Differential speed of activation in antioxidant system in three oat genotypes
The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition,...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Pereira, Luciane Belmonte [verfasserIn] |
|---|
| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2013transfer abstract |
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| Umfang: |
6 |
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| Übergeordnetes Werk: |
Enthalten in: Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study - 2015, an interdisciplinary journal, New York, NY [u.a.] |
|---|---|
| Übergeordnetes Werk: |
volume:128 ; year:2013 ; pages:202-207 ; extent:6 |
| Links: |
|---|
| DOI / URN: |
10.1016/j.jinorgbio.2013.07.025 |
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| 245 | 1 | 0 | |a Differential speed of activation in antioxidant system in three oat genotypes |
| 264 | 1 | |c 2013transfer abstract | |
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| 520 | |a The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. | ||
| 520 | |a The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. | ||
| 700 | 1 | |a Cargnelutti, Denise |4 oth | |
| 700 | 1 | |a Rossato, Liana Verônica |4 oth | |
| 700 | 1 | |a Gonçalves, Jamile Fabbrin |4 oth | |
| 700 | 1 | |a Tabaldi, Luciane Almeri |4 oth | |
| 700 | 1 | |a Schmatz, Roberta |4 oth | |
| 700 | 1 | |a Vieira, Juliano Marchi |4 oth | |
| 700 | 1 | |a Dressler, Valderi |4 oth | |
| 700 | 1 | |a Nicoloso, Fernando Teixeira |4 oth | |
| 700 | 1 | |a Federizzi, Luiz Carlos |4 oth | |
| 700 | 1 | |a Morsch, Vera M. |4 oth | |
| 700 | 1 | |a Schetinger, Maria R.C. |4 oth | |
| 773 | 0 | 8 | |i Enthalten in |n Elsevier |t Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study |d 2015 |d an interdisciplinary journal |g New York, NY [u.a.] |w (DE-627)ELV018319009 |
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10.1016/j.jinorgbio.2013.07.025 doi GBVA2013003000029.pica (DE-627)ELV032811845 (ELSEVIER)S0162-0134(13)00189-X DE-627 ger DE-627 rakwb eng 540 540 DE-600 610 VZ 333.7 610 VZ 43.12 bkl 43.13 bkl 44.13 bkl Pereira, Luciane Belmonte verfasserin aut Differential speed of activation in antioxidant system in three oat genotypes 2013transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. Cargnelutti, Denise oth Rossato, Liana Verônica oth Gonçalves, Jamile Fabbrin oth Tabaldi, Luciane Almeri oth Schmatz, Roberta oth Vieira, Juliano Marchi oth Dressler, Valderi oth Nicoloso, Fernando Teixeira oth Federizzi, Luiz Carlos oth Morsch, Vera M. oth Schetinger, Maria R.C. oth Enthalten in Elsevier Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study 2015 an interdisciplinary journal New York, NY [u.a.] (DE-627)ELV018319009 volume:128 year:2013 pages:202-207 extent:6 https://doi.org/10.1016/j.jinorgbio.2013.07.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA SSG-OPC-GGO GBV_ILN_40 43.12 Umweltchemie VZ 43.13 Umwelttoxikologie VZ 44.13 Medizinische Ökologie VZ AR 128 2013 202-207 6 045F 540 |
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10.1016/j.jinorgbio.2013.07.025 doi GBVA2013003000029.pica (DE-627)ELV032811845 (ELSEVIER)S0162-0134(13)00189-X DE-627 ger DE-627 rakwb eng 540 540 DE-600 610 VZ 333.7 610 VZ 43.12 bkl 43.13 bkl 44.13 bkl Pereira, Luciane Belmonte verfasserin aut Differential speed of activation in antioxidant system in three oat genotypes 2013transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. Cargnelutti, Denise oth Rossato, Liana Verônica oth Gonçalves, Jamile Fabbrin oth Tabaldi, Luciane Almeri oth Schmatz, Roberta oth Vieira, Juliano Marchi oth Dressler, Valderi oth Nicoloso, Fernando Teixeira oth Federizzi, Luiz Carlos oth Morsch, Vera M. oth Schetinger, Maria R.C. oth Enthalten in Elsevier Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study 2015 an interdisciplinary journal New York, NY [u.a.] (DE-627)ELV018319009 volume:128 year:2013 pages:202-207 extent:6 https://doi.org/10.1016/j.jinorgbio.2013.07.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA SSG-OPC-GGO GBV_ILN_40 43.12 Umweltchemie VZ 43.13 Umwelttoxikologie VZ 44.13 Medizinische Ökologie VZ AR 128 2013 202-207 6 045F 540 |
