0291 : Mitochondrial permeability transition pore opening might contribute to ventricular arrhythmia maintenance
Calcium leak from the sarcoplasmic reticulum (SR) favors cardiac sudden death through a calcium overload mechanism. Here we investigate the crosstalk between the calcium overload SR-mediated and the mitochondrial permeability transition pore (mPTP) opening during a ventricular arrhythmic episode.
Autor*in: |
Pasdois, Philippe [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2016 |
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Übergeordnetes Werk: |
Enthalten in: A behaviorally-integrated individual-level state-transition model that can predict rapid changes in evacuation demand days earlier - Guan, Xiangyang ELSEVIER, 2021, Amsterdam [u.a.] |
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Übergeordnetes Werk: |
volume:8 ; year:2016 ; number:3 ; pages:208 |
Links: |
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DOI / URN: |
10.1016/S1878-6480(16)30379-2 |
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Calcium leak from the sarcoplasmic reticulum (SR) favors cardiac sudden death through a calcium overload mechanism. Here we investigate the crosstalk between the calcium overload SR-mediated and the mitochondrial permeability transition pore (mPTP) opening during a ventricular arrhythmic episode. |
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Calcium leak from the sarcoplasmic reticulum (SR) favors cardiac sudden death through a calcium overload mechanism. Here we investigate the crosstalk between the calcium overload SR-mediated and the mitochondrial permeability transition pore (mPTP) opening during a ventricular arrhythmic episode. |
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