THE HYPEREOSINOPHILIC SYNDROME REVISITED
Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiop...
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E-Artikel |
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2003 |
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16 |
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Annual Reviews Electronic Back Volume Collection 1932-2005ff |
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in: Annual review of medicine - Palo Alto, Calif. : Annual Reviews, 1950, 54(2003), Seite 169-184 |
| Übergeordnetes Werk: |
volume:54 ; year:2003 ; pages:169-184 ; extent:16 |
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| 520 | |a Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. | ||
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(DE-627)NLEJ164129847 DE-627 ger DE-627 rakwb THE HYPEREOSINOPHILIC SYNDROME REVISITED 2003 16 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. Annual Reviews Electronic Back Volume Collection 1932-2005ff Roufosse, Florence oth Cogan, Elie oth Goldman, Michel oth in Annual review of medicine Palo Alto, Calif. : Annual Reviews, 1950 54(2003), Seite 169-184 Online-Ressource (DE-627)NLEJ164018786 (DE-600)1481484-5 1545-326X nnns volume:54 year:2003 pages:169-184 extent:16 http://dx.doi.org/10.1146/annurev.med.54.101601.152431 text/html Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-ANR GBV_NL_ARTICLE AR 54 2003 169-184 16 |
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(DE-627)NLEJ164129847 DE-627 ger DE-627 rakwb THE HYPEREOSINOPHILIC SYNDROME REVISITED 2003 16 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. Annual Reviews Electronic Back Volume Collection 1932-2005ff Roufosse, Florence oth Cogan, Elie oth Goldman, Michel oth in Annual review of medicine Palo Alto, Calif. : Annual Reviews, 1950 54(2003), Seite 169-184 Online-Ressource (DE-627)NLEJ164018786 (DE-600)1481484-5 1545-326X nnns volume:54 year:2003 pages:169-184 extent:16 http://dx.doi.org/10.1146/annurev.med.54.101601.152431 text/html Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-ANR GBV_NL_ARTICLE AR 54 2003 169-184 16 |
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(DE-627)NLEJ164129847 DE-627 ger DE-627 rakwb THE HYPEREOSINOPHILIC SYNDROME REVISITED 2003 16 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. Annual Reviews Electronic Back Volume Collection 1932-2005ff Roufosse, Florence oth Cogan, Elie oth Goldman, Michel oth in Annual review of medicine Palo Alto, Calif. : Annual Reviews, 1950 54(2003), Seite 169-184 Online-Ressource (DE-627)NLEJ164018786 (DE-600)1481484-5 1545-326X nnns volume:54 year:2003 pages:169-184 extent:16 http://dx.doi.org/10.1146/annurev.med.54.101601.152431 text/html Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-ANR GBV_NL_ARTICLE AR 54 2003 169-184 16 |
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(DE-627)NLEJ164129847 DE-627 ger DE-627 rakwb THE HYPEREOSINOPHILIC SYNDROME REVISITED 2003 16 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. Annual Reviews Electronic Back Volume Collection 1932-2005ff Roufosse, Florence oth Cogan, Elie oth Goldman, Michel oth in Annual review of medicine Palo Alto, Calif. : Annual Reviews, 1950 54(2003), Seite 169-184 Online-Ressource (DE-627)NLEJ164018786 (DE-600)1481484-5 1545-326X nnns volume:54 year:2003 pages:169-184 extent:16 http://dx.doi.org/10.1146/annurev.med.54.101601.152431 text/html Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-ANR GBV_NL_ARTICLE AR 54 2003 169-184 16 |
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THE HYPEREOSINOPHILIC SYNDROME REVISITED |
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Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. |
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Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. |
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Abstract Clinical and biological features of patients with the idiopathic hypereosinophilic syndrome (HES) are heterogeneous. Recent evidence suggests at least two distinct underlying hematological disorders involving myeloid and lymphoid cells, respectively. We therefore suggest that the term idiopathic should be abandoned in the classification of HES. This review defines the "myeloproliferative" and "lymphocytic" variants of the HES and addresses the management of each variant, focusing on diagnosis and treatment of the newly identified lymphocytic variant. |
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