HIV-1 Tat and methamphetamine co-induced oxidative cellular injury is mitigated by <ce:italic>N</ce:italic>-acetylcysteine amide (NACA) through rectifying mTOR signaling
• Meth and Tat synergistically increased cell death in dose- and time-dependent manners. • Meth and Tat-induced autophagy and apoptosis in vitro and in vivo. • NACA confers protection against Tat- and/or Meth-induced and/or autophagy through mTOR signaling in vitro and in vivo.
Gespeichert in:
| Autor*in: |
Zeng, Xiao-Feng [verfasserIn] |
|---|
| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2018 |
|---|
| Schlagwörter: |
|---|
| Umfang: |
13 |
|---|
| Übergeordnetes Werk: |
Enthalten in: Toxicology letters - Chen, Yun-Ru ELSEVIER, 2019, an international journal for the rapid publication of short reports on all aspects of toxicology especially mechanisms of toxicity, Amsterdam [u.a.] |
|---|---|
| Übergeordnetes Werk: |
volume:299 ; year:2018 ; day:15 ; month:12 ; pages:159-171 ; extent:13 |
| Links: |
|---|
| DOI / URN: |
10.1016/j.toxlet.2018.09.009 |
|---|
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ELV044584741 |
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hiv-1 tat and methamphetamine co-induced oxidative cellular injury is mitigated by <ce:italic>n</ce:italic>-acetylcysteine amide (naca) through rectifying mtor signaling |
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HIV-1 Tat and methamphetamine co-induced oxidative cellular injury is mitigated by <ce:italic>N</ce:italic>-acetylcysteine amide (NACA) through rectifying mTOR signaling |
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• Meth and Tat synergistically increased cell death in dose- and time-dependent manners. • Meth and Tat-induced autophagy and apoptosis in vitro and in vivo. • NACA confers protection against Tat- and/or Meth-induced and/or autophagy through mTOR signaling in vitro and in vivo. |
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• Meth and Tat synergistically increased cell death in dose- and time-dependent manners. • Meth and Tat-induced autophagy and apoptosis in vitro and in vivo. • NACA confers protection against Tat- and/or Meth-induced and/or autophagy through mTOR signaling in vitro and in vivo. |
| abstract_unstemmed |
• Meth and Tat synergistically increased cell death in dose- and time-dependent manners. • Meth and Tat-induced autophagy and apoptosis in vitro and in vivo. • NACA confers protection against Tat- and/or Meth-induced and/or autophagy through mTOR signaling in vitro and in vivo. |
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GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA |
| title_short |
HIV-1 Tat and methamphetamine co-induced oxidative cellular injury is mitigated by <ce:italic>N</ce:italic>-acetylcysteine amide (NACA) through rectifying mTOR signaling |
| url |
https://doi.org/10.1016/j.toxlet.2018.09.009 |
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Li, Qi Li, Juan Wong, Naikei Li, Zhen Huang, Jian Yang, Genmeng Sham, Pak C. Li, Sheng-Bin Lu, Gang |
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Li, Qi Li, Juan Wong, Naikei Li, Zhen Huang, Jian Yang, Genmeng Sham, Pak C. Li, Sheng-Bin Lu, Gang |
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| doi_str |
10.1016/j.toxlet.2018.09.009 |
| up_date |
2024-07-06T21:52:49.204Z |
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