Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection
A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new gluc...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1988 |
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| Reproduktion: |
Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 |
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| Übergeordnetes Werk: |
in: Biochemical and Biophysical Research Communications - Amsterdam : Elsevier, 152(1988), 1, Seite 155-160 |
| Übergeordnetes Werk: |
volume:152 ; year:1988 ; number:1 ; pages:155-160 |
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NLEJ184953790 |
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| 520 | |a A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. | ||
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(DE-627)NLEJ184953790 (DE-599)GBVNLZ184953790 DE-627 ger DE-627 rakwb eng Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection 1988 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Datta, S.C. oth Radin, N.S. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 152(1988), 1, Seite 155-160 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:152 year:1988 number:1 pages:155-160 http://dx.doi.org/10.1016/S0006-291X(88)80693-4 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 152 1988 1 155-160 |
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(DE-627)NLEJ184953790 (DE-599)GBVNLZ184953790 DE-627 ger DE-627 rakwb eng Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection 1988 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Datta, S.C. oth Radin, N.S. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 152(1988), 1, Seite 155-160 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:152 year:1988 number:1 pages:155-160 http://dx.doi.org/10.1016/S0006-291X(88)80693-4 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 152 1988 1 155-160 |
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(DE-627)NLEJ184953790 (DE-599)GBVNLZ184953790 DE-627 ger DE-627 rakwb eng Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection 1988 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Datta, S.C. oth Radin, N.S. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 152(1988), 1, Seite 155-160 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:152 year:1988 number:1 pages:155-160 http://dx.doi.org/10.1016/S0006-291X(88)80693-4 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 152 1988 1 155-160 |
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(DE-627)NLEJ184953790 (DE-599)GBVNLZ184953790 DE-627 ger DE-627 rakwb eng Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection 1988 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Datta, S.C. oth Radin, N.S. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 152(1988), 1, Seite 155-160 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:152 year:1988 number:1 pages:155-160 http://dx.doi.org/10.1016/S0006-291X(88)80693-4 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 152 1988 1 155-160 |
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(DE-627)NLEJ184953790 (DE-599)GBVNLZ184953790 DE-627 ger DE-627 rakwb eng Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection 1988 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. Elsevier Journal Backfiles on ScienceDirect 1907 - 2002 Datta, S.C. oth Radin, N.S. oth in Biochemical and Biophysical Research Communications Amsterdam : Elsevier 152(1988), 1, Seite 155-160 (DE-627)NLEJ176855645 (DE-600)1461396-7 0006-291X nnns volume:152 year:1988 number:1 pages:155-160 http://dx.doi.org/10.1016/S0006-291X(88)80693-4 GBV_USEFLAG_H ZDB-1-SDJ GBV_NL_ARTICLE AR 152 1988 1 155-160 |
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normalization of liver glucosylceramide levels in the ''gaucher'' mouse by phosphatidylserine injection |
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Normalization of liver glucosylceramide levels in the ''Gaucher'' mouse by phosphatidylserine injection |
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A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. |
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A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. |
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A model of the human genetic disorder, Gaucher disease, can be rapidly generated in mice by the injection of emulsified glucosylceramide and an inhibitor of the lipid's hydrolase, conduritol B epoxide. The liver grows rapidly as it absorbs the load of lipid but the effect disappears as new glucosidase is formed and the load is hydrolyzed. This normalization process is accelerated by treatment with phosphatidylserine, which is a known stimulator of the enzyme. It is possible that injecting the phospholipid into Gaucher patients would have a therapeutic effect since it might help them utilize their residual glucosidase to destroy stored glycolipid. |
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