Methylprednisolone prevents nerve injury-induced hyperalgesia in neprilysin knockout mice
Enhanced pain behavior and edema after nerve injury in mice deficient of neutral endopeptidase respond to methylprednisolone, possibly through an endothelin-related pathway.
Gespeichert in:
| Autor*in: |
He, Lan [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2014 |
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| Schlagwörter: |
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| Umfang: |
7 |
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| Übergeordnetes Werk: |
Enthalten in: 336 MECHANISMS OF EGFR INHIBITORS - 2012, the journal of the International Association for the Study of Pain, New York, NY [u.a.] |
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| Übergeordnetes Werk: |
volume:155 ; year:2014 ; number:3 ; pages:574-580 ; extent:7 |
| Links: |
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| DOI / URN: |
10.1016/j.pain.2013.12.003 |
|---|
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Enhanced pain behavior and edema after nerve injury in mice deficient of neutral endopeptidase respond to methylprednisolone, possibly through an endothelin-related pathway. |
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Enhanced pain behavior and edema after nerve injury in mice deficient of neutral endopeptidase respond to methylprednisolone, possibly through an endothelin-related pathway. |
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