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10.1016/j.jinorgbio.2013.07.025 doi GBVA2013003000029.pica (DE-627)ELV032811845 (ELSEVIER)S0162-0134(13)00189-X DE-627 ger DE-627 rakwb eng 540 540 DE-600 610 VZ 333.7 610 VZ 43.12 bkl 43.13 bkl 44.13 bkl Pereira, Luciane Belmonte verfasserin aut Differential speed of activation in antioxidant system in three oat genotypes 2013transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. Cargnelutti, Denise oth Rossato, Liana Verônica oth Gonçalves, Jamile Fabbrin oth Tabaldi, Luciane Almeri oth Schmatz, Roberta oth Vieira, Juliano Marchi oth Dressler, Valderi oth Nicoloso, Fernando Teixeira oth Federizzi, Luiz Carlos oth Morsch, Vera M. oth Schetinger, Maria R.C. oth Enthalten in Elsevier Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study 2015 an interdisciplinary journal New York, NY [u.a.] (DE-627)ELV018319009 volume:128 year:2013 pages:202-207 extent:6 https://doi.org/10.1016/j.jinorgbio.2013.07.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA SSG-OPC-GGO GBV_ILN_40 43.12 Umweltchemie VZ 43.13 Umwelttoxikologie VZ 44.13 Medizinische Ökologie VZ AR 128 2013 202-207 6 045F 540 |
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10.1016/j.jinorgbio.2013.07.025 doi GBVA2013003000029.pica (DE-627)ELV032811845 (ELSEVIER)S0162-0134(13)00189-X DE-627 ger DE-627 rakwb eng 540 540 DE-600 610 VZ 333.7 610 VZ 43.12 bkl 43.13 bkl 44.13 bkl Pereira, Luciane Belmonte verfasserin aut Differential speed of activation in antioxidant system in three oat genotypes 2013transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. Cargnelutti, Denise oth Rossato, Liana Verônica oth Gonçalves, Jamile Fabbrin oth Tabaldi, Luciane Almeri oth Schmatz, Roberta oth Vieira, Juliano Marchi oth Dressler, Valderi oth Nicoloso, Fernando Teixeira oth Federizzi, Luiz Carlos oth Morsch, Vera M. oth Schetinger, Maria R.C. oth Enthalten in Elsevier Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study 2015 an interdisciplinary journal New York, NY [u.a.] (DE-627)ELV018319009 volume:128 year:2013 pages:202-207 extent:6 https://doi.org/10.1016/j.jinorgbio.2013.07.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA SSG-OPC-GGO GBV_ILN_40 43.12 Umweltchemie VZ 43.13 Umwelttoxikologie VZ 44.13 Medizinische Ökologie VZ AR 128 2013 202-207 6 045F 540 |
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10.1016/j.jinorgbio.2013.07.025 doi GBVA2013003000029.pica (DE-627)ELV032811845 (ELSEVIER)S0162-0134(13)00189-X DE-627 ger DE-627 rakwb eng 540 540 DE-600 610 VZ 333.7 610 VZ 43.12 bkl 43.13 bkl 44.13 bkl Pereira, Luciane Belmonte verfasserin aut Differential speed of activation in antioxidant system in three oat genotypes 2013transfer abstract 6 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. Cargnelutti, Denise oth Rossato, Liana Verônica oth Gonçalves, Jamile Fabbrin oth Tabaldi, Luciane Almeri oth Schmatz, Roberta oth Vieira, Juliano Marchi oth Dressler, Valderi oth Nicoloso, Fernando Teixeira oth Federizzi, Luiz Carlos oth Morsch, Vera M. oth Schetinger, Maria R.C. oth Enthalten in Elsevier Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study 2015 an interdisciplinary journal New York, NY [u.a.] (DE-627)ELV018319009 volume:128 year:2013 pages:202-207 extent:6 https://doi.org/10.1016/j.jinorgbio.2013.07.025 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA SSG-OPC-GGO GBV_ILN_40 43.12 Umweltchemie VZ 43.13 Umwelttoxikologie VZ 44.13 Medizinische Ökologie VZ AR 128 2013 202-207 6 045F 540 |
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Pereira, Luciane Belmonte @@aut@@ Cargnelutti, Denise @@oth@@ Rossato, Liana Verônica @@oth@@ Gonçalves, Jamile Fabbrin @@oth@@ Tabaldi, Luciane Almeri @@oth@@ Schmatz, Roberta @@oth@@ Vieira, Juliano Marchi @@oth@@ Dressler, Valderi @@oth@@ Nicoloso, Fernando Teixeira @@oth@@ Federizzi, Luiz Carlos @@oth@@ Morsch, Vera M. @@oth@@ Schetinger, Maria R.C. @@oth@@ |
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differential speed of activation in antioxidant system in three oat genotypes |
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Differential speed of activation in antioxidant system in three oat genotypes |
| abstract |
The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. |
| abstractGer |
The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. |
| abstract_unstemmed |
The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al. |
