Cyclic mechanical stretch-induced oxidative stress occurs via a NOX-dependent mechanism in type II alveolar epithelial cells
• Cyclic mechanical stretching (CMS) contributes to alveolar epithelial injury. • Oxidative stress was increased in alveolar epithelial cells undergoing CMS. • The primary source of CMS-induced oxidative stress was a NOX-dependent mechanism. • NOX may be a therapeutic target for CMS-induced alveolar...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Tanaka, Toru [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2017 |
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| Schlagwörter: |
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| Umfang: |
9 |
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| Übergeordnetes Werk: |
Enthalten in: Usage of formal financial services in India: Demand barriers or supply constraints? - Kumar, Abhishek ELSEVIER, 2018, Amsterdam [u.a.] |
|---|---|
| Übergeordnetes Werk: |
volume:242 ; year:2017 ; pages:108-116 ; extent:9 |
| Links: |
|---|
| DOI / URN: |
10.1016/j.resp.2017.04.007 |
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ELV030369509 |
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| 520 | |a • Cyclic mechanical stretching (CMS) contributes to alveolar epithelial injury. • Oxidative stress was increased in alveolar epithelial cells undergoing CMS. • The primary source of CMS-induced oxidative stress was a NOX-dependent mechanism. • NOX may be a therapeutic target for CMS-induced alveolar epithelial injury. | ||
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• Cyclic mechanical stretching (CMS) contributes to alveolar epithelial injury. • Oxidative stress was increased in alveolar epithelial cells undergoing CMS. • The primary source of CMS-induced oxidative stress was a NOX-dependent mechanism. • NOX may be a therapeutic target for CMS-induced alveolar epithelial injury. |
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• Cyclic mechanical stretching (CMS) contributes to alveolar epithelial injury. • Oxidative stress was increased in alveolar epithelial cells undergoing CMS. • The primary source of CMS-induced oxidative stress was a NOX-dependent mechanism. • NOX may be a therapeutic target for CMS-induced alveolar epithelial injury. |
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• Cyclic mechanical stretching (CMS) contributes to alveolar epithelial injury. • Oxidative stress was increased in alveolar epithelial cells undergoing CMS. • The primary source of CMS-induced oxidative stress was a NOX-dependent mechanism. • NOX may be a therapeutic target for CMS-induced alveolar epithelial injury. |
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