Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques
Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scar...
Ausführliche Beschreibung
Autor*in: |
Rafnsson, Arnar [verfasserIn] |
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E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2020 |
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Umfang: |
9 |
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Übergeordnetes Werk: |
Enthalten in: No title available - 292(2020), Seite 215-223 |
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Übergeordnetes Werk: |
volume:292 ; year:2020 ; pages:215-223 ; extent:9 |
Links: |
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DOI / URN: |
10.1016/j.atherosclerosis.2019.09.020 |
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10.1016/j.atherosclerosis.2019.09.020 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001073.pica (DE-627)ELV048847003 (ELSEVIER)S0021-9150(19)31507-2 DE-627 ger DE-627 rakwb eng Rafnsson, Arnar verfasserin aut Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques 2020 9 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. Endothelin receptor A Elsevier Endothelin receptor B Elsevier Endothelin-1 Elsevier Endothelial dysfunction Elsevier Carotid artery endarterectomy Elsevier Arginase Elsevier Endothelin receptor antagonist Elsevier Matic, Ljubica Perisic oth Lengquist, Mariette oth Mahdi, Ali oth Shemyakin, Alexey oth Paulsson-Berne, Gabrielle oth Hansson, Göran K. oth Gabrielsen, Anders oth Hedin, Ulf oth Yang, Jiangning oth Pernow, John oth Enthalten in No title available 292(2020), Seite 215-223 (DE-627)ELV012595616 (DE-600)1-9150 nnns volume:292 year:2020 pages:215-223 extent:9 https://doi.org/10.1016/j.atherosclerosis.2019.09.020 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U GBV_ILN_20 GBV_ILN_40 GBV_ILN_105 AR 292 2020 215-223 9 |
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10.1016/j.atherosclerosis.2019.09.020 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001073.pica (DE-627)ELV048847003 (ELSEVIER)S0021-9150(19)31507-2 DE-627 ger DE-627 rakwb eng Rafnsson, Arnar verfasserin aut Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques 2020 9 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. Endothelin receptor A Elsevier Endothelin receptor B Elsevier Endothelin-1 Elsevier Endothelial dysfunction Elsevier Carotid artery endarterectomy Elsevier Arginase Elsevier Endothelin receptor antagonist Elsevier Matic, Ljubica Perisic oth Lengquist, Mariette oth Mahdi, Ali oth Shemyakin, Alexey oth Paulsson-Berne, Gabrielle oth Hansson, Göran K. oth Gabrielsen, Anders oth Hedin, Ulf oth Yang, Jiangning oth Pernow, John oth Enthalten in No title available 292(2020), Seite 215-223 (DE-627)ELV012595616 (DE-600)1-9150 nnns volume:292 year:2020 pages:215-223 extent:9 https://doi.org/10.1016/j.atherosclerosis.2019.09.020 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U GBV_ILN_20 GBV_ILN_40 GBV_ILN_105 AR 292 2020 215-223 9 |
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10.1016/j.atherosclerosis.2019.09.020 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001073.pica (DE-627)ELV048847003 (ELSEVIER)S0021-9150(19)31507-2 DE-627 ger DE-627 rakwb eng Rafnsson, Arnar verfasserin aut Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques 2020 9 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. Endothelin receptor A Elsevier Endothelin receptor B Elsevier Endothelin-1 Elsevier Endothelial dysfunction Elsevier Carotid artery endarterectomy Elsevier Arginase Elsevier Endothelin receptor antagonist Elsevier Matic, Ljubica Perisic oth Lengquist, Mariette oth Mahdi, Ali oth Shemyakin, Alexey oth Paulsson-Berne, Gabrielle oth Hansson, Göran K. oth Gabrielsen, Anders oth Hedin, Ulf oth Yang, Jiangning oth Pernow, John oth Enthalten in No title available 292(2020), Seite 215-223 (DE-627)ELV012595616 (DE-600)1-9150 nnns volume:292 year:2020 pages:215-223 extent:9 https://doi.org/10.1016/j.atherosclerosis.2019.09.020 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U GBV_ILN_20 GBV_ILN_40 GBV_ILN_105 AR 292 2020 215-223 9 |
