Enteral nutrition in the critically ill patient: A critical review of the evidence
Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary stu...
Ausführliche Beschreibung
Autor*in: |
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E-Artikel |
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Englisch |
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1993 |
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8 |
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Springer Online Journal Archives 1860-2002 |
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Übergeordnetes Werk: |
in: Intensive care medicine - 1975, 19(1993) vom: Aug., Seite 435-442 |
Übergeordnetes Werk: |
volume:19 ; year:1993 ; month:08 ; pages:435-442 ; extent:8 |
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NLEJ199999198 |
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520 | |a Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. | ||
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700 | 1 | |a Guyatt, G. H. |4 oth | |
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(DE-627)NLEJ199999198 DE-627 ger DE-627 rakwb eng Enteral nutrition in the critically ill patient: A critical review of the evidence 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. Springer Online Journal Archives 1860-2002 Heyland, D. K. oth Cook, D. J. oth Guyatt, G. H. oth in Intensive care medicine 1975 19(1993) vom: Aug., Seite 435-442 (DE-627)NLEJ188995587 (DE-600)1459201-0 1432-1238 nnns volume:19 year:1993 month:08 pages:435-442 extent:8 http://dx.doi.org/10.1007/BF01711083 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 19 1993 8 435-442 8 |
spelling |
(DE-627)NLEJ199999198 DE-627 ger DE-627 rakwb eng Enteral nutrition in the critically ill patient: A critical review of the evidence 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. Springer Online Journal Archives 1860-2002 Heyland, D. K. oth Cook, D. J. oth Guyatt, G. H. oth in Intensive care medicine 1975 19(1993) vom: Aug., Seite 435-442 (DE-627)NLEJ188995587 (DE-600)1459201-0 1432-1238 nnns volume:19 year:1993 month:08 pages:435-442 extent:8 http://dx.doi.org/10.1007/BF01711083 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 19 1993 8 435-442 8 |
allfields_unstemmed |
(DE-627)NLEJ199999198 DE-627 ger DE-627 rakwb eng Enteral nutrition in the critically ill patient: A critical review of the evidence 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. Springer Online Journal Archives 1860-2002 Heyland, D. K. oth Cook, D. J. oth Guyatt, G. H. oth in Intensive care medicine 1975 19(1993) vom: Aug., Seite 435-442 (DE-627)NLEJ188995587 (DE-600)1459201-0 1432-1238 nnns volume:19 year:1993 month:08 pages:435-442 extent:8 http://dx.doi.org/10.1007/BF01711083 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 19 1993 8 435-442 8 |
allfieldsGer |
(DE-627)NLEJ199999198 DE-627 ger DE-627 rakwb eng Enteral nutrition in the critically ill patient: A critical review of the evidence 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. Springer Online Journal Archives 1860-2002 Heyland, D. K. oth Cook, D. J. oth Guyatt, G. H. oth in Intensive care medicine 1975 19(1993) vom: Aug., Seite 435-442 (DE-627)NLEJ188995587 (DE-600)1459201-0 1432-1238 nnns volume:19 year:1993 month:08 pages:435-442 extent:8 http://dx.doi.org/10.1007/BF01711083 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 19 1993 8 435-442 8 |
allfieldsSound |
(DE-627)NLEJ199999198 DE-627 ger DE-627 rakwb eng Enteral nutrition in the critically ill patient: A critical review of the evidence 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. Springer Online Journal Archives 1860-2002 Heyland, D. K. oth Cook, D. J. oth Guyatt, G. H. oth in Intensive care medicine 1975 19(1993) vom: Aug., Seite 435-442 (DE-627)NLEJ188995587 (DE-600)1459201-0 1432-1238 nnns volume:19 year:1993 month:08 pages:435-442 extent:8 http://dx.doi.org/10.1007/BF01711083 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 19 1993 8 435-442 8 |
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enteral nutrition in the critically ill patient: a critical review of the evidence |
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Enteral nutrition in the critically ill patient: A critical review of the evidence |
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Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. |
abstractGer |
Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. |
abstract_unstemmed |
Abstract Objective To examine the relationship between enteral nutrition (EN) and infection in the critically ill.Setting: Computerized search of published research and review of relevant reference lists.Study selection: 151 citations were reviewed and 39 articles met selection criteria. Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity. |
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Primary studies were included if they evaluated EN in critically ill humans and its effect on infectious morbidity and mortality. Measurements and results Relevant data were abstracted on the timing and impact of EN on morbidity, the optimal route of administration, composition and pH of EN, and bacterial contamination of EN. The evidence from human studies that EN, particularly early EN, results in reduced septic morbidity as compared to parenteral nutrition is limited to small, unblinded studies with non-rigorous definitions of pneumonia. There is no evidence to support a preference of feeding into the stomach versus the small bowel. The addition of fish oil, arginine, glutamine and fiber to enteral feeds has a variable impact on survival in animal models; there are no trials in critically ill patients that demonstrate a reduction in infectious morbidity and mortality. Acidification of enteral nutrition results in decreased bacterial colonization of the stomach in critically ill patients. Bacterial contamination of enteral nutrition is an important source of infection. Conclusions Evidence from experimental data in critically ill patients suggests that enteral nutrition may have a favourable impact on gastrointestinal immunological function and infectious morbidity.</subfield></datafield><datafield tag="533" ind1=" " ind2=" "><subfield code="f">Springer Online Journal Archives 1860-2002</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Heyland, D. K.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Cook, D. J.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Guyatt, G. H.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">in</subfield><subfield code="t">Intensive care medicine</subfield><subfield code="d">1975</subfield><subfield code="g">19(1993) vom: Aug., Seite 435-442</subfield><subfield code="w">(DE-627)NLEJ188995587</subfield><subfield code="w">(DE-600)1459201-0</subfield><subfield code="x">1432-1238</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:19</subfield><subfield code="g">year:1993</subfield><subfield code="g">month:08</subfield><subfield code="g">pages:435-442</subfield><subfield code="g">extent:8</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">http://dx.doi.org/10.1007/BF01711083</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-1-SOJ</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_NL_ARTICLE</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">19</subfield><subfield code="j">1993</subfield><subfield code="c">8</subfield><subfield code="h">435-442</subfield><subfield code="g">8</subfield></datafield></record></collection>
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