OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation
Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keepin...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Brancaleone, Vincenzo [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2013transfer abstract |
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| Umfang: |
2 |
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| Übergeordnetes Werk: |
Enthalten in: Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways - Gao, Ying ELSEVIER, 2013, biology and chemistry : the official journal of the Nitric Oxide Society, Orlando, Fla |
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| Übergeordnetes Werk: |
volume:31 ; year:2013 ; day:1 ; month:09 ; pages:21-22 ; extent:2 |
| Links: |
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| DOI / URN: |
10.1016/j.niox.2013.06.036 |
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ELV027456641 |
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| 520 | |a Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. | ||
| 520 | |a Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. | ||
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10.1016/j.niox.2013.06.036 doi GBVA2013018000010.pica (DE-627)ELV027456641 (ELSEVIER)S1089-8603(13)00190-0 DE-627 ger DE-627 rakwb eng 570 540 570 DE-600 540 DE-600 570 VZ 670 VZ 51.60 bkl 58.45 bkl Brancaleone, Vincenzo verfasserin aut OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation 2013transfer abstract 2 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Flower, Roderick J. oth Cirino, Giuseppe oth Perretti, Mauro oth Enthalten in Acad. Press Gao, Ying ELSEVIER Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways 2013 biology and chemistry : the official journal of the Nitric Oxide Society Orlando, Fla (DE-627)ELV017091187 volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 https://doi.org/10.1016/j.niox.2013.06.036 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 51.60 Keramische Werkstoffe Hartstoffe Werkstoffkunde VZ 58.45 Gesteinshüttenkunde VZ AR 31 2013 1 0901 21-22 2 31.2013, S21-, (2 S.) 045F 570 |
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10.1016/j.niox.2013.06.036 doi GBVA2013018000010.pica (DE-627)ELV027456641 (ELSEVIER)S1089-8603(13)00190-0 DE-627 ger DE-627 rakwb eng 570 540 570 DE-600 540 DE-600 570 VZ 670 VZ 51.60 bkl 58.45 bkl Brancaleone, Vincenzo verfasserin aut OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation 2013transfer abstract 2 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Flower, Roderick J. oth Cirino, Giuseppe oth Perretti, Mauro oth Enthalten in Acad. Press Gao, Ying ELSEVIER Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways 2013 biology and chemistry : the official journal of the Nitric Oxide Society Orlando, Fla (DE-627)ELV017091187 volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 https://doi.org/10.1016/j.niox.2013.06.036 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 51.60 Keramische Werkstoffe Hartstoffe Werkstoffkunde VZ 58.45 Gesteinshüttenkunde VZ AR 31 2013 1 0901 21-22 2 31.2013, S21-, (2 S.) 045F 570 |
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10.1016/j.niox.2013.06.036 doi GBVA2013018000010.pica (DE-627)ELV027456641 (ELSEVIER)S1089-8603(13)00190-0 DE-627 ger DE-627 rakwb eng 570 540 570 DE-600 540 DE-600 570 VZ 670 VZ 51.60 bkl 58.45 bkl Brancaleone, Vincenzo verfasserin aut OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation 2013transfer abstract 2 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Flower, Roderick J. oth Cirino, Giuseppe oth Perretti, Mauro oth Enthalten in Acad. Press Gao, Ying ELSEVIER Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways 2013 biology and chemistry : the official journal of the Nitric Oxide Society Orlando, Fla (DE-627)ELV017091187 volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 https://doi.org/10.1016/j.niox.2013.06.036 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 51.60 Keramische Werkstoffe Hartstoffe Werkstoffkunde VZ 58.45 Gesteinshüttenkunde VZ AR 31 2013 1 0901 21-22 2 31.2013, S21-, (2 S.) 045F 570 |
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10.1016/j.niox.2013.06.036 doi GBVA2013018000010.pica (DE-627)ELV027456641 (ELSEVIER)S1089-8603(13)00190-0 DE-627 ger DE-627 rakwb eng 570 540 570 DE-600 540 DE-600 570 VZ 670 VZ 51.60 bkl 58.45 bkl Brancaleone, Vincenzo verfasserin aut OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation 2013transfer abstract 2 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Flower, Roderick J. oth Cirino, Giuseppe oth Perretti, Mauro oth Enthalten in Acad. Press Gao, Ying ELSEVIER Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways 2013 biology and chemistry : the official journal of the Nitric Oxide Society Orlando, Fla (DE-627)ELV017091187 volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 https://doi.org/10.1016/j.niox.2013.06.036 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 51.60 Keramische Werkstoffe Hartstoffe Werkstoffkunde VZ 58.45 Gesteinshüttenkunde VZ AR 31 2013 1 0901 21-22 2 31.2013, S21-, (2 S.) 045F 570 |
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10.1016/j.niox.2013.06.036 doi GBVA2013018000010.pica (DE-627)ELV027456641 (ELSEVIER)S1089-8603(13)00190-0 DE-627 ger DE-627 rakwb eng 570 540 570 DE-600 540 DE-600 570 VZ 670 VZ 51.60 bkl 58.45 bkl Brancaleone, Vincenzo verfasserin aut OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation 2013transfer abstract 2 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. Flower, Roderick J. oth Cirino, Giuseppe oth Perretti, Mauro oth Enthalten in Acad. Press Gao, Ying ELSEVIER Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways 2013 biology and chemistry : the official journal of the Nitric Oxide Society Orlando, Fla (DE-627)ELV017091187 volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 https://doi.org/10.1016/j.niox.2013.06.036 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U 51.60 Keramische Werkstoffe Hartstoffe Werkstoffkunde VZ 58.45 Gesteinshüttenkunde VZ AR 31 2013 1 0901 21-22 2 31.2013, S21-, (2 S.) 045F 570 |
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English |
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Enthalten in Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways Orlando, Fla volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 |
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Enthalten in Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways Orlando, Fla volume:31 year:2013 day:1 month:09 pages:21-22 extent:2 |
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Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways |
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Brancaleone, Vincenzo @@aut@@ Flower, Roderick J. @@oth@@ Cirino, Giuseppe @@oth@@ Perretti, Mauro @@oth@@ |
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Brancaleone, Vincenzo |
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Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways |
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Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways |
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OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation |
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Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways |
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Enhancement of (−)-epigallocatechin-3-gallate and theaflavin-3-3′-digallate induced apoptosis by ascorbic acid in human lung adenocarcinoma SPC-A-1 cells and esophageal carcinoma Eca-109 cells via MAPK pathways |
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op06 annexin a1 (anxa1) mediates hydrogen sulfide (h2s) effects in the control of inflammation |
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OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation |
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Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. |
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Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. |
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Hydrogen sulfide (H2S), a gaseous mediator synthesized in several mammalian tissues by two main enzymes CBS and CSE, increases under inflammatory conditions or sepsis [1,2]. Inflammation per se does not represent an unwanted event, since it also provides a physiological response to an injury, keeping inflammation under control [3]. Annexin A1 (AnxA1) represent the front line of innate immunity as it is externalized on leukocytes membrane, preventing their adhesion to vascular endothelium and their transmigration into inflamed tissues [4]. Since H2S affords potent inhibitory properties on the process of leukocyte trafficking [5], we tested whether endogenous AnxA1 could display intermediary functions. |
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OP06 Annexin A1 (AnxA1) mediates hydrogen sulfide (H2S) effects in the control of inflammation |
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Flower, Roderick J. Cirino, Giuseppe Perretti, Mauro |
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