Dolutegravir derivative inhibits proliferation and induces apoptosis of non-small cell lung cancer cells via calcium signaling pathway
DTHP, a novel derivative of DTG, inhibits cell proliferation and induces apoptosis of NSCLC cells via the Ca2+ signaling pathway, leading to the activation of CaMKK / AMPK proteins, excessive ROS production and reduction of mitochondria membrane potential.
Gespeichert in:
| Autor*in: |
Wang, Wen-Jun [verfasserIn] |
|---|
| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2020 |
|---|
| Schlagwörter: |
|---|
| Übergeordnetes Werk: |
Enthalten in: Social insurance premiums and corporate cash holdings: Evidence from social insurance law in China - Deng, Lixing ELSEVIER, 2022, the official journal of the Italian Pharmacological Society, London |
|---|---|
| Übergeordnetes Werk: |
volume:161 ; year:2020 ; pages:0 |
| Links: |
|---|
| DOI / URN: |
10.1016/j.phrs.2020.105129 |
|---|
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10.1016/j.phrs.2020.105129 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001465.pica (DE-627)ELV05250882X (ELSEVIER)S1043-6618(20)31437-7 DE-627 ger DE-627 rakwb eng 330 VZ Wang, Wen-Jun verfasserin aut Dolutegravir derivative inhibits proliferation and induces apoptosis of non-small cell lung cancer cells via calcium signaling pathway 2020 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier DTHP, a novel derivative of DTG, inhibits cell proliferation and induces apoptosis of NSCLC cells via the Ca2+ signaling pathway, leading to the activation of CaMKK / AMPK proteins, excessive ROS production and reduction of mitochondria membrane potential. NSCLC Elsevier Ca2+ levels Elsevier ROS Elsevier AMPK Elsevier Dolutegravir derivative Elsevier SERCA Elsevier Mao, Long-Fei oth Lai, Huan-Ling oth Wang, Yu-Wei oth Jiang, Ze-Bo oth Li, Wei oth Huang, Ju-Min oth Xie, Ya-Jia oth Xu, Cong oth Liu, Pei oth Li, Yue-Ming oth Leung, Elaine Lai Han oth Yao, Xiao-Jun oth Enthalten in Academic Press Deng, Lixing ELSEVIER Social insurance premiums and corporate cash holdings: Evidence from social insurance law in China 2022 the official journal of the Italian Pharmacological Society London (DE-627)ELV008224951 volume:161 year:2020 pages:0 https://doi.org/10.1016/j.phrs.2020.105129 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U AR 161 2020 0 |
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10.1016/j.phrs.2020.105129 doi /cbs_pica/cbs_olc/import_discovery/elsevier/einzuspielen/GBV00000000001465.pica (DE-627)ELV05250882X (ELSEVIER)S1043-6618(20)31437-7 DE-627 ger DE-627 rakwb eng 330 VZ Wang, Wen-Jun verfasserin aut Dolutegravir derivative inhibits proliferation and induces apoptosis of non-small cell lung cancer cells via calcium signaling pathway 2020 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier DTHP, a novel derivative of DTG, inhibits cell proliferation and induces apoptosis of NSCLC cells via the Ca2+ signaling pathway, leading to the activation of CaMKK / AMPK proteins, excessive ROS production and reduction of mitochondria membrane potential. NSCLC Elsevier Ca2+ levels Elsevier ROS Elsevier AMPK Elsevier Dolutegravir derivative Elsevier SERCA Elsevier Mao, Long-Fei oth Lai, Huan-Ling oth Wang, Yu-Wei oth Jiang, Ze-Bo oth Li, Wei oth Huang, Ju-Min oth Xie, Ya-Jia oth Xu, Cong oth Liu, Pei oth Li, Yue-Ming oth Leung, Elaine Lai Han oth Yao, Xiao-Jun oth Enthalten in Academic Press Deng, Lixing ELSEVIER Social insurance premiums and corporate cash holdings: Evidence from social insurance law in China 2022 the official journal of the Italian Pharmacological Society London (DE-627)ELV008224951 volume:161 year:2020 pages:0 https://doi.org/10.1016/j.phrs.2020.105129 Volltext GBV_USEFLAG_U GBV_ELV SYSFLAG_U AR 161 2020 0 |
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DTHP, a novel derivative of DTG, inhibits cell proliferation and induces apoptosis of NSCLC cells via the Ca2+ signaling pathway, leading to the activation of CaMKK / AMPK proteins, excessive ROS production and reduction of mitochondria membrane potential. |
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DTHP, a novel derivative of DTG, inhibits cell proliferation and induces apoptosis of NSCLC cells via the Ca2+ signaling pathway, leading to the activation of CaMKK / AMPK proteins, excessive ROS production and reduction of mitochondria membrane potential. |
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