Human eosinophils: Apoptosis versus survival in the mediation of inflammation
Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1996 |
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| Umfang: |
8 |
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| Reproduktion: |
Springer Online Journal Archives 1860-2002 |
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| Übergeordnetes Werk: |
in: Apoptosis - 1996, 1(1996) vom: Feb., Seite 111-118 |
| Übergeordnetes Werk: |
volume:1 ; year:1996 ; month:02 ; pages:111-118 ; extent:8 |
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| 520 | |a Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. | ||
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| 700 | 1 | |a Walsh, Garry M. |4 oth | |
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(DE-627)NLEJ189933585 DE-627 ger DE-627 rakwb eng Human eosinophils: Apoptosis versus survival in the mediation of inflammation 1996 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. Springer Online Journal Archives 1860-2002 Dewson, Grant oth Wardlaw, Andrew J. oth Walsh, Garry M. oth in Apoptosis 1996 1(1996) vom: Feb., Seite 111-118 (DE-627)NLEJ188989056 (DE-600)1495863-6 1573-675X nnns volume:1 year:1996 month:02 pages:111-118 extent:8 http://dx.doi.org/10.1007/BF01321016 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 1 1996 2 111-118 8 |
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(DE-627)NLEJ189933585 DE-627 ger DE-627 rakwb eng Human eosinophils: Apoptosis versus survival in the mediation of inflammation 1996 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. Springer Online Journal Archives 1860-2002 Dewson, Grant oth Wardlaw, Andrew J. oth Walsh, Garry M. oth in Apoptosis 1996 1(1996) vom: Feb., Seite 111-118 (DE-627)NLEJ188989056 (DE-600)1495863-6 1573-675X nnns volume:1 year:1996 month:02 pages:111-118 extent:8 http://dx.doi.org/10.1007/BF01321016 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 1 1996 2 111-118 8 |
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(DE-627)NLEJ189933585 DE-627 ger DE-627 rakwb eng Human eosinophils: Apoptosis versus survival in the mediation of inflammation 1996 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. Springer Online Journal Archives 1860-2002 Dewson, Grant oth Wardlaw, Andrew J. oth Walsh, Garry M. oth in Apoptosis 1996 1(1996) vom: Feb., Seite 111-118 (DE-627)NLEJ188989056 (DE-600)1495863-6 1573-675X nnns volume:1 year:1996 month:02 pages:111-118 extent:8 http://dx.doi.org/10.1007/BF01321016 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 1 1996 2 111-118 8 |
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(DE-627)NLEJ189933585 DE-627 ger DE-627 rakwb eng Human eosinophils: Apoptosis versus survival in the mediation of inflammation 1996 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. Springer Online Journal Archives 1860-2002 Dewson, Grant oth Wardlaw, Andrew J. oth Walsh, Garry M. oth in Apoptosis 1996 1(1996) vom: Feb., Seite 111-118 (DE-627)NLEJ188989056 (DE-600)1495863-6 1573-675X nnns volume:1 year:1996 month:02 pages:111-118 extent:8 http://dx.doi.org/10.1007/BF01321016 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 1 1996 2 111-118 8 |
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(DE-627)NLEJ189933585 DE-627 ger DE-627 rakwb eng Human eosinophils: Apoptosis versus survival in the mediation of inflammation 1996 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. Springer Online Journal Archives 1860-2002 Dewson, Grant oth Wardlaw, Andrew J. oth Walsh, Garry M. oth in Apoptosis 1996 1(1996) vom: Feb., Seite 111-118 (DE-627)NLEJ188989056 (DE-600)1495863-6 1573-675X nnns volume:1 year:1996 month:02 pages:111-118 extent:8 http://dx.doi.org/10.1007/BF01321016 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 1 1996 2 111-118 8 |
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Human eosinophils: Apoptosis versus survival in the mediation of inflammation |
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Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. |
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Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. |
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Abstract Eosinophil-derived mediators are thought to make a major contribution to the inflammation underlying a number of allergic diseases, most notably asthma. The toxic potential of eosinophils at tissue sites of inflammation might be limited if they were cleared by the process of programmed cell death or apoptosis. In this review we have examined the relationship between the signal transduction pathways important in controlling cytokine-induced prolonged survival and the mechanisms responsible for the induction and control of apoptosis in the eosinophil. A greater understanding of these processes might result in the development of novel therapeutic agents which would promote the safe and rapid removal by apoptosis of this important pro-inflammatory cell. |
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