Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic>
It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transiti...
Ausführliche Beschreibung
Autor*in: |
han, Yangyang [verfasserIn] |
---|
Format: |
E-Artikel |
---|---|
Sprache: |
Englisch |
Erschienen: |
2019transfer abstract |
---|
Schlagwörter: |
---|
Übergeordnetes Werk: |
Enthalten in: Concentration of trisodium citrate by electrodialysis - Fidaleo, Marcello ELSEVIER, 2013, München |
---|---|
Übergeordnetes Werk: |
volume:215 ; year:2019 ; number:10 ; pages:0 |
Links: |
---|
DOI / URN: |
10.1016/j.prp.2019.152537 |
---|
Katalog-ID: |
ELV047989459 |
---|
LEADER | 01000caa a22002652 4500 | ||
---|---|---|---|
001 | ELV047989459 | ||
003 | DE-627 | ||
005 | 20230626020945.0 | ||
007 | cr uuu---uuuuu | ||
008 | 191023s2019 xx |||||o 00| ||eng c | ||
024 | 7 | |a 10.1016/j.prp.2019.152537 |2 doi | |
028 | 5 | 2 | |a GBV00000000000754.pica |
035 | |a (DE-627)ELV047989459 | ||
035 | |a (ELSEVIER)S0344-0338(19)30588-6 | ||
040 | |a DE-627 |b ger |c DE-627 |e rakwb | ||
041 | |a eng | ||
082 | 0 | 4 | |a 570 |q VZ |
082 | 0 | 4 | |a 540 |q VZ |
084 | |a 35.17 |2 bkl | ||
084 | |a 58.50 |2 bkl | ||
084 | |a 43.12 |2 bkl | ||
100 | 1 | |a han, Yangyang |e verfasserin |4 aut | |
245 | 1 | 0 | |a Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> |
264 | 1 | |c 2019transfer abstract | |
336 | |a nicht spezifiziert |b zzz |2 rdacontent | ||
337 | |a nicht spezifiziert |b z |2 rdamedia | ||
338 | |a nicht spezifiziert |b zu |2 rdacarrier | ||
520 | |a It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. | ||
520 | |a It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. | ||
650 | 7 | |a Epithelial-mesenchymal transition |2 Elsevier | |
650 | 7 | |a WNT2B |2 Elsevier | |
650 | 7 | |a LncRNA |2 Elsevier | |
650 | 7 | |a SNHG7 |2 Elsevier | |
650 | 7 | |a Prostate cancer |2 Elsevier | |
700 | 1 | |a Hu, Haibo |4 oth | |
700 | 1 | |a zhou, jinsong |4 oth | |
773 | 0 | 8 | |i Enthalten in |n Elsevier |a Fidaleo, Marcello ELSEVIER |t Concentration of trisodium citrate by electrodialysis |d 2013 |g München |w (DE-627)ELV016908384 |
773 | 1 | 8 | |g volume:215 |g year:2019 |g number:10 |g pages:0 |
856 | 4 | 0 | |u https://doi.org/10.1016/j.prp.2019.152537 |3 Volltext |
912 | |a GBV_USEFLAG_U | ||
912 | |a GBV_ELV | ||
912 | |a SYSFLAG_U | ||
912 | |a SSG-OLC-PHA | ||
912 | |a GBV_ILN_70 | ||
936 | b | k | |a 35.17 |j Katalyse |q VZ |
936 | b | k | |a 58.50 |j Umwelttechnik: Allgemeines |q VZ |
936 | b | k | |a 43.12 |j Umweltchemie |q VZ |
951 | |a AR | ||
952 | |d 215 |j 2019 |e 10 |h 0 |
author_variant |
y h yh |
---|---|
matchkey_str |
hanyangyanghuhaibozhoujinsong:2019----:ncdwolcnsh7niieeihlamsnhmlrniinnrsaeacrhuhi34p |
hierarchy_sort_str |
2019transfer abstract |
bklnumber |
35.17 58.50 43.12 |
publishDate |
2019 |
allfields |
10.1016/j.prp.2019.152537 doi GBV00000000000754.pica (DE-627)ELV047989459 (ELSEVIER)S0344-0338(19)30588-6 DE-627 ger DE-627 rakwb eng 570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl han, Yangyang verfasserin aut Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> 2019transfer abstract nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Elsevier Hu, Haibo oth zhou, jinsong oth Enthalten in Elsevier Fidaleo, Marcello ELSEVIER Concentration of trisodium citrate by electrodialysis 2013 München (DE-627)ELV016908384 volume:215 year:2019 number:10 pages:0 https://doi.org/10.1016/j.prp.2019.152537 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_70 35.17 Katalyse VZ 58.50 Umwelttechnik: Allgemeines VZ 43.12 Umweltchemie VZ AR 215 2019 10 0 |
spelling |
10.1016/j.prp.2019.152537 doi GBV00000000000754.pica (DE-627)ELV047989459 (ELSEVIER)S0344-0338(19)30588-6 DE-627 ger DE-627 rakwb eng 570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl han, Yangyang verfasserin aut Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> 2019transfer abstract nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Elsevier Hu, Haibo oth zhou, jinsong oth Enthalten in Elsevier Fidaleo, Marcello ELSEVIER Concentration of trisodium citrate by electrodialysis 2013 München (DE-627)ELV016908384 volume:215 year:2019 number:10 pages:0 https://doi.org/10.1016/j.prp.2019.152537 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_70 35.17 Katalyse VZ 58.50 Umwelttechnik: Allgemeines VZ 43.12 Umweltchemie VZ AR 215 2019 10 0 |
allfields_unstemmed |