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Cargnelutti, Denise Rossato, Liana Verônica Gonçalves, Jamile Fabbrin Tabaldi, Luciane Almeri Schmatz, Roberta Vieira, Juliano Marchi Dressler, Valderi Nicoloso, Fernando Teixeira Federizzi, Luiz Carlos Morsch, Vera M. Schetinger, Maria R.C. |
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Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al.</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">The objective of this study was to evaluate whether the oxidative stress caused by aluminum (Al) toxicity is a symptom that can trigger root growth inhibition in oat genotype seedlings. Oat seedlings were grown in a nutrient solution (pH4.0) with 0 and 370μM Al. At 12, 24, and 36h after Al addition, growth (root length) and biochemical parameters (catalase — CAT, ascorbate peroxidase — APX, and superoxide dismutase — SOD activities, lipid peroxidation, ascorbic acid (ASA) and non-protein thiol group (NPSH) concentration) were determined. The aluminum content was measured in oat seedlings. Regardless of the exposure time, root of the tolerant genotype grew normally with any Al treatments. Al supply caused lipid peroxidation only in the Al-sensitive genotype in roots and shoots (at 12, 24, and 36h). In sensitive genotype seedlings, CAT, APX, and SOD were activated only at 24 or 36h. In tolerant and intermediate genotypes, CAT, APX, and SOD were activated at 12, 24, and 36h. Data for root growth and lipid peroxidation suggested that lipid peroxidation in the sensitive genotype may be an effect of Al toxicity on root growth. Therefore, the tolerant, intermediate, and sensitive genotypes differ in the expression of the amount, type of antioxidants, and speed of activation of antioxidant system, suggesting a varying capacity of these genotypes to deal with oxidative stress, which resulted in varying sensitivity and tolerance to Al.</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Cargnelutti, Denise</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Rossato, Liana Verônica</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Gonçalves, Jamile Fabbrin</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Tabaldi, Luciane Almeri</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Schmatz, Roberta</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Vieira, Juliano Marchi</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Dressler, Valderi</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Nicoloso, Fernando Teixeira</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Federizzi, Luiz Carlos</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Morsch, Vera M.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Schetinger, Maria R.C.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="n">Elsevier</subfield><subfield code="t">Severe Staphylococcus aureus infection and melanoma risk: A large single center observational cohort study</subfield><subfield code="d">2015</subfield><subfield code="d">an interdisciplinary journal</subfield><subfield code="g">New York, NY [u.a.]</subfield><subfield code="w">(DE-627)ELV018319009</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:128</subfield><subfield code="g">year:2013</subfield><subfield code="g">pages:202-207</subfield><subfield code="g">extent:6</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1016/j.jinorgbio.2013.07.025</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ELV</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OLC-PHA</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OPC-GGO</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_40</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">43.12</subfield><subfield code="j">Umweltchemie</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">43.13</subfield><subfield code="j">Umwelttoxikologie</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">44.13</subfield><subfield code="j">Medizinische Ökologie</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">128</subfield><subfield code="j">2013</subfield><subfield code="h">202-207</subfield><subfield code="g">6</subfield></datafield><datafield tag="953" ind1=" " ind2=" "><subfield code="2">045F</subfield><subfield code="a">540</subfield></datafield></record></collection>
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