Targeting c-Met to Improve Immune Checkpoint Inhibition in Metastatic Renal Cell Carcinoma
c-Met inhibition has direct antitumor effects on proliferation, angiogenesis, and metastasis, and indirect antitumor effects via prevention of the induction of immunosuppressive mechanisms (PD-L1, TGFβ, IDO1). c-Met inhibition is key to overcome resistance to immune checkpoint inhibition and to maxi...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Lindner, Andrea Katharina [verfasserIn] |
|---|
| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2022 |
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| Schlagwörter: |
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| Umfang: |
2 |
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| Übergeordnetes Werk: |
Enthalten in: Phase transition and alternation in a model of perceptual bistability in the presence of Lévy noise - Feng, Jing ELSEVIER, 2018, official organ of the European Association of Urology, the European Organization for Research and Treatment of Cancer - Genito-Urinary Group, the European Society for Urological Oncology and Endocrinology, Amsterdam [u.a.] |
|---|---|
| Übergeordnetes Werk: |
volume:81 ; year:2022 ; number:1 ; pages:1-2 ; extent:2 |
| Links: |
|---|
| DOI / URN: |
10.1016/j.eururo.2021.10.025 |
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ELV056261764 |
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c-Met inhibition has direct antitumor effects on proliferation, angiogenesis, and metastasis, and indirect antitumor effects via prevention of the induction of immunosuppressive mechanisms (PD-L1, TGFβ, IDO1). c-Met inhibition is key to overcome resistance to immune checkpoint inhibition and to maximize the efficacy of immunotherapy. |
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c-Met inhibition has direct antitumor effects on proliferation, angiogenesis, and metastasis, and indirect antitumor effects via prevention of the induction of immunosuppressive mechanisms (PD-L1, TGFβ, IDO1). c-Met inhibition is key to overcome resistance to immune checkpoint inhibition and to maximize the efficacy of immunotherapy. |
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c-Met inhibition has direct antitumor effects on proliferation, angiogenesis, and metastasis, and indirect antitumor effects via prevention of the induction of immunosuppressive mechanisms (PD-L1, TGFβ, IDO1). c-Met inhibition is key to overcome resistance to immune checkpoint inhibition and to maximize the efficacy of immunotherapy. |
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