Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor
In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholin...
Ausführliche Beschreibung
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Englisch |
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1993 |
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Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 |
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Übergeordnetes Werk: |
in: Biochemical and Biophysical Research Communications - Amsterdam : Elsevier, 197(1993), 1, Seite 137-144 |
Übergeordnetes Werk: |
volume:197 ; year:1993 ; number:1 ; pages:137-144 |
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NLEJ183486382 |
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520 | |a In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. | ||
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(DE-627)NLEJ183486382 (DE-599)GBVNLZ183486382 DE-627 ger DE-627 rakwb eng Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor 1993 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Mihovilovic, M. oth Mai, Y. oth Herbstreith, M. oth Rubboli, F. oth Tarroni, P. oth Clementi, F. oth Roses, A.D. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 197(1993), 1, Seite 137-144 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:197 year:1993 number:1 pages:137-144 http://dx.doi.org/10.1006/bbrc.1993.2452 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 197 1993 1 137-144 |
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(DE-627)NLEJ183486382 (DE-599)GBVNLZ183486382 DE-627 ger DE-627 rakwb eng Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor 1993 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Mihovilovic, M. oth Mai, Y. oth Herbstreith, M. oth Rubboli, F. oth Tarroni, P. oth Clementi, F. oth Roses, A.D. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 197(1993), 1, Seite 137-144 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:197 year:1993 number:1 pages:137-144 http://dx.doi.org/10.1006/bbrc.1993.2452 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 197 1993 1 137-144 |
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(DE-627)NLEJ183486382 (DE-599)GBVNLZ183486382 DE-627 ger DE-627 rakwb eng Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor 1993 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Mihovilovic, M. oth Mai, Y. oth Herbstreith, M. oth Rubboli, F. oth Tarroni, P. oth Clementi, F. oth Roses, A.D. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 197(1993), 1, Seite 137-144 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:197 year:1993 number:1 pages:137-144 http://dx.doi.org/10.1006/bbrc.1993.2452 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 197 1993 1 137-144 |
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(DE-627)NLEJ183486382 (DE-599)GBVNLZ183486382 DE-627 ger DE-627 rakwb eng Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor 1993 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Mihovilovic, M. oth Mai, Y. oth Herbstreith, M. oth Rubboli, F. oth Tarroni, P. oth Clementi, F. oth Roses, A.D. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 197(1993), 1, Seite 137-144 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:197 year:1993 number:1 pages:137-144 http://dx.doi.org/10.1006/bbrc.1993.2452 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 197 1993 1 137-144 |
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(DE-627)NLEJ183486382 (DE-599)GBVNLZ183486382 DE-627 ger DE-627 rakwb eng Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor 1993 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Mihovilovic, M. oth Mai, Y. oth Herbstreith, M. oth Rubboli, F. oth Tarroni, P. oth Clementi, F. oth Roses, A.D. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 197(1993), 1, Seite 137-144 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:197 year:1993 number:1 pages:137-144 http://dx.doi.org/10.1006/bbrc.1993.2452 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 197 1993 1 137-144 |
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Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor |
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Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor |
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Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor |
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Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor |
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splicing of an anti-sense alu sequence generates a coding sequence variant for the α-3 subunit of a neuronal acetylcholine receptor |
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Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor |
abstract |
In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. |
abstractGer |
In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. |
abstract_unstemmed |
In this report we demonstrate that an α-3 acetylcholine receptor subunit transcriptional variant originates through alternative splicing of a complementary sequence of the right arm of an Alu element. This element is located within the 5.1Kb intron found between axons 5 and 6 of the α-3 acetylcholine receptor subunit gene. The transcriptional variant originates from the normal splicing process and carries an in-frame stop codon. If translated, it should encode for a peptide lacking the 4th transmembrane domain of the normal subunit. |
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Splicing of an Anti-sense Alu Sequence Generates a Coding Sequence Variant for the α-3 Subunit of a Neuronal Acetylcholine Receptor |
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