Neonatal C57BL/6J and parkin mice respond differently following developmental manganese exposure: Result of a high dose pilot study
• Parkin and C57BL/6J mice have similar brain manganese (Mn) levels following neonatal Mn exposure. • Altered motor activity and hepatic gene expression was seen in C57BL/6J mice following Mn exposure. • The Parkin gene defect did not increase the susceptibility of neonatal mice to Mn toxicity....
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Foster, Melanie L. [verfasserIn] |
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E-Artikel |
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Englisch |
| Erschienen: |
2018 |
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| Schlagwörter: |
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| Umfang: |
9 |
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| Übergeordnetes Werk: |
Enthalten in: Revealing the fate of cell surface human P-glycoprotein (ABCB1): The lysosomal degradation pathway - Katayama, Kazuhiro ELSEVIER, 2015transfer abstract, the interdisciplinary journal of effects to toxic substances on the nervous system, Amsterdam [u.a.] |
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| Übergeordnetes Werk: |
volume:64 ; year:2018 ; pages:291-299 ; extent:9 |
| Links: |
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| DOI / URN: |
10.1016/j.neuro.2017.10.002 |
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| 520 | |a • Parkin and C57BL/6J mice have similar brain manganese (Mn) levels following neonatal Mn exposure. • Altered motor activity and hepatic gene expression was seen in C57BL/6J mice following Mn exposure. • The Parkin gene defect did not increase the susceptibility of neonatal mice to Mn toxicity. | ||
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Neonatal C57BL/6J and parkin mice respond differently following developmental manganese exposure: Result of a high dose pilot study |
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• Parkin and C57BL/6J mice have similar brain manganese (Mn) levels following neonatal Mn exposure. • Altered motor activity and hepatic gene expression was seen in C57BL/6J mice following Mn exposure. • The Parkin gene defect did not increase the susceptibility of neonatal mice to Mn toxicity. |
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• Parkin and C57BL/6J mice have similar brain manganese (Mn) levels following neonatal Mn exposure. • Altered motor activity and hepatic gene expression was seen in C57BL/6J mice following Mn exposure. • The Parkin gene defect did not increase the susceptibility of neonatal mice to Mn toxicity. |
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• Parkin and C57BL/6J mice have similar brain manganese (Mn) levels following neonatal Mn exposure. • Altered motor activity and hepatic gene expression was seen in C57BL/6J mice following Mn exposure. • The Parkin gene defect did not increase the susceptibility of neonatal mice to Mn toxicity. |
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Neonatal C57BL/6J and parkin mice respond differently following developmental manganese exposure: Result of a high dose pilot study |
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Bartnikas, Thomas B. Maresca-Fichter, Hailey C. Mercadante, Courtney Dash, Miriam Miller, Chelsea Dorman, David C. |
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2024-07-06T21:21:52.930Z |
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