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10.1016/j.atherosclerosis.2019.09.020 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001073.pica (DE-627)ELV048847003 (ELSEVIER)S0021-9150(19)31507-2 DE-627 ger DE-627 rakwb eng Rafnsson, Arnar verfasserin aut Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques 2020 9 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. Endothelin receptor A Elsevier Endothelin receptor B Elsevier Endothelin-1 Elsevier Endothelial dysfunction Elsevier Carotid artery endarterectomy Elsevier Arginase Elsevier Endothelin receptor antagonist Elsevier Matic, Ljubica Perisic oth Lengquist, Mariette oth Mahdi, Ali oth Shemyakin, Alexey oth Paulsson-Berne, Gabrielle oth Hansson, Göran K. oth Gabrielsen, Anders oth Hedin, Ulf oth Yang, Jiangning oth Pernow, John oth Enthalten in No title available 292(2020), Seite 215-223 (DE-627)ELV012595616 (DE-600)1-9150 nnns volume:292 year:2020 pages:215-223 extent:9 https://doi.org/10.1016/j.atherosclerosis.2019.09.020 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U GBV_ILN_20 GBV_ILN_40 GBV_ILN_105 AR 292 2020 215-223 9 |
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10.1016/j.atherosclerosis.2019.09.020 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001073.pica (DE-627)ELV048847003 (ELSEVIER)S0021-9150(19)31507-2 DE-627 ger DE-627 rakwb eng Rafnsson, Arnar verfasserin aut Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques 2020 9 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. Endothelin receptor A Elsevier Endothelin receptor B Elsevier Endothelin-1 Elsevier Endothelial dysfunction Elsevier Carotid artery endarterectomy Elsevier Arginase Elsevier Endothelin receptor antagonist Elsevier Matic, Ljubica Perisic oth Lengquist, Mariette oth Mahdi, Ali oth Shemyakin, Alexey oth Paulsson-Berne, Gabrielle oth Hansson, Göran K. oth Gabrielsen, Anders oth Hedin, Ulf oth Yang, Jiangning oth Pernow, John oth Enthalten in No title available 292(2020), Seite 215-223 (DE-627)ELV012595616 (DE-600)1-9150 nnns volume:292 year:2020 pages:215-223 extent:9 https://doi.org/10.1016/j.atherosclerosis.2019.09.020 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U GBV_ILN_20 GBV_ILN_40 GBV_ILN_105 AR 292 2020 215-223 9 |
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Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques |
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Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. |
abstractGer |
Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. |
abstract_unstemmed |
Endothelin-1 (ET-1) and arginase are both suggested to be involved in the inflammatory processes and development of endothelial dysfunction in atherosclerosis. However, information regarding the roles of ET-1 and arginase, as well as the interactions between the two in human atherosclerosis, is scarce. We investigated the expression of ET-1 and its receptors, ETA and ETB, as well as arginase in human carotid atherosclerotic plaques and determined the functional interactions between ET-1 and arginase in endothelial cells and THP-1-derived macrophages. |
collection_details |
GBV_USEFLAG_U GBV_ELV SYSFLAG_U GBV_ILN_20 GBV_ILN_40 GBV_ILN_105 |
title_short |
Endothelin-1 increases expression and activity of arginase 2 via ETB receptors and is co-expressed with arginase 2 in human atherosclerotic plaques |
url |
https://doi.org/10.1016/j.atherosclerosis.2019.09.020 |
remote_bool |
true |
author2 |
Matic, Ljubica Perisic Lengquist, Mariette Mahdi, Ali Shemyakin, Alexey Paulsson-Berne, Gabrielle Hansson, Göran K. Gabrielsen, Anders Hedin, Ulf Yang, Jiangning Pernow, John |
author2Str |
Matic, Ljubica Perisic Lengquist, Mariette Mahdi, Ali Shemyakin, Alexey Paulsson-Berne, Gabrielle Hansson, Göran K. Gabrielsen, Anders Hedin, Ulf Yang, Jiangning Pernow, John |
ppnlink |
ELV012595616 |
mediatype_str_mv |
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isOA_txt |
false |
hochschulschrift_bool |
false |
author2_role |
oth oth oth oth oth oth oth oth oth oth |
doi_str |
10.1016/j.atherosclerosis.2019.09.020 |
up_date |
2024-07-06T19:56:48.301Z |
_version_ |
1803860897487126528 |
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7.400717 |