Enterovirus strain and type-specific differences in growth kinetics and virus-induced cell destruction in human pancreatic duct epithelial HPDE cells
• The pancreatic ductal cells were highly susceptible to enterovirus infections. • Strain-specific differences in viral growth and the ability to cause cpe exist. • Adaptation of non-lytic CV-B6 strain resulted in cpe and increased virus growth. • Adaptation was due to a single amino acid substituti...
Ausführliche Beschreibung
Autor*in: |
Smura, Teemu [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2015 |
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Schlagwörter: |
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Umfang: |
10 |
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Übergeordnetes Werk: |
Enthalten in: AKT-mediated enhanced aerobic glycolysis causes acquired radioresistance by human tumor cells - Shimura, Tsutomu ELSEVIER, 2014, an international journal of molecular and cellular virology, Amsterdam [u.a.] |
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Übergeordnetes Werk: |
volume:210 ; year:2015 ; day:2 ; month:12 ; pages:188-197 ; extent:10 |
Links: |
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DOI / URN: |
10.1016/j.virusres.2015.08.003 |
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ELV028774728 |
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• The pancreatic ductal cells were highly susceptible to enterovirus infections. • Strain-specific differences in viral growth and the ability to cause cpe exist. • Adaptation of non-lytic CV-B6 strain resulted in cpe and increased virus growth. • Adaptation was due to a single amino acid substitution in the VP1 protein. • Adaptation was associated with the ability to use DAF as a receptor. |
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• The pancreatic ductal cells were highly susceptible to enterovirus infections. • Strain-specific differences in viral growth and the ability to cause cpe exist. • Adaptation of non-lytic CV-B6 strain resulted in cpe and increased virus growth. • Adaptation was due to a single amino acid substitution in the VP1 protein. • Adaptation was associated with the ability to use DAF as a receptor. |
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• The pancreatic ductal cells were highly susceptible to enterovirus infections. • Strain-specific differences in viral growth and the ability to cause cpe exist. • Adaptation of non-lytic CV-B6 strain resulted in cpe and increased virus growth. • Adaptation was due to a single amino acid substitution in the VP1 protein. • Adaptation was associated with the ability to use DAF as a receptor. |
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