X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons
Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymo...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1992 |
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| Umfang: |
13 |
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| Reproduktion: |
Springer Online Journal Archives 1860-2002 |
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| Übergeordnetes Werk: |
in: Biochemical genetics - 1967, 30(1992) vom: Nov./Dez., Seite 567-579 |
| Übergeordnetes Werk: |
volume:30 ; year:1992 ; month:11/12 ; pages:567-579 ; extent:13 |
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NLEJ196698006 |
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| 520 | |a Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. | ||
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| 700 | 1 | |a Aivaliotis, Mary Jo |4 oth | |
| 700 | 1 | |a Samollow, Paul B. |4 oth | |
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(DE-627)NLEJ196698006 DE-627 ger DE-627 rakwb eng X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons 1992 13 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. Springer Online Journal Archives 1860-2002 VandeBerg, John L. oth Aivaliotis, Mary Jo oth Samollow, Paul B. oth in Biochemical genetics 1967 30(1992) vom: Nov./Dez., Seite 567-579 (DE-627)NLEJ18899274X (DE-600)1496197-0 1573-4927 nnns volume:30 year:1992 month:11/12 pages:567-579 extent:13 http://dx.doi.org/10.1007/BF00553801 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 30 1992 11/12 567-579 13 |
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(DE-627)NLEJ196698006 DE-627 ger DE-627 rakwb eng X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons 1992 13 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. Springer Online Journal Archives 1860-2002 VandeBerg, John L. oth Aivaliotis, Mary Jo oth Samollow, Paul B. oth in Biochemical genetics 1967 30(1992) vom: Nov./Dez., Seite 567-579 (DE-627)NLEJ18899274X (DE-600)1496197-0 1573-4927 nnns volume:30 year:1992 month:11/12 pages:567-579 extent:13 http://dx.doi.org/10.1007/BF00553801 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 30 1992 11/12 567-579 13 |
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(DE-627)NLEJ196698006 DE-627 ger DE-627 rakwb eng X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons 1992 13 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. Springer Online Journal Archives 1860-2002 VandeBerg, John L. oth Aivaliotis, Mary Jo oth Samollow, Paul B. oth in Biochemical genetics 1967 30(1992) vom: Nov./Dez., Seite 567-579 (DE-627)NLEJ18899274X (DE-600)1496197-0 1573-4927 nnns volume:30 year:1992 month:11/12 pages:567-579 extent:13 http://dx.doi.org/10.1007/BF00553801 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 30 1992 11/12 567-579 13 |
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(DE-627)NLEJ196698006 DE-627 ger DE-627 rakwb eng X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons 1992 13 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. Springer Online Journal Archives 1860-2002 VandeBerg, John L. oth Aivaliotis, Mary Jo oth Samollow, Paul B. oth in Biochemical genetics 1967 30(1992) vom: Nov./Dez., Seite 567-579 (DE-627)NLEJ18899274X (DE-600)1496197-0 1573-4927 nnns volume:30 year:1992 month:11/12 pages:567-579 extent:13 http://dx.doi.org/10.1007/BF00553801 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 30 1992 11/12 567-579 13 |
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(DE-627)NLEJ196698006 DE-627 ger DE-627 rakwb eng X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons 1992 13 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. Springer Online Journal Archives 1860-2002 VandeBerg, John L. oth Aivaliotis, Mary Jo oth Samollow, Paul B. oth in Biochemical genetics 1967 30(1992) vom: Nov./Dez., Seite 567-579 (DE-627)NLEJ18899274X (DE-600)1496197-0 1573-4927 nnns volume:30 year:1992 month:11/12 pages:567-579 extent:13 http://dx.doi.org/10.1007/BF00553801 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 30 1992 11/12 567-579 13 |
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x-linked glucose-6-phosphate dehydrogenase (g6pd) and autosomal 6-phosphogluconate dehydrogenase (6pgd) polymorphisms in baboons |
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X-linked glucose-6-phosphate dehydrogenase (G6PD) and autosomal 6-phosphogluconate dehydrogenase (6PGD) polymorphisms in baboons |
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Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. |
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Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. |
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Abstract Electrophoretic polymorphisms of glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate dehydrogenase (6PGD) were examined in captive colonies of five subspecies of baboons (Papio hamadryas). Phenotype frequencies and family data verified the X-linked inheritance of the G6PD polymorphism. Insufficient family data were available to confirm autosomal inheritance of the 6PGD polymorphism, but the electrophoretic patterns of variant types (putative heterozygotes) suggested the codominant expression of alleles at an autosomal locus. Implications of the G6PD polymorphism are discussed with regard to its utility as a marker system for research on X-chromosome inactivation during baboon development and for studies of clonal cell proliferation and/or cell selection during the development of atherosclerotic lesions in the baboon model. |
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