MiR-29a in mesenchymal stem cells inhibits FSTL1 secretion and promotes cardiac myocyte apoptosis in hypoxia-reoxygenation injury
Mesenchymal stem cells (MSCs) are under consideration for myocardial ischemia-reperfusion (I/R) injury therapy, but their mechanism remains to be evaluated. In this article, we aimed to study the effects of the miR-29a/follistatin-like 1 axis in bone marrow–derived mesenchymal stem cells on modulati...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Li, Kun-Sheng [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2020 |
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| Schlagwörter: |
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| Übergeordnetes Werk: |
Enthalten in: Structural and magnetic properties of geometrically frustrated multiferroic ErMnO3 nanoparticles - 2013, Amsterdam [u.a.] |
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| Übergeordnetes Werk: |
volume:46 ; year:2020 ; pages:0 |
| Links: |
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| DOI / URN: |
10.1016/j.carpath.2019.107180 |
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ELV049671367 |
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| 520 | |a Mesenchymal stem cells (MSCs) are under consideration for myocardial ischemia-reperfusion (I/R) injury therapy, but their mechanism remains to be evaluated. In this article, we aimed to study the effects of the miR-29a/follistatin-like 1 axis in bone marrow–derived mesenchymal stem cells on modulating myocyte apoptosis after hypoxia-reoxygenation (H/R) injury. | ||
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10.1016/j.carpath.2019.107180 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000000941.pica (DE-627)ELV049671367 (ELSEVIER)S1054-8807(19)30345-X DE-627 ger DE-627 rakwb eng 670 VZ 540 VZ 630 VZ Li, Kun-Sheng verfasserin aut MiR-29a in mesenchymal stem cells inhibits FSTL1 secretion and promotes cardiac myocyte apoptosis in hypoxia-reoxygenation injury 2020 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Mesenchymal stem cells (MSCs) are under consideration for myocardial ischemia-reperfusion (I/R) injury therapy, but their mechanism remains to be evaluated. In this article, we aimed to study the effects of the miR-29a/follistatin-like 1 axis in bone marrow–derived mesenchymal stem cells on modulating myocyte apoptosis after hypoxia-reoxygenation (H/R) injury. MiR-29a Elsevier FSTL1 Elsevier JAK2/STAT3 pathway Elsevier Mesenchymal stem cells Elsevier Myocardial injury Elsevier Jiang, Wei-Peng oth Li, Qiu-Chang oth Zhang, Hao-Wen oth Bai, Yang oth Zhang, Xia oth Li, Hai-Ying oth Enthalten in Elsevier Science Structural and magnetic properties of geometrically frustrated multiferroic ErMnO3 nanoparticles 2013 Amsterdam [u.a.] (DE-627)ELV011715340 volume:46 year:2020 pages:0 https://doi.org/10.1016/j.carpath.2019.107180 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U SSG-OLC-PHA GBV_ILN_120 AR 46 2020 0 |
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MiR-29a in mesenchymal stem cells inhibits FSTL1 secretion and promotes cardiac myocyte apoptosis in hypoxia-reoxygenation injury |
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Mesenchymal stem cells (MSCs) are under consideration for myocardial ischemia-reperfusion (I/R) injury therapy, but their mechanism remains to be evaluated. In this article, we aimed to study the effects of the miR-29a/follistatin-like 1 axis in bone marrow–derived mesenchymal stem cells on modulating myocyte apoptosis after hypoxia-reoxygenation (H/R) injury. |
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Mesenchymal stem cells (MSCs) are under consideration for myocardial ischemia-reperfusion (I/R) injury therapy, but their mechanism remains to be evaluated. In this article, we aimed to study the effects of the miR-29a/follistatin-like 1 axis in bone marrow–derived mesenchymal stem cells on modulating myocyte apoptosis after hypoxia-reoxygenation (H/R) injury. |
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