10.1016/j.prp.2019.152537 doi GBV00000000000754.pica (DE-627)ELV047989459 (ELSEVIER)S0344-0338(19)30588-6 DE-627 ger DE-627 rakwb eng 570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl han, Yangyang verfasserin aut Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> 2019transfer abstract nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Elsevier Hu, Haibo oth zhou, jinsong oth Enthalten in Elsevier Fidaleo, Marcello ELSEVIER Concentration of trisodium citrate by electrodialysis 2013 München (DE-627)ELV016908384 volume:215 year:2019 number:10 pages:0 https://doi.org/10.1016/j.prp.2019.152537 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_70 35.17 Katalyse VZ 58.50 Umwelttechnik: Allgemeines VZ 43.12 Umweltchemie VZ AR 215 2019 10 0 |
allfieldsGer |
10.1016/j.prp.2019.152537 doi GBV00000000000754.pica (DE-627)ELV047989459 (ELSEVIER)S0344-0338(19)30588-6 DE-627 ger DE-627 rakwb eng 570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl han, Yangyang verfasserin aut Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> 2019transfer abstract nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Elsevier Hu, Haibo oth zhou, jinsong oth Enthalten in Elsevier Fidaleo, Marcello ELSEVIER Concentration of trisodium citrate by electrodialysis 2013 München (DE-627)ELV016908384 volume:215 year:2019 number:10 pages:0 https://doi.org/10.1016/j.prp.2019.152537 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_70 35.17 Katalyse VZ 58.50 Umwelttechnik: Allgemeines VZ 43.12 Umweltchemie VZ AR 215 2019 10 0 |
allfieldsSound |
10.1016/j.prp.2019.152537 doi GBV00000000000754.pica (DE-627)ELV047989459 (ELSEVIER)S0344-0338(19)30588-6 DE-627 ger DE-627 rakwb eng 570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl han, Yangyang verfasserin aut Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> 2019transfer abstract nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Elsevier Hu, Haibo oth zhou, jinsong oth Enthalten in Elsevier Fidaleo, Marcello ELSEVIER Concentration of trisodium citrate by electrodialysis 2013 München (DE-627)ELV016908384 volume:215 year:2019 number:10 pages:0 https://doi.org/10.1016/j.prp.2019.152537 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_70 35.17 Katalyse VZ 58.50 Umwelttechnik: Allgemeines VZ 43.12 Umweltchemie VZ AR 215 2019 10 0 |
language |
English |
source |
Enthalten in Concentration of trisodium citrate by electrodialysis München volume:215 year:2019 number:10 pages:0 |
sourceStr |
Enthalten in Concentration of trisodium citrate by electrodialysis München volume:215 year:2019 number:10 pages:0 |
format_phy_str_mv |
Article |
bklname |
Katalyse Umwelttechnik: Allgemeines Umweltchemie |
institution |
findex.gbv.de |
topic_facet |
Epithelial-mesenchymal transition WNT2B LncRNA SNHG7 Prostate cancer |
dewey-raw |
570 |
isfreeaccess_bool |
false |
container_title |
Concentration of trisodium citrate by electrodialysis |
authorswithroles_txt_mv |
han, Yangyang @@aut@@ Hu, Haibo @@oth@@ zhou, jinsong @@oth@@ |
publishDateDaySort_date |
2019-01-01T00:00:00Z |
hierarchy_top_id |
ELV016908384 |
dewey-sort |
3570 |
id |
ELV047989459 |
language_de |
englisch |
fullrecord |
<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">ELV047989459</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230626020945.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">191023s2019 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1016/j.prp.2019.152537</subfield><subfield code="2">doi</subfield></datafield><datafield tag="028" ind1="5" ind2="2"><subfield code="a">GBV00000000000754.pica</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)ELV047989459</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ELSEVIER)S0344-0338(19)30588-6</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">570</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">540</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">35.17</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">58.50</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">43.12</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">han, Yangyang</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic></subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2019transfer abstract</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment.</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment.</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Epithelial-mesenchymal transition</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">WNT2B</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">LncRNA</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">SNHG7</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Prostate cancer</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Hu, Haibo</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">zhou, jinsong</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="n">Elsevier</subfield><subfield code="a">Fidaleo, Marcello ELSEVIER</subfield><subfield code="t">Concentration of trisodium citrate by electrodialysis</subfield><subfield code="d">2013</subfield><subfield code="g">München</subfield><subfield code="w">(DE-627)ELV016908384</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:215</subfield><subfield code="g">year:2019</subfield><subfield code="g">number:10</subfield><subfield code="g">pages:0</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1016/j.prp.2019.152537</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ELV</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OLC-PHA</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_70</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">35.17</subfield><subfield code="j">Katalyse</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">58.50</subfield><subfield code="j">Umwelttechnik: Allgemeines</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">43.12</subfield><subfield code="j">Umweltchemie</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">215</subfield><subfield code="j">2019</subfield><subfield code="e">10</subfield><subfield code="h">0</subfield></datafield></record></collection>
|
author |
han, Yangyang |
spellingShingle |
han, Yangyang ddc 570 ddc 540 bkl 35.17 bkl 58.50 bkl 43.12 Elsevier Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> |
authorStr |
han, Yangyang |
ppnlink_with_tag_str_mv |
@@773@@(DE-627)ELV016908384 |
format |
electronic Article |
dewey-ones |
570 - Life sciences; biology 540 - Chemistry & allied sciences |
delete_txt_mv |
keep |
author_role |
aut |
collection |
elsevier |
remote_str |
true |
illustrated |
Not Illustrated |
topic_title |
570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer Elsevier |
topic |
ddc 570 ddc 540 bkl 35.17 bkl 58.50 bkl 43.12 Elsevier Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer |
topic_unstemmed |
ddc 570 ddc 540 bkl 35.17 bkl 58.50 bkl 43.12 Elsevier Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer |
topic_browse |
ddc 570 ddc 540 bkl 35.17 bkl 58.50 bkl 43.12 Elsevier Epithelial-mesenchymal transition Elsevier WNT2B Elsevier LncRNA Elsevier SNHG7 Elsevier Prostate cancer |
format_facet |
Elektronische Aufsätze Aufsätze Elektronische Ressource |
format_main_str_mv |
Text Zeitschrift/Artikel |
carriertype_str_mv |
zu |
author2_variant |
h h hh j z jz |
hierarchy_parent_title |
Concentration of trisodium citrate by electrodialysis |
hierarchy_parent_id |
ELV016908384 |
dewey-tens |
570 - Life sciences; biology 540 - Chemistry |
hierarchy_top_title |
Concentration of trisodium citrate by electrodialysis |
isfreeaccess_txt |
false |
familylinks_str_mv |
(DE-627)ELV016908384 |
title |
Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> |
ctrlnum |
(DE-627)ELV047989459 (ELSEVIER)S0344-0338(19)30588-6 |
title_full |
Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> |
author_sort |
han, Yangyang |
journal |
Concentration of trisodium citrate by electrodialysis |
journalStr |
Concentration of trisodium citrate by electrodialysis |
lang_code |
eng |
isOA_bool |
false |
dewey-hundreds |
500 - Science |
recordtype |
marc |
publishDateSort |
2019 |
contenttype_str_mv |
zzz |
container_start_page |
0 |
author_browse |
han, Yangyang |
container_volume |
215 |
class |
570 VZ 540 VZ 35.17 bkl 58.50 bkl 43.12 bkl |
format_se |
Elektronische Aufsätze |
author-letter |
han, Yangyang |
doi_str_mv |
10.1016/j.prp.2019.152537 |
dewey-full |
570 540 |
title_sort |
knockdown of lncrna snhg7 inhibited epithelial-mesenchymal transition in prostate cancer though mir-324-3p/wnt2b axis <ce:italic>in vitro</ce:italic> |
title_auth |
Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> |
abstract |
It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. |
abstractGer |
It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. |
abstract_unstemmed |
It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment. |
collection_details |
GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_70 |
container_issue |
10 |
title_short |
Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic> |
url |
https://doi.org/10.1016/j.prp.2019.152537 |
remote_bool |
true |
author2 |
Hu, Haibo zhou, jinsong |
author2Str |
Hu, Haibo zhou, jinsong |
ppnlink |
ELV016908384 |
mediatype_str_mv |
z |
isOA_txt |
false |
hochschulschrift_bool |
false |
author2_role |
oth oth |
doi_str |
10.1016/j.prp.2019.152537 |
up_date |
2024-07-06T17:39:47.985Z |
_version_ |
1803852277860007936 |
fullrecord_marcxml |
<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">ELV047989459</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230626020945.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">191023s2019 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1016/j.prp.2019.152537</subfield><subfield code="2">doi</subfield></datafield><datafield tag="028" ind1="5" ind2="2"><subfield code="a">GBV00000000000754.pica</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)ELV047989459</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ELSEVIER)S0344-0338(19)30588-6</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">570</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">540</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">35.17</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">58.50</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">43.12</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">han, Yangyang</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Knockdown of LncRNA SNHG7 inhibited epithelial-mesenchymal transition in prostate cancer though miR-324-3p/WNT2B axis <ce:italic>in vitro</ce:italic></subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2019transfer abstract</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment.</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">It is identified that long non-coding RNAs (lncRNAs) play important roles in cancer progression and metastasis. LncRNA SNHG7 was reported to play an oncogenic role in the progression of cancers including prostate cancer. However, its potential regulatory mechanism of endothelial-mesenchymal transition in PCa remains unclear. In this study, We found a lncRNA SNHG7 was overexpressed in PCa cell lines and tissues. LncRNA SNHG7 promotes prostate cancer migration and invasion by modulating EMT. Further study indicated that lncRNA SNHG7 acts as a sponge for miRNA-324-3p and positively regulates WNT2B by a sponge effect. Moreover, We confirmed that WNT2B, an important protein in the Wnt signal pathway, promotes the malignant phenotype of PCa cells and mediated the biological effects exerted by lncRNA SNHG7. Overall, our study suggested that lncRNA SNHG7 could promote PCa EMT via miR-324-3p and WNT2B in vitro. The lncRNA SNHG7/miR-324-3p /WNT2B axis regulatory network might provide a potential new therapeutic strategy for PCa treatment.</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Epithelial-mesenchymal transition</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">WNT2B</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">LncRNA</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">SNHG7</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="650" ind1=" " ind2="7"><subfield code="a">Prostate cancer</subfield><subfield code="2">Elsevier</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Hu, Haibo</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">zhou, jinsong</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="n">Elsevier</subfield><subfield code="a">Fidaleo, Marcello ELSEVIER</subfield><subfield code="t">Concentration of trisodium citrate by electrodialysis</subfield><subfield code="d">2013</subfield><subfield code="g">München</subfield><subfield code="w">(DE-627)ELV016908384</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:215</subfield><subfield code="g">year:2019</subfield><subfield code="g">number:10</subfield><subfield code="g">pages:0</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1016/j.prp.2019.152537</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ELV</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OLC-PHA</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_70</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">35.17</subfield><subfield code="j">Katalyse</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">58.50</subfield><subfield code="j">Umwelttechnik: Allgemeines</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="936" ind1="b" ind2="k"><subfield code="a">43.12</subfield><subfield code="j">Umweltchemie</subfield><subfield code="q">VZ</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">215</subfield><subfield code="j">2019</subfield><subfield code="e">10</subfield><subfield code="h">0</subfield></datafield></record></collection>
|
score |
7.398529 |