Increased susceptibility of prenatal food restricted offspring to high-fat diet-induced nonalcoholic fatty liver disease is intrauterine programmed
• The potential mechanism under prenatal food restriction and adult NAFLD was explored. • The adverse intrauterine programming of hepatic lipid metabolism could increase the susceptibility to NAFLD. • The adverse intrauterine programming of glucocorticoid-IGF1 axis could accelerate the occurrence of...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Zhang, Li [verfasserIn] |
|---|
| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2016 |
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| Schlagwörter: |
Glucocorticoid-insulin-like growth factor 1 axis |
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| Umfang: |
12 |
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| Übergeordnetes Werk: |
Enthalten in: Porous silicon Bragg mirrors on single- and multi-crystalline silicon for solar cells - 2013transfer abstract, Amsterdam [u.a.] |
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| Übergeordnetes Werk: |
volume:65 ; year:2016 ; pages:236-247 ; extent:12 |
| Links: |
|---|
| DOI / URN: |
10.1016/j.reprotox.2016.08.006 |
|---|
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ELV035388501 |
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| 520 | |a • The potential mechanism under prenatal food restriction and adult NAFLD was explored. • The adverse intrauterine programming of hepatic lipid metabolism could increase the susceptibility to NAFLD. • The adverse intrauterine programming of glucocorticoid-IGF1 axis could accelerate the occurrence of NAFLD. | ||
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increased susceptibility of prenatal food restricted offspring to high-fat diet-induced nonalcoholic fatty liver disease is intrauterine programmed |
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Increased susceptibility of prenatal food restricted offspring to high-fat diet-induced nonalcoholic fatty liver disease is intrauterine programmed |
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• The potential mechanism under prenatal food restriction and adult NAFLD was explored. • The adverse intrauterine programming of hepatic lipid metabolism could increase the susceptibility to NAFLD. • The adverse intrauterine programming of glucocorticoid-IGF1 axis could accelerate the occurrence of NAFLD. |
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• The potential mechanism under prenatal food restriction and adult NAFLD was explored. • The adverse intrauterine programming of hepatic lipid metabolism could increase the susceptibility to NAFLD. • The adverse intrauterine programming of glucocorticoid-IGF1 axis could accelerate the occurrence of NAFLD. |
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• The potential mechanism under prenatal food restriction and adult NAFLD was explored. • The adverse intrauterine programming of hepatic lipid metabolism could increase the susceptibility to NAFLD. • The adverse intrauterine programming of glucocorticoid-IGF1 axis could accelerate the occurrence of NAFLD. |
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| title_short |
Increased susceptibility of prenatal food restricted offspring to high-fat diet-induced nonalcoholic fatty liver disease is intrauterine programmed |
| url |
https://doi.org/10.1016/j.reprotox.2016.08.006 |
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Shen, Lang Xu, Dan Wang, Linlong Guo, Yuming Liu, Zhongfen Liu, Yansong Liu, Lian Magdalou, Jacques Chen, Liaobin Wang, Hui |
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Shen, Lang Xu, Dan Wang, Linlong Guo, Yuming Liu, Zhongfen Liu, Yansong Liu, Lian Magdalou, Jacques Chen, Liaobin Wang, Hui |
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10.1016/j.reprotox.2016.08.006 |
| up_date |
2024-07-06T17:25:52.512Z |
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