Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China
This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in sp...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Wang, Tao [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2017transfer abstract |
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| Umfang: |
11 |
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| Übergeordnetes Werk: |
Enthalten in: The development of a computational platform to design and simulate on-board hydrogen storage systems - Mazzucco, Andrea ELSEVIER, 2017transfer abstract, [Amsterdam] |
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| Übergeordnetes Werk: |
volume:55 ; year:2017 ; pages:118-128 ; extent:11 |
| Links: |
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| DOI / URN: |
10.1016/j.jes.2016.06.025 |
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| 520 | |a This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. | ||
| 520 | |a This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. | ||
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| 700 | 1 | |a Wu, Minmin |4 oth | |
| 700 | 1 | |a Zhang, Qianqian |4 oth | |
| 700 | 1 | |a Sun, Shiqi |4 oth | |
| 700 | 1 | |a Yin, Jing |4 oth | |
| 700 | 1 | |a Zhou, Yanchi |4 oth | |
| 700 | 1 | |a Yang, Hao |4 oth | |
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10.1016/j.jes.2016.06.025 doi GBV00000000000076A.pica (DE-627)ELV025449567 (ELSEVIER)S1001-0742(16)30273-X DE-627 ger DE-627 rakwb eng 690 690 DE-600 660 VZ 620 VZ 610 VZ 44.94 bkl Wang, Tao verfasserin aut Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China 2017transfer abstract 11 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. Xia, Zhonghuan oth Wu, Minmin oth Zhang, Qianqian oth Sun, Shiqi oth Yin, Jing oth Zhou, Yanchi oth Yang, Hao oth Enthalten in Elsevier Mazzucco, Andrea ELSEVIER The development of a computational platform to design and simulate on-board hydrogen storage systems 2017transfer abstract [Amsterdam] (DE-627)ELV015065863 volume:55 year:2017 pages:118-128 extent:11 https://doi.org/10.1016/j.jes.2016.06.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_24 GBV_ILN_40 GBV_ILN_70 GBV_ILN_252 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 55 2017 118-128 11 045F 690 |
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10.1016/j.jes.2016.06.025 doi GBV00000000000076A.pica (DE-627)ELV025449567 (ELSEVIER)S1001-0742(16)30273-X DE-627 ger DE-627 rakwb eng 690 690 DE-600 660 VZ 620 VZ 610 VZ 44.94 bkl Wang, Tao verfasserin aut Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China 2017transfer abstract 11 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. Xia, Zhonghuan oth Wu, Minmin oth Zhang, Qianqian oth Sun, Shiqi oth Yin, Jing oth Zhou, Yanchi oth Yang, Hao oth Enthalten in Elsevier Mazzucco, Andrea ELSEVIER The development of a computational platform to design and simulate on-board hydrogen storage systems 2017transfer abstract [Amsterdam] (DE-627)ELV015065863 volume:55 year:2017 pages:118-128 extent:11 https://doi.org/10.1016/j.jes.2016.06.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_24 GBV_ILN_40 GBV_ILN_70 GBV_ILN_252 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 55 2017 118-128 11 045F 690 |
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10.1016/j.jes.2016.06.025 doi GBV00000000000076A.pica (DE-627)ELV025449567 (ELSEVIER)S1001-0742(16)30273-X DE-627 ger DE-627 rakwb eng 690 690 DE-600 660 VZ 620 VZ 610 VZ 44.94 bkl Wang, Tao verfasserin aut Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China 2017transfer abstract 11 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. Xia, Zhonghuan oth Wu, Minmin oth Zhang, Qianqian oth Sun, Shiqi oth Yin, Jing oth Zhou, Yanchi oth Yang, Hao oth Enthalten in Elsevier Mazzucco, Andrea ELSEVIER The development of a computational platform to design and simulate on-board hydrogen storage systems 2017transfer abstract [Amsterdam] (DE-627)ELV015065863 volume:55 year:2017 pages:118-128 extent:11 https://doi.org/10.1016/j.jes.2016.06.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_24 GBV_ILN_40 GBV_ILN_70 GBV_ILN_252 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 55 2017 118-128 11 045F 690 |
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10.1016/j.jes.2016.06.025 doi GBV00000000000076A.pica (DE-627)ELV025449567 (ELSEVIER)S1001-0742(16)30273-X DE-627 ger DE-627 rakwb eng 690 690 DE-600 660 VZ 620 VZ 610 VZ 44.94 bkl Wang, Tao verfasserin aut Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China 2017transfer abstract 11 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. Xia, Zhonghuan oth Wu, Minmin oth Zhang, Qianqian oth Sun, Shiqi oth Yin, Jing oth Zhou, Yanchi oth Yang, Hao oth Enthalten in Elsevier Mazzucco, Andrea ELSEVIER The development of a computational platform to design and simulate on-board hydrogen storage systems 2017transfer abstract [Amsterdam] (DE-627)ELV015065863 volume:55 year:2017 pages:118-128 extent:11 https://doi.org/10.1016/j.jes.2016.06.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_24 GBV_ILN_40 GBV_ILN_70 GBV_ILN_252 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 55 2017 118-128 11 045F 690 |
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10.1016/j.jes.2016.06.025 doi GBV00000000000076A.pica (DE-627)ELV025449567 (ELSEVIER)S1001-0742(16)30273-X DE-627 ger DE-627 rakwb eng 690 690 DE-600 660 VZ 620 VZ 610 VZ 44.94 bkl Wang, Tao verfasserin aut Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China 2017transfer abstract 11 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. Xia, Zhonghuan oth Wu, Minmin oth Zhang, Qianqian oth Sun, Shiqi oth Yin, Jing oth Zhou, Yanchi oth Yang, Hao oth Enthalten in Elsevier Mazzucco, Andrea ELSEVIER The development of a computational platform to design and simulate on-board hydrogen storage systems 2017transfer abstract [Amsterdam] (DE-627)ELV015065863 volume:55 year:2017 pages:118-128 extent:11 https://doi.org/10.1016/j.jes.2016.06.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_24 GBV_ILN_40 GBV_ILN_70 GBV_ILN_252 44.94 Hals-Nasen-Ohrenheilkunde VZ AR 55 2017 118-128 11 045F 690 |
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Wang, Tao @@aut@@ Xia, Zhonghuan @@oth@@ Wu, Minmin @@oth@@ Zhang, Qianqian @@oth@@ Sun, Shiqi @@oth@@ Yin, Jing @@oth@@ Zhou, Yanchi @@oth@@ Yang, Hao @@oth@@ |
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Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China |
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This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. |
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This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. |
| abstract_unstemmed |
This paper focused on the pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons (PAHs) in a new urban district of Nanjing, China. Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR. |
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Pollution characteristics, sources and lung cancer risk of atmospheric polycyclic aromatic hydrocarbons in a new urban district of Nanjing, China |
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Gaseous and aerosol PM2.5 (particulate matter with aerodynamic diameter smaller than 2.5μm) samples were collected in spring of 2015. Sixteen PAHs were extracted and analyzed after sampling. Firstly, arithmetic mean concentrations of PAHs and BaPeq (benzo[a]pyrene equivalent) were calculated. The mean concentrations of PAHs were 29.26±14.13, 18.14±5.37 and 48.47±16.03ng/m3 in gas phase, particle phase and both phases, respectively. The mean concentrations of BaPeq were 0.87±0.51, 2.71±2.17 and 4.06±2.31ng/m3 in gas phase, particle phase and both phases, respectively. Secondly, diagnostic ratios and principal component analysis were adopted to identify the sources of PAHs and the outcomes were the same: traffic exhaust was the predominant source followed by fuel combustion and industrial process. Finally, incremental lung cancer risk (ILCR) induced by whole year inhalation exposure to PAHs for population groups of different age and gender were estimated based on a Monte Carlo simulation. ILCR values caused by particle phase PAHs were greater than those caused by gas phase PAHs. ILCR values for adults were greater than those for other age groups. ILCR values caused by total (gas+particle) PAHs for diverse groups were all greater than the significant level (l0−6), indicating high potential lung cancer risk. Sensitivity analysis results showed that cancer slope factor for BaP inhalation exposure and BaPeq concentration had greater impact than body weight and inhalation rate on the ILCR.</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Xia, Zhonghuan</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Wu, Minmin</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Zhang, Qianqian</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Sun, Shiqi</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Yin, Jing</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Zhou, Yanchi</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Yang, Hao</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="n">Elsevier</subfield><subfield code="a">Mazzucco, Andrea ELSEVIER</subfield><subfield code="t">The development of a computational platform to design and simulate on-board hydrogen storage systems</subfield><subfield code="d">2017transfer abstract</subfield><subfield code="g">[Amsterdam]</subfield><subfield code="w">(DE-627)ELV015065863</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:55</subfield><subfield code="g">year:2017</subfield><subfield code="g">pages:118-128</subfield><subfield code="g">extent:11</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1016/j.jes.2016.06.025</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ELV</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OLC-PHA</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_20</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_22</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_24</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_40</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_70</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_252</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">44.94</subfield><subfield code="j">Hals-Nasen-Ohrenheilkunde</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">55</subfield><subfield code="j">2017</subfield><subfield code="h">118-128</subfield><subfield code="g">11</subfield></datafield><datafield tag="953" ind1=" " ind2=" "><subfield code="2">045F</subfield><subfield code="a">690</subfield></datafield></record></collection>
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