Intestinal microcirculation in the neonate
Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of p...
Ausführliche Beschreibung
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1992 |
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| Umfang: |
7 |
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| Reproduktion: |
Springer Online Journal Archives 1860-2002 |
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| Übergeordnetes Werk: |
in: Pediatric surgery international - 1986, 7(1992) vom: Juni, Seite 408-414 |
| Übergeordnetes Werk: |
volume:7 ; year:1992 ; month:06 ; pages:408-414 ; extent:7 |
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| 520 | |a Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. | ||
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(DE-627)NLEJ203526511 DE-627 ger DE-627 rakwb eng Intestinal microcirculation in the neonate 1992 7 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. Springer Online Journal Archives 1860-2002 Velásquez, Otto R. oth Neil Granger, D. oth Crissinger, Karen D. oth in Pediatric surgery international 1986 7(1992) vom: Juni, Seite 408-414 (DE-627)NLEJ188989978 (DE-600)1463010-2 1437-9813 nnns volume:7 year:1992 month:06 pages:408-414 extent:7 http://dx.doi.org/10.1007/BF00178818 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 7 1992 6 408-414 7 |
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(DE-627)NLEJ203526511 DE-627 ger DE-627 rakwb eng Intestinal microcirculation in the neonate 1992 7 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. Springer Online Journal Archives 1860-2002 Velásquez, Otto R. oth Neil Granger, D. oth Crissinger, Karen D. oth in Pediatric surgery international 1986 7(1992) vom: Juni, Seite 408-414 (DE-627)NLEJ188989978 (DE-600)1463010-2 1437-9813 nnns volume:7 year:1992 month:06 pages:408-414 extent:7 http://dx.doi.org/10.1007/BF00178818 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 7 1992 6 408-414 7 |
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(DE-627)NLEJ203526511 DE-627 ger DE-627 rakwb eng Intestinal microcirculation in the neonate 1992 7 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. Springer Online Journal Archives 1860-2002 Velásquez, Otto R. oth Neil Granger, D. oth Crissinger, Karen D. oth in Pediatric surgery international 1986 7(1992) vom: Juni, Seite 408-414 (DE-627)NLEJ188989978 (DE-600)1463010-2 1437-9813 nnns volume:7 year:1992 month:06 pages:408-414 extent:7 http://dx.doi.org/10.1007/BF00178818 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 7 1992 6 408-414 7 |
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(DE-627)NLEJ203526511 DE-627 ger DE-627 rakwb eng Intestinal microcirculation in the neonate 1992 7 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. Springer Online Journal Archives 1860-2002 Velásquez, Otto R. oth Neil Granger, D. oth Crissinger, Karen D. oth in Pediatric surgery international 1986 7(1992) vom: Juni, Seite 408-414 (DE-627)NLEJ188989978 (DE-600)1463010-2 1437-9813 nnns volume:7 year:1992 month:06 pages:408-414 extent:7 http://dx.doi.org/10.1007/BF00178818 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 7 1992 6 408-414 7 |
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(DE-627)NLEJ203526511 DE-627 ger DE-627 rakwb eng Intestinal microcirculation in the neonate 1992 7 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. Springer Online Journal Archives 1860-2002 Velásquez, Otto R. oth Neil Granger, D. oth Crissinger, Karen D. oth in Pediatric surgery international 1986 7(1992) vom: Juni, Seite 408-414 (DE-627)NLEJ188989978 (DE-600)1463010-2 1437-9813 nnns volume:7 year:1992 month:06 pages:408-414 extent:7 http://dx.doi.org/10.1007/BF00178818 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 7 1992 6 408-414 7 |
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Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. |
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Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. |
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Abstract The hypermetabolic neonatal intestine requires an appropriate supply of energy to meet its needs. Adequate regulatory mechanisms for intestinal blood flow must be present to cope with a high oxygen demand. In the developing intestine, intrinsic hemodynamic control matures as a function of postnatal age, and the newborn appears to balance this vascular immaturity by an enhanced ability to extract oxygen from the blood. When this mechanism is maximized and an additional stress is superimposed, the neonatal intestine may be at a higher risk of ischemia and injury than older intestine. Furthermore, sympathetic vasoconstrictor control predominates at birth, and the newborn is less capable of vasodilatation. This extrinsic control provides and additional ischemic risk during conditions involving sympathetic discharge. An understanding of circulatory hemodynamics in the developing intestine may allow elucidation of the role played by ischemia in mucosal injury in the newborn. |
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