Inflammatory pathways in spondyloarthritis
• Genes in TNF, IL-1, IL-6 and IL-23/IL-17 pathways are genetically associated with SpA. • Functional and clinical studies identify TNF and IL-23/IL-17 as key pathways in SpA. • Recent data point out transmembrane TNF and IL-17 as important players in SpA.
Autor*in: |
Hreggvidsdottir, Hulda S. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2014 |
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Schlagwörter: |
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Umfang: |
10 |
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Übergeordnetes Werk: |
Enthalten in: Letter regarding the article: Changes in BNP and cardiac troponin I after high-intensity interval and endurance exercise in heart failure patients and healthy controls - Gayda, Mathieu ELSEVIER, 2015, Amsterdam [u.a.] |
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Übergeordnetes Werk: |
volume:57 ; year:2014 ; number:1 ; pages:28-37 ; extent:10 |
Links: |
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DOI / URN: |
10.1016/j.molimm.2013.07.016 |
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• Genes in TNF, IL-1, IL-6 and IL-23/IL-17 pathways are genetically associated with SpA. • Functional and clinical studies identify TNF and IL-23/IL-17 as key pathways in SpA. • Recent data point out transmembrane TNF and IL-17 as important players in SpA. |
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• Genes in TNF, IL-1, IL-6 and IL-23/IL-17 pathways are genetically associated with SpA. • Functional and clinical studies identify TNF and IL-23/IL-17 as key pathways in SpA. • Recent data point out transmembrane TNF and IL-17 as important players in SpA. |
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• Genes in TNF, IL-1, IL-6 and IL-23/IL-17 pathways are genetically associated with SpA. • Functional and clinical studies identify TNF and IL-23/IL-17 as key pathways in SpA. • Recent data point out transmembrane TNF and IL-17 as important players in SpA. |
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