Imaging of cellular aging in human retinal blood vessels
To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessel...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Catita, J. [verfasserIn] |
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E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2015transfer abstract |
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| Umfang: |
12 |
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| Übergeordnetes Werk: |
Enthalten in: Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary - Billo, Georis ELSEVIER, 2022, official journal of the ISER, Amsterdam [u.a.] |
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| Übergeordnetes Werk: |
volume:135 ; year:2015 ; pages:14-25 ; extent:12 |
| Links: |
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| DOI / URN: |
10.1016/j.exer.2015.03.019 |
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| 520 | |a To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. | ||
| 520 | |a To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. | ||
| 650 | 7 | |a Microaneurysms |2 Elsevier | |
| 650 | 7 | |a Cellular senescence |2 Elsevier | |
| 650 | 7 | |a Aging |2 Elsevier | |
| 650 | 7 | |a Lipofuscin accumulation |2 Elsevier | |
| 650 | 7 | |a Acellular capillaries |2 Elsevier | |
| 700 | 1 | |a López-Luppo, M. |4 oth | |
| 700 | 1 | |a Ramos, D. |4 oth | |
| 700 | 1 | |a Nacher, V. |4 oth | |
| 700 | 1 | |a Navarro, M. |4 oth | |
| 700 | 1 | |a Carretero, A. |4 oth | |
| 700 | 1 | |a Sánchez-Chardi, A. |4 oth | |
| 700 | 1 | |a Mendes-Jorge, L. |4 oth | |
| 700 | 1 | |a Rodriguez-Baeza, A. |4 oth | |
| 700 | 1 | |a Ruberte, J. |4 oth | |
| 773 | 0 | 8 | |i Enthalten in |n Elsevier |a Billo, Georis ELSEVIER |t Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |d 2022 |d official journal of the ISER |g Amsterdam [u.a.] |w (DE-627)ELV008415374 |
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10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
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10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
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10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
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10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
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10.1016/j.exer.2015.03.019 doi GBVA2015021000010.pica (DE-627)ELV018910971 (ELSEVIER)S0014-4835(15)00109-8 DE-627 ger DE-627 rakwb eng 610 610 DE-600 510 004 VZ 31.80 bkl 54.80 bkl Catita, J. verfasserin aut Imaging of cellular aging in human retinal blood vessels 2015transfer abstract 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. Microaneurysms Elsevier Cellular senescence Elsevier Aging Elsevier Lipofuscin accumulation Elsevier Acellular capillaries Elsevier López-Luppo, M. oth Ramos, D. oth Nacher, V. oth Navarro, M. oth Carretero, A. oth Sánchez-Chardi, A. oth Mendes-Jorge, L. oth Rodriguez-Baeza, A. oth Ruberte, J. oth Enthalten in Elsevier Billo, Georis ELSEVIER Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary 2022 official journal of the ISER Amsterdam [u.a.] (DE-627)ELV008415374 volume:135 year:2015 pages:14-25 extent:12 https://doi.org/10.1016/j.exer.2015.03.019 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OPC-MAT 31.80 Angewandte Mathematik VZ 54.80 Angewandte Informatik VZ AR 135 2015 14-25 12 045F 610 |
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Enthalten in Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary Amsterdam [u.a.] volume:135 year:2015 pages:14-25 extent:12 |
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Comparison of several interpolation methods to reconstruct field data in the vicinity of a finite element immersed boundary |
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imaging of cellular aging in human retinal blood vessels |
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To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. |
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To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. |
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To date two main aging vascular lesions have been reported in elderly human retinas: acellular capillaries and microaneurysms. However, their exact mechanism of formation remains unclear. Using high resolution microscopy techniques we revise cellular alterations observed in aged human retinal vessels, such as lipofuscin accumulation, caveolae malfunction, blood basement membrane disruption and enhanced apoptosis that could trigger the development of these aging vascular lesions. Moreover, we have generated a set of original images comparing retinal vasculature between middle and old aged healthy humans to show in a comprehensive manner the main structural and ultrastructural alterations occurred during age in retinal blood vessels. |
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