. |
abstractGer |
To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. |
abstract_unstemmed |
To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. |
collection_details |
GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT |
title_short |
Imaging of cellular aging in human retinal blood vessels |
url |
https://doi.org/10.1016/j.exer.2015.03.019 |
remote_bool |
true |
author2 |
López-Luppo, M. Ramos, D. Nacher, V. Navarro, M. Carretero, A. Sánchez-Chardi, A. Mendes-Jorge, L. Rodriguez-Baeza, A. Ruberte, J. |
author2Str |
López-Luppo, M. Ramos, D. Nacher, V. Navarro, M. Carretero, A. Sánchez-Chardi, A. Mendes-Jorge, L. Rodriguez-Baeza, A. Ruberte, J. |
ppnlink |
ELV008415374 |
mediatype_str_mv |
z |
isOA_txt |
false |
hochschulschrift_bool |
false |
author2_role |
oth oth oth oth oth oth oth oth oth |
doi_str |
10.1016/j.exer.2015.03.019 |
up_date |
2024-07-06T20:03:41.265Z |
_version_ |
1803861330509168640 |
fullrecord_marcxml |
<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">ELV018910971</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230625124919.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">180603s2015 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1016/j.exer.2015.03.019</subfield><subfield code="2">doi</subfield></datafield><datafield tag="028" ind1="5" ind2="2"><subfield code="a">GBVA2015021000010.pica</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)ELV018910971</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ELSEVIER)S0014-4835(15)00109-8</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2=" "><subfield code="a">610</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="q">DE-600</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">510</subfield><subfield code="a">004</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">31.80</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">54.80</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Catita, J.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Imaging of cellular aging in human retinal blood vessels</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2015transfer abstract</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">12</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels.</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels.</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Microaneurysms</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Cellular senescence</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Aging</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Lipofuscin accumulation</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Acellular capillaries</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">López-Luppo, M.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Ramos, D.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Nacher, V.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Navarro, M.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Carretero, A.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Sánchez-Chardi, A.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Mendes-Jorge, L.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Rodriguez-Baeza, A.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Ruberte, J.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="n">Elsevier</subfield><subfield code="a">Billo, Georis ELSEVIER</subfield><subfield code="t">Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary</subfield><subfield code="d">2022</subfield><subfield code="d">official journal of the ISER</subfield><subfield code="g">Amsterdam [u.a.]</subfield><subfield code="w">(DE-627)ELV008415374</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:135</subfield><subfield code="g">year:2015</subfield><subfield code="g">pages:14-25</subfield><subfield code="g">extent:12</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1016/j.exer.2015.03.019</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ELV</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OPC-MAT</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">31.80</subfield><subfield code="j">Angewandte Mathematik</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">54.80</subfield><subfield code="j">Angewandte Informatik</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">135</subfield><subfield code="j">2015</subfield><subfield code="h">14-25</subfield><subfield code="g">12</subfield></datafield><datafield tag="953" ind1=" " ind2=" "><subfield code="2">045F</subfield><subfield code="a">610</subfield></datafield></record></collection>
|
score |
7.4001493 |