Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury
Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and ca...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Tóth, Andrea E. [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2014 |
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| Schlagwörter: |
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| Umfang: |
12 |
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| Übergeordnetes Werk: |
Enthalten in: Knowledge transfer between brain lesion segmentation tasks with increased model capacity - Liu, Yanlin ELSEVIER, 2020, official publication of the Instituto Mexicano del Seguro Social (IMSS), Amsterdam [u.a.] |
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| Übergeordnetes Werk: |
volume:45 ; year:2014 ; number:8 ; pages:753-764 ; extent:12 |
| Links: |
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| DOI / URN: |
10.1016/j.arcmed.2014.10.009 |
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| 520 | |a Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. | ||
| 650 | 7 | |a Methylglyoxal |2 Elsevier | |
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10.1016/j.arcmed.2014.10.009 doi GBVA2014006000018.pica (DE-627)ELV039248631 (ELSEVIER)S0188-4409(14)00224-0 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.64 bkl 44.32 bkl Tóth, Andrea E. verfasserin aut Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury 2014 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. Methylglyoxal Elsevier Aminoguanidine Elsevier Human brain endothelial cell Elsevier U0126 Elsevier All-trans-retinoic acid Elsevier Protein modification Elsevier Tóth, András oth Walter, Fruzsina R. oth Kiss, Lóránd oth Veszelka, Szilvia oth Ózsvári, Béla oth Puskás, László G. oth Heimesaat, Markus M. oth Dohgu, Shinya oth Kataoka, Yasufumi oth Rákhely, Gábor oth Deli, Mária A. oth Enthalten in Elsevier Science Liu, Yanlin ELSEVIER Knowledge transfer between brain lesion segmentation tasks with increased model capacity 2020 official publication of the Instituto Mexicano del Seguro Social (IMSS) Amsterdam [u.a.] (DE-627)ELV005571200 volume:45 year:2014 number:8 pages:753-764 extent:12 https://doi.org/10.1016/j.arcmed.2014.10.009 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.64 Radiologie VZ 44.32 Medizinische Mathematik medizinische Statistik VZ AR 45 2014 8 753-764 12 045F 610 |
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10.1016/j.arcmed.2014.10.009 doi GBVA2014006000018.pica (DE-627)ELV039248631 (ELSEVIER)S0188-4409(14)00224-0 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.64 bkl 44.32 bkl Tóth, Andrea E. verfasserin aut Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury 2014 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. Methylglyoxal Elsevier Aminoguanidine Elsevier Human brain endothelial cell Elsevier U0126 Elsevier All-trans-retinoic acid Elsevier Protein modification Elsevier Tóth, András oth Walter, Fruzsina R. oth Kiss, Lóránd oth Veszelka, Szilvia oth Ózsvári, Béla oth Puskás, László G. oth Heimesaat, Markus M. oth Dohgu, Shinya oth Kataoka, Yasufumi oth Rákhely, Gábor oth Deli, Mária A. oth Enthalten in Elsevier Science Liu, Yanlin ELSEVIER Knowledge transfer between brain lesion segmentation tasks with increased model capacity 2020 official publication of the Instituto Mexicano del Seguro Social (IMSS) Amsterdam [u.a.] (DE-627)ELV005571200 volume:45 year:2014 number:8 pages:753-764 extent:12 https://doi.org/10.1016/j.arcmed.2014.10.009 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.64 Radiologie VZ 44.32 Medizinische Mathematik medizinische Statistik VZ AR 45 2014 8 753-764 12 045F 610 |
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10.1016/j.arcmed.2014.10.009 doi GBVA2014006000018.pica (DE-627)ELV039248631 (ELSEVIER)S0188-4409(14)00224-0 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.64 bkl 44.32 bkl Tóth, Andrea E. verfasserin aut Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury 2014 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. Methylglyoxal Elsevier Aminoguanidine Elsevier Human brain endothelial cell Elsevier U0126 Elsevier All-trans-retinoic acid Elsevier Protein modification Elsevier Tóth, András oth Walter, Fruzsina R. oth Kiss, Lóránd oth Veszelka, Szilvia oth Ózsvári, Béla oth Puskás, László G. oth Heimesaat, Markus M. oth Dohgu, Shinya oth Kataoka, Yasufumi oth Rákhely, Gábor oth Deli, Mária A. oth Enthalten in Elsevier Science Liu, Yanlin ELSEVIER Knowledge transfer between brain lesion segmentation tasks with increased model capacity 2020 official publication of the Instituto Mexicano del Seguro Social (IMSS) Amsterdam [u.a.] (DE-627)ELV005571200 volume:45 year:2014 number:8 pages:753-764 extent:12 https://doi.org/10.1016/j.arcmed.2014.10.009 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.64 Radiologie VZ 44.32 Medizinische Mathematik medizinische Statistik VZ AR 45 2014 8 753-764 12 045F 610 |
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10.1016/j.arcmed.2014.10.009 doi GBVA2014006000018.pica (DE-627)ELV039248631 (ELSEVIER)S0188-4409(14)00224-0 DE-627 ger DE-627 rakwb eng 610 610 DE-600 610 VZ 44.64 bkl 44.32 bkl Tóth, Andrea E. verfasserin aut Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury 2014 12 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. Methylglyoxal Elsevier Aminoguanidine Elsevier Human brain endothelial cell Elsevier U0126 Elsevier All-trans-retinoic acid Elsevier Protein modification Elsevier Tóth, András oth Walter, Fruzsina R. oth Kiss, Lóránd oth Veszelka, Szilvia oth Ózsvári, Béla oth Puskás, László G. oth Heimesaat, Markus M. oth Dohgu, Shinya oth Kataoka, Yasufumi oth Rákhely, Gábor oth Deli, Mária A. oth Enthalten in Elsevier Science Liu, Yanlin ELSEVIER Knowledge transfer between brain lesion segmentation tasks with increased model capacity 2020 official publication of the Instituto Mexicano del Seguro Social (IMSS) Amsterdam [u.a.] (DE-627)ELV005571200 volume:45 year:2014 number:8 pages:753-764 extent:12 https://doi.org/10.1016/j.arcmed.2014.10.009 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA 44.64 Radiologie VZ 44.32 Medizinische Mathematik medizinische Statistik VZ AR 45 2014 8 753-764 12 045F 610 |
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Knowledge transfer between brain lesion segmentation tasks with increased model capacity |
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Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury |
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Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury |
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compounds blocking methylglyoxal-induced protein modification and brain endothelial injury |
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Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury |
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Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. |
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Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. |
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Elevated levels of reactive carbonyl species such as methylglyoxal triggers carbonyl stress and activates a series of inflammatory responses leading to accelerated vascular damage. Carbonyl stress is implicated in conditions and diseases like aging, diabetes mellitus, Alzheimer's disease and cardiovascular diseases. Our aim was to examine the effects of methylglyoxal on human hCMEC/D3 brain endothelial cells and search for protective molecules to prevent endothelial damage. |
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Compounds Blocking Methylglyoxal-induced Protein Modification and Brain Endothelial Injury |
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Tóth, András Walter, Fruzsina R. Kiss, Lóránd Veszelka, Szilvia Ózsvári, Béla Puskás, László G. Heimesaat, Markus M. Dohgu, Shinya Kataoka, Yasufumi Rákhely, Gábor Deli, Mária A. |
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