Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference
A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be o...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Rubin, Sergio [verfasserIn] |
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| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2023 |
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| Schlagwörter: |
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| Übergeordnetes Werk: |
Enthalten in: Biosystems - Amsterdam [u.a.] : Elsevier Science, 1967, 230 |
|---|---|
| Übergeordnetes Werk: |
volume:230 |
| DOI / URN: |
10.1016/j.biosystems.2023.104955 |
|---|
| Katalog-ID: |
ELV060281820 |
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| 245 | 1 | 0 | |a Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference |
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| 520 | |a A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. | ||
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10.1016/j.biosystems.2023.104955 doi (DE-627)ELV060281820 (ELSEVIER)S0303-2647(23)00130-2 DE-627 ger DE-627 rda eng 570 VZ BIODIV DE-30 fid 44.00 bkl Rubin, Sergio verfasserin (orcid)0000-0002-3387-7760 aut Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference 2023 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. Self-fabrication Operational closure Closure to efficient causation Calculus of self-reference Non-algorithmic Enaction Final cause Enthalten in Biosystems Amsterdam [u.a.] : Elsevier Science, 1967 230 Online-Ressource (DE-627)306320843 (DE-600)1496359-0 (DE-576)081952953 nnns volume:230 GBV_USEFLAG_U GBV_ELV SYSFLAG_U FID-BIODIV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.00 Medizin: Allgemeines VZ AR 230 |
| spelling |
10.1016/j.biosystems.2023.104955 doi (DE-627)ELV060281820 (ELSEVIER)S0303-2647(23)00130-2 DE-627 ger DE-627 rda eng 570 VZ BIODIV DE-30 fid 44.00 bkl Rubin, Sergio verfasserin (orcid)0000-0002-3387-7760 aut Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference 2023 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. Self-fabrication Operational closure Closure to efficient causation Calculus of self-reference Non-algorithmic Enaction Final cause Enthalten in Biosystems Amsterdam [u.a.] : Elsevier Science, 1967 230 Online-Ressource (DE-627)306320843 (DE-600)1496359-0 (DE-576)081952953 nnns volume:230 GBV_USEFLAG_U GBV_ELV SYSFLAG_U FID-BIODIV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.00 Medizin: Allgemeines VZ AR 230 |
| allfields_unstemmed |
10.1016/j.biosystems.2023.104955 doi (DE-627)ELV060281820 (ELSEVIER)S0303-2647(23)00130-2 DE-627 ger DE-627 rda eng 570 VZ BIODIV DE-30 fid 44.00 bkl Rubin, Sergio verfasserin (orcid)0000-0002-3387-7760 aut Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference 2023 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. Self-fabrication Operational closure Closure to efficient causation Calculus of self-reference Non-algorithmic Enaction Final cause Enthalten in Biosystems Amsterdam [u.a.] : Elsevier Science, 1967 230 Online-Ressource (DE-627)306320843 (DE-600)1496359-0 (DE-576)081952953 nnns volume:230 GBV_USEFLAG_U GBV_ELV SYSFLAG_U FID-BIODIV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.00 Medizin: Allgemeines VZ AR 230 |
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10.1016/j.biosystems.2023.104955 doi (DE-627)ELV060281820 (ELSEVIER)S0303-2647(23)00130-2 DE-627 ger DE-627 rda eng 570 VZ BIODIV DE-30 fid 44.00 bkl Rubin, Sergio verfasserin (orcid)0000-0002-3387-7760 aut Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference 2023 nicht spezifiziert zzz rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. Self-fabrication Operational closure Closure to efficient causation Calculus of self-reference Non-algorithmic Enaction Final cause Enthalten in Biosystems Amsterdam [u.a.] : Elsevier Science, 1967 230 Online-Ressource (DE-627)306320843 (DE-600)1496359-0 (DE-576)081952953 nnns volume:230 GBV_USEFLAG_U GBV_ELV SYSFLAG_U FID-BIODIV SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_150 GBV_ILN_151 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2034 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2088 GBV_ILN_2106 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2470 GBV_ILN_2507 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.00 Medizin: Allgemeines VZ AR 230 |
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cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference |
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Cartography of the multiple formal systems of molecular autopoiesis: from the biology of cognition and enaction to anticipation and active inference |
| abstract |
A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. |
| abstractGer |
A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. |
| abstract_unstemmed |
A rich literature has grown up over the years that bears with autopoiesis, which tends to assume that it is a model, a theory, a principle, a definition of life, a property, refers to self-organization or even to hastily conclude that it is hylomorphic, hylozoist, in need of reformulation or to be overcome, making its status even more unclear. Maturana insists that autopoiesis is none of these and rather it is the causal organization of living systems as natural systems (NS) such that when it stops, they die. He calls this molecular autopoiesis (MA), which comprises two domains of existence: that of the self-producing organization (self-fabrication) and that of the structural coupling/enaction (cognition). Like all-NS in the universe, MA is amenable to be defined in theoretical terms, i.e. encoded in mathematical models and/or formal systems (FS). Framing the multiple formal systems of autopoiesis (FSA) into the Rosen's modeling relation (a process of bringing into equivalence the causality of NS and the inferential rules of FS), allows a classification of FSA into analytical categories, most importantly Turing machine (algorithmic) vs non-Turing machine (non-algorithmic) based, and FSA with a purely reactive mathematical image as cybernetic systems, i.e. feedbacks based, or conversely, as anticipatory systems making active inferences. It is thus the intent of the present work to advance the precision with which different FS may be observed to comply (preserve correspondence) with MA in its worldly state as a NS. The modeling relation between MA and the range of FS proposed as potentially illuminating their processes forecloses the applicability of Turing-based algorithmic computational models. This outcome indicates that MA, as modelled through Varela's calculus of self-reference or more especially through Rosen's (M,R)-system, is essentially anticipatory without violating structural determinism nor causality whatsoever, hence enaction may involve it. This quality may capture a fundamentally different mode of being in living systems as opposed to mechanical-computational systems. Implications in different fields of biology from the origin of life to planetary biology as well as in cognitive science and artificial intelligence are of interest. |
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| score |
7.4007